UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Gene Therapy Strategies for RAG1 Deficiency: Challenges and Breakthroughs

Immunology letters, 2024-09, p.106931, Article 106931

2024

Volltextzugriff (PDF)

Advances in gene therapy for inborn errors of immunity

Current opinion in allergy and clinical immunology, 2023-12, Vol.23 (6), p.467-477

2023

Volltextzugriff (PDF)

Persistent Hypogammaglobulinemia after Receiving Rituximab Post-HSCT Is Not Caused by an Intrinsic B Cell Defect

International journal of molecular sciences, 2023-11, Vol.24 (21), p.16012

2023

Volltextzugriff (PDF)

Partial correction of immunodeficiency by lentiviral vector gene therapy in mouse models carrying Rag1 hypomorphic mutations

Frontiers in immunology, 2023, Vol.14, p.1268620-1268620

2023

Volltextzugriff (PDF)

Cysteine and hydrophobic residues in CDR3 serve as distinct T-cell self-reactivity indices

Journal of allergy and clinical immunology, 2019-07, Vol.144 (1), p.333-336

2019

Volltextzugriff (PDF)

Abnormalities of T-cell receptor repertoire in CD4+ regulatory and conventional T cells in patients with RAG mutations: Implications for autoimmunity

Journal of allergy and clinical immunology, 2017-12, Vol.140 (6), p.1739-1743.e7

2017

Volltextzugriff (PDF)

Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56bright NKG2A+++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Frontiers in immunology, 2017-07, Vol.8, p.798-798

2017

Volltextzugriff (PDF)

EXTL3 mutations cause skeletal dysplasia, immune deficiency, and developmental delay

The Journal of experimental medicine, 2017-03, Vol.214 (3), p.623-637

2017

Volltextzugriff (PDF)

Corrigendum: Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56 bright NKG2A +++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Frontiers in immunology, 2017, Vol.8, p.1244

2017

Volltextzugriff (PDF)

Corrigendum: Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56bright NKG2A+++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Frontiers in immunology, 2017-01, Vol.8, p.1244-1244

2017

Volltextzugriff (PDF)

Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56 bright NKG2A +++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Frontiers in immunology, 2017, Vol.8, p.798

2017

Volltextzugriff (PDF)

Novel Genome-Editing Tools to Model and Correct Primary Immunodeficiencies

Frontiers in immunology, 2015-05, Vol.6, p.250-250

2015

Volltextzugriff (PDF)

1 – 15 von 15

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(14)
Mit Volltextzugriff
(13)

Publikationsform

Artikel
(15)

Sprache

Englisch
(15)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Gene Therapy Strategies for RAG1 Deficiency: Challenges and Breakthroughs

Advances in gene therapy for inborn errors of immunity

Persistent Hypogammaglobulinemia after Receiving Rituximab Post-HSCT Is Not Caused by an Intrinsic B Cell Defect

Partial correction of immunodeficiency by lentiviral vector gene therapy in mouse models carrying Rag1 hypomorphic mutations

Human JAK1 gain of function causes dysregulated myelopoeisis and severe allergic inflammation

Cysteine and hydrophobic residues in CDR3 serve as distinct T-cell self-reactivity indices

Abnormalities of T-cell receptor repertoire in CD4+ regulatory and conventional T cells in patients with RAG mutations: Implications for autoimmunity

Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56bright NKG2A+++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

EXTL3 mutations cause skeletal dysplasia, immune deficiency, and developmental delay

Corrigendum: Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56 bright NKG2A +++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Corrigendum: Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56bright NKG2A+++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Natural Killer Cells from Patients with Recombinase-Activating Gene and Non-Homologous End Joining Gene Defects Comprise a Higher Frequency of CD56 bright NKG2A +++ Cells, and Yet Display Increased Degranulation and Higher Perforin Content

Characterization of T and B cell repertoire diversity in patients with RAG deficiency

Modeling altered T-cell development with induced pluripotent stem cells from patients with RAG1-dependent immune deficiencies

Novel Genome-Editing Tools to Model and Correct Primary Immunodeficiencies

Suchergebnisse filtern