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Complexity of Generating Mouse Models to Study the Upper Motor Neurons: Let Us Shift Focus from Mice to Neurons

International journal of molecular sciences, 2019-08, Vol.20 (16), p.3848

2019

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Mitochondrial dysregulation occurs early in ALS motor cortex with TDP-43 pathology and suggests maintaining NAD + balance as a therapeutic strategy

Scientific reports, 2022-03, Vol.12 (1), p.4287-4287, Article 4287

2022

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Incorporating upper motor neuron health in ALS drug discovery

Drug discovery today, 2018-03, Vol.23 (3), p.696-703

2018

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IGF-I specifically enhances axon outgrowth of corticospinal motor neurons

Nature neuroscience, 2006-11, Vol.9 (11), p.1371-1381

2006

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Mitoautophagy: A Unique Self-Destructive Path Mitochondria of Upper Motor Neurons With TDP-43 Pathology Take, Very Early in ALS

Frontiers in cellular neuroscience, 2019-11, Vol.13, p.489-489

2019

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NU-9 improves health of hSOD1G93A mouse upper motor neurons in vitro, especially in combination with riluzole or edaravone

Scientific reports, 2022-03, Vol.12 (1), p.1-11, Article 5383

2022

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Spastin and alsin protein interactome analyses begin to reveal key canonical pathways and suggest novel druggable targets

Neural regeneration research, 2025-03, Vol.20 (3), p.725-739

2025

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Mutant Profilin1 transgenic mice recapitulate cardinal features of motor neuron disease

Human molecular genetics, 2017-02, Vol.26 (4), p.686

2017

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A novel SOD1-ALS mutation separates central and peripheral effects of mutant SOD1 toxicity

Human molecular genetics, 2015-04, Vol.24 (7), p.1883-1897

2015

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An autism-associated variant of Epac2 reveals a role for Ras/Epac2 signaling in controlling basal dendrite maintenance in mice

PLoS biology, 2012-06, Vol.10 (6), p.e1001350-e1001350

2012

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Mutations and Protein Interaction Landscape Reveal Key Cellular Events Perturbed in Upper Motor Neurons with HSP and PLS

Brain sciences, 2021-04, Vol.11 (5), p.578

2021

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The Timing and Extent of Motor Neuron Vulnerability in ALS Correlates with Accumulation of Misfolded SOD1 Protein in the Cortex and in the Spinal Cord

Cells (Basel, Switzerland), 2020-02, Vol.9 (2), p.502

2020

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Expanded access: opening doors to personalized medicine for rare disease patients and patients with neurodegenerative diseases

The FEBS journal, 2021-03, Vol.288 (5), p.1457-1461

Ozdinler, P. Hande

2021

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The Electrophysiological Determinants of Corticospinal Motor Neuron Vulnerability in ALS

Frontiers in molecular neuroscience, 2020-05, Vol.13, p.73-73

2020

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TDP-43 protein interactome informs about perturbed canonical pathways and may help develop personalized medicine approaches for patients with TDP-43 pathology

Drug discovery today, 2023-11, Vol.28 (11), p.103769-103769, Article 103769

2023

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Mitochondria, ER, and nuclear membrane defects reveal early mechanisms for upper motor neuron vulnerability with respect to TDP-43 pathology

Acta neuropathologica, 2019-01, Vol.137 (1), p.47-69

2019

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Help from peripheral macrophages in ALS?

Nature neuroscience, 2020-11, Vol.23 (11), p.1311

Özdinler, P Hande

2020

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Protein-protein interactions reveal key canonical pathways, upstream regulators, interactome domains, and novel targets in ALS

Scientific reports, 2018-10, Vol.8 (1), p.14732-19, Article 14732

2018

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Upper motor neurons are a target for gene therapy and UCHL1 is necessary and sufficient to improve cellular integrity of diseased upper motor neurons

Gene therapy, 2022-04, Vol.29 (3-4), p.178-192

2022

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Importance of lipids for upper motor neuron health and disease

Seminars in cell & developmental biology, 2021-04, Vol.112, p.92-104

2021

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Treatment of Amyotrophic Lateral Sclerosis: Lessons Learned from Many Failures

ACS medicinal chemistry letters, 2014-11, Vol.5 (11), p.1179-1181

2014

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MCP1-CCR2 and neuroinflammation in the ALS motor cortex with TDP-43 pathology

Journal of neuroinflammation, 2019-10, Vol.16 (1), p.196-196, Article 196

2019

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Slit2, a Branching-Arborization Factor for Sensory Axons in the Mammalian CNS

The Journal of neuroscience, 2002-06, Vol.22 (11), p.4540-4549

2002

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Apical dendrite degeneration, a novel cellular pathology for Betz cells in ALS

Scientific reports, 2017-02, Vol.7 (1), p.41765-41765, Article 41765

2017

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Modeling mammalian spermatogonial differentiation and meiotic initiation in vitro

Development (Cambridge), 2022-11, Vol.149 (22)

2022

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Complexity of Generating Mouse Models to Study the Upper Motor Neurons: Let Us Shift Focus from Mice to Neurons

Mitochondrial dysregulation occurs early in ALS motor cortex with TDP-43 pathology and suggests maintaining NAD + balance as a therapeutic strategy

Incorporating upper motor neuron health in ALS drug discovery

IGF-I specifically enhances axon outgrowth of corticospinal motor neurons

Mitoautophagy: A Unique Self-Destructive Path Mitochondria of Upper Motor Neurons With TDP-43 Pathology Take, Very Early in ALS

NU-9 improves health of hSOD1G93A mouse upper motor neurons in vitro, especially in combination with riluzole or edaravone

Spastin and alsin protein interactome analyses begin to reveal key canonical pathways and suggest novel druggable targets

Mutant Profilin1 transgenic mice recapitulate cardinal features of motor neuron disease

A novel SOD1-ALS mutation separates central and peripheral effects of mutant SOD1 toxicity

An autism-associated variant of Epac2 reveals a role for Ras/Epac2 signaling in controlling basal dendrite maintenance in mice

Mutations and Protein Interaction Landscape Reveal Key Cellular Events Perturbed in Upper Motor Neurons with HSP and PLS

The Timing and Extent of Motor Neuron Vulnerability in ALS Correlates with Accumulation of Misfolded SOD1 Protein in the Cortex and in the Spinal Cord

Expanded access: opening doors to personalized medicine for rare disease patients and patients with neurodegenerative diseases

The Electrophysiological Determinants of Corticospinal Motor Neuron Vulnerability in ALS

TDP-43 protein interactome informs about perturbed canonical pathways and may help develop personalized medicine approaches for patients with TDP-43 pathology

Mitochondria, ER, and nuclear membrane defects reveal early mechanisms for upper motor neuron vulnerability with respect to TDP-43 pathology

Help from peripheral macrophages in ALS?

Protein-protein interactions reveal key canonical pathways, upstream regulators, interactome domains, and novel targets in ALS

Upper motor neurons are a target for gene therapy and UCHL1 is necessary and sufficient to improve cellular integrity of diseased upper motor neurons

Importance of lipids for upper motor neuron health and disease

Treatment of Amyotrophic Lateral Sclerosis: Lessons Learned from Many Failures

MCP1-CCR2 and neuroinflammation in the ALS motor cortex with TDP-43 pathology

Slit2, a Branching-Arborization Factor for Sensory Axons in the Mammalian CNS

Apical dendrite degeneration, a novel cellular pathology for Betz cells in ALS

Modeling mammalian spermatogonial differentiation and meiotic initiation in vitro

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