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Transcriptional Activity of CCAAT/Enhancer-binding Proteins Is Controlled by a Conserved Inhibitory Domain That Is a Target for Sumoylation

The Journal of biological chemistry, 2002-10, Vol.277 (41), p.38037-38044

2002

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A c-jun dominant negative mutant protects sympathetic neurons against programmed cell death

Neuron (Cambridge, Mass.), 1995-05, Vol.14 (5), p.927-939

1995

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Transformation by ras modifies AP1 composition and activity

Oncogene, 1997-02, Vol.14 (7), p.837-847

1997

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Regulation of Two JunD Isoforms by Jun N-terminal Kinases

The Journal of biological chemistry, 2002-08, Vol.277 (33), p.29710-29718

2002

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Translational Regulation of the JunD Messenger RNA

The Journal of biological chemistry, 2002-09, Vol.277 (36), p.32697-32705

2002

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Analysis of Pre-clerkship Medical Students’ Perceptions and Performance During the COVID-19 Pandemic

Medical science educator, 2023-02, Vol.33 (1), p.147-156

2023

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Variations in Jun and Fos protein expression and AP-1 activity in cycling, resting and stimulated fibroblasts

Oncogene, 1997-02, Vol.14 (7), p.819-830

1997

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Personalized Medicine in Undergraduate Medical Education: a Spiral Learning Model

Medical science educator, 2020-12, Vol.30 (4), p.1741-1744

2020

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Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras

Cell, 1994-02, Vol.76 (4), p.747-760

1994

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Differential binding of the Menin tumor suppressor protein to JunD isoforms

Cancer research (Chicago, Ill.), 2001-02, Vol.61 (3), p.916-920

2001

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Stress-activated protein kinases are negatively regulated by cell density

The EMBO journal, 1998-10, Vol.17 (19), p.5615-5626

1998

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Requirement of an Upstream AP-1 Motif for the Constitutive and Phorbol Ester-inducible Expression of the Urokinase-type Plasminogen Activator Receptor Gene

The Journal of biological chemistry, 1996-09, Vol.271 (38), p.23176-23184

1996

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A Scholarly Activity and Research Requirement at the Paul L. Foster School of Medicine

Medical science educator, 2011-04, Vol.21 (Suppl 1), p.91-97

2011

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A Laboratory for Education in Molecular Medicine: a Dedicated Resource for Medical Student Research

Medical science educator, 2013-04, Vol.23 (Suppl 1), p.108-118

2013

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Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Molecular and Cellular Biology, 1996-05, Vol.16 (5), p.1881-1888

1996

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Mitotic Motors

The Journal of cell biology, 1991-11, Vol.115 (3), p.577-585

1991

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The glucocorticoid receptor and AP-1 are involved in a positive regulation of the muscle regulatory gene myf5 in cultured myoblasts

Journal of cell science, 1997-11, Vol.110 ( Pt 22) (22), p.2771-2779

1997

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Cyclic AMP stimulates a JunD/Fra-2 AP-1 complex and inhibits the proliferation of interleukin-6-dependent cell lines

Oncogene, 1995-09, Vol.11 (6), p.1069-1078

1995

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Increased transforming activity of JunB and JunD by introduction of an heterologous homodimerization domain

Oncogene, 1995-02, Vol.10 (3), p.495-507

1995

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Antisense c-jun overcomes a differentiation block in a murine erythroleukemia cell line

Oncogene, 1994-07, Vol.9 (7), p.1957-1964

1994

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In vitro transforming capacities of mouse c-jun:junD chimeric genes

Oncogene, 1993-08, Vol.8 (8), p.2311-2315

1993

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Stepwise transformation of rat embryo fibroblasts : c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Molecular and cellular biology, 1996 (16), p.1881-1888

1996

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Increased transforming activity of JunB and JunD by introduction of an heterologous homodimerization domain

Oncogene, 1995 (10), p.495-507

1995

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In vitro transforming capacities of mouse c-jun : junD chimeric genes

Oncogene, 1993 (88), p.2311-2315

1993

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Transcriptional Activity of CCAAT/Enhancer-binding Proteins Is Controlled by a Conserved Inhibitory Domain That Is a Target for Sumoylation

A c-jun dominant negative mutant protects sympathetic neurons against programmed cell death

Transformation by ras modifies AP1 composition and activity

Regulation of Two JunD Isoforms by Jun N-terminal Kinases

Translational Regulation of the JunD Messenger RNA

Analysis of Pre-clerkship Medical Students’ Perceptions and Performance During the COVID-19 Pandemic

Variations in Jun and Fos protein expression and AP-1 activity in cycling, resting and stimulated fibroblasts

Personalized Medicine in Undergraduate Medical Education: a Spiral Learning Model

Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras

Differential binding of the Menin tumor suppressor protein to JunD isoforms

Stress-activated protein kinases are negatively regulated by cell density

Requirement of an Upstream AP-1 Motif for the Constitutive and Phorbol Ester-inducible Expression of the Urokinase-type Plasminogen Activator Receptor Gene

A Scholarly Activity and Research Requirement at the Paul L. Foster School of Medicine

A Laboratory for Education in Molecular Medicine: a Dedicated Resource for Medical Student Research

Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Mitotic Motors

The glucocorticoid receptor and AP-1 are involved in a positive regulation of the muscle regulatory gene myf5 in cultured myoblasts

Cyclic AMP stimulates a JunD/Fra-2 AP-1 complex and inhibits the proliferation of interleukin-6-dependent cell lines

Increased transforming activity of JunB and JunD by introduction of an heterologous homodimerization domain

Antisense c-jun overcomes a differentiation block in a murine erythroleukemia cell line

In vitro transforming capacities of mouse c-jun:junD chimeric genes

Stepwise transformation of rat embryo fibroblasts : c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Increased transforming activity of JunB and JunD by introduction of an heterologous homodimerization domain

In vitro transforming capacities of mouse c-jun : junD chimeric genes

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