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Resveratrol pretreatment protects rat brain from cerebral ischemic damage via a sirtuin 1–uncoupling protein 2 pathway

Neuroscience, 2009-03, Vol.159 (3), p.993-1002

2009

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Anoxic preconditioning in hippocampal slices: role of adenosine

Neuroscience, 1996-12, Vol.75 (3), p.687-694

1996

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Folistatina, resistencia a la insulina y composición corporal en adultos colombianos

Revista Colombiana de endocrinología, diabetes & metabolismo, 2018-03, Vol.5 (1), p.7-14

2018

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Post-ischemic activation of protein kinase C epsilon protects the hippocampus from cerebral ischemic injury via alterations in cerebral blood flow

Neuroscience letters, 2011-01, Vol.487 (2), p.158-162

2011

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Estrogen preconditioning protects the hippocampal CA1 against ischemia

Neuroscience, 2006-01, Vol.141 (4), p.1721-1730

2006

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Derangements of post-ischemic cerebral blood flow by protein kinase C delta

Neuroscience, 2010-12, Vol.171 (2), p.566-576

2010

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Ischemic preconditioning via epsilon protein kinase C activation requires cyclooxygenase-2 activation in vitro

Neuroscience, 2007-03, Vol.145 (3), p.931-941

2007

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Pretreatment with a single estradiol-17β bolus activates cyclic-AMP response element binding protein and protects CA1 neurons against global cerebral ischemia

Neuroscience, 2009-05, Vol.160 (2), p.307-318

2009

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Oxygen‐conserving reflexes of the brain: the current molecular knowledge

Journal of cellular and molecular medicine, 2009-04, Vol.13 (4), p.644-647

2009

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Ischemic Preconditioning Preserves Mitochondrial Function After Global Cerebral Ischemia in Rat Hippocampus

Journal of cerebral blood flow and metabolism, 2001-12, Vol.21 (12), p.1401-1410

2001

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Rapid preconditioning neuroprotection following anoxia in hippocampal slices: Role of the k + ATP channel and protein kinase C

Neuroscience, 1999-03, Vol.89 (2), p.453-459

1999

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Focal Ischemic Preconditioning Induces Rapid Tolerance to Middle Cerebral Artery Occlusion in Mice

Journal of cerebral blood flow and metabolism, 1999-07, Vol.19 (7), p.757-761

1999

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The effect of rapid preconditioning on the microglial, astrocytic and neuronal consequences of global cerebral ischemia

Acta neuropathologica, 1999-05, Vol.97 (5), p.495-501

1999

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Chronic failure in the maintenance of long-term potentiation following fluid percussion injury in the rat

Brain research, 2000-04, Vol.861 (1), p.69-76

2000

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Extracellular potassium, volume fraction, and tortuosity in rat hippocampal CA1, CA3, and cortical slices during ischemia

Journal of neurophysiology, 1995-08, Vol.74 (2), p.565-573

1995

Volltextzugriff (PDF)

Cytochrome C Is Released from Mitochondria Into the Cytosol after Cerebral Anoxia or Ischemia

Journal of cerebral blood flow and metabolism, 1999-01, Vol.19 (1), p.39-43

1999

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SNX-111, a novel, presynaptic N-type calcium channel antagonist, is neuroprotective against focal cerebral ischemia in rabbits

Journal of the neurological sciences, 1997-12, Vol.153 (1), p.25-31

1997

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Nitric oxide is involved in anoxic preconditioning neuroprotection in rat hippocampal slices

Brain research, 1999-07, Vol.836 (1), p.62-69

1999

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Antioxidants, mitochondrial hyperoxidation and electrical recovery after anoxia in hippocampal slices

Brain research, 1997-04, Vol.754 (1), p.163-170

1997

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Impaired expression of long-term potentiation in hippocampal slices 4 and 48 h following mild fluid-percussion brain injury in vivo

Brain research, 1998-03, Vol.785 (2), p.287-292

1998

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Adenosine, a "retaliatory" metabolite, promotes anoxia tolerance in turtle brain

Journal of cerebral blood flow and metabolism, 1993-07, Vol.13 (4), p.728

1993

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Ascorbic acid, but not glutathione, is taken up by brain slices and preserves cell morphology

Journal of neurophysiology, 1994-04, Vol.71 (4), p.1591-1596

1994

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Induction of tolerance against traumatic brain injury by ischemic preconditioning

Neuroreport, 1999-09, Vol.10 (14), p.2951-2954

1999

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Calcium and increase excitability promote tolerance against anoxia in hippocampal slices

Brain research, 1999-06, Vol.833 (1), p.20-26

1999

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Narrow temporal therapeutic window for NMDA antagonist protection against focal cerebral ischaemia

Neurobiology of disease, 1995-04, Vol.2 (2), p.109-118

1995

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Resveratrol pretreatment protects rat brain from cerebral ischemic damage via a sirtuin 1–uncoupling protein 2 pathway

Anoxic preconditioning in hippocampal slices: role of adenosine

Folistatina, resistencia a la insulina y composición corporal en adultos colombianos

Post-ischemic activation of protein kinase C epsilon protects the hippocampus from cerebral ischemic injury via alterations in cerebral blood flow

Estrogen preconditioning protects the hippocampal CA1 against ischemia

Derangements of post-ischemic cerebral blood flow by protein kinase C delta

Ischemic preconditioning via epsilon protein kinase C activation requires cyclooxygenase-2 activation in vitro

Pretreatment with a single estradiol-17β bolus activates cyclic-AMP response element binding protein and protects CA1 neurons against global cerebral ischemia

Oxygen‐conserving reflexes of the brain: the current molecular knowledge

Ischemic Preconditioning Preserves Mitochondrial Function After Global Cerebral Ischemia in Rat Hippocampus

Rapid preconditioning neuroprotection following anoxia in hippocampal slices: Role of the k + ATP channel and protein kinase C

Focal Ischemic Preconditioning Induces Rapid Tolerance to Middle Cerebral Artery Occlusion in Mice

The effect of rapid preconditioning on the microglial, astrocytic and neuronal consequences of global cerebral ischemia

Chronic failure in the maintenance of long-term potentiation following fluid percussion injury in the rat

Extracellular potassium, volume fraction, and tortuosity in rat hippocampal CA1, CA3, and cortical slices during ischemia

Cytochrome C Is Released from Mitochondria Into the Cytosol after Cerebral Anoxia or Ischemia

SNX-111, a novel, presynaptic N-type calcium channel antagonist, is neuroprotective against focal cerebral ischemia in rabbits

Nitric oxide is involved in anoxic preconditioning neuroprotection in rat hippocampal slices

Antioxidants, mitochondrial hyperoxidation and electrical recovery after anoxia in hippocampal slices

Impaired expression of long-term potentiation in hippocampal slices 4 and 48 h following mild fluid-percussion brain injury in vivo

Adenosine, a "retaliatory" metabolite, promotes anoxia tolerance in turtle brain

Ascorbic acid, but not glutathione, is taken up by brain slices and preserves cell morphology

Induction of tolerance against traumatic brain injury by ischemic preconditioning

Calcium and increase excitability promote tolerance against anoxia in hippocampal slices

Narrow temporal therapeutic window for NMDA antagonist protection against focal cerebral ischaemia

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