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Development affects in vitro vascular tone and calcium sensitivity in ovine cerebral arteries

The Journal of physiology, 2004-08, Vol.558 (3), p.883-896

2004

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N-terminal chromogranin-derived peptides as dilators of bovine coronary resistance arteries

Regulatory peptides, 2002-05, Vol.105 (2), p.93-100

2002

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Augmented Expression of Piezo1 in the Uterine Vasculature During Pregnancy

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Pressure‐induced actin polymerization in vascular smooth muscle as a mechanism underlying myogenic behavior

The FASEB journal, 2002-01, Vol.16 (1), p.72-76

2002

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Myogenic tone, reactivity, and forced dilatation: a three-phase model of in vitro arterial myogenic behavior

American journal of physiology. Heart and circulatory physiology, 2002-12, Vol.283 (6), p.H2260-H2267

2002

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Loss of SM-B myosin affects muscle shortening velocity and maximal force development

Nature cell biology, 2001-11, Vol.3 (11), p.1025-1029

2001

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Gestation increases nitric oxide–mediated vasodilation in rat uterine arteries

American journal of obstetrics and gynecology, 1997-04, Vol.176 (4), p.856-864

1997

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Vascular smooth muscle actin cytoskeleton in cerebral artery forced dilatation

Stroke (1970), 1998-06, Vol.29 (6), p.1223-1228

1998

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Estrogen augments the vasodilatory effects of vascular endothelial growth factor in the uterine circulation of the rat

American journal of obstetrics and gynecology, 2000-08, Vol.183 (2), p.449-453

2000

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Sildenafil Increases Uterine Blood Flow in Nonpregnant Nulliparous Women

Reproductive sciences (Thousand Oaks, Calif.), 2010-04, Vol.17 (4), p.358-365

2010

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High glucose concentrations dilate cerebral arteries and diminish myogenic tone through an endothelial mechanism

Stroke (1970), 1997-02, Vol.28 (2), p.405-411

1997

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Isoform switching from SM-B to SM-A myosin results in decreased contractility and altered expression of thin filament regulatory proteins

American Journal of Physiology: Cell Physiology, 2004-09, Vol.287 (3), p.C723-C729

2004

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Pregnancy augments uteroplacental vascular endothelial growth factor gene expression and vasodilator effects

American journal of physiology. Heart and circulatory physiology, 1997-08, Vol.273 (2), p.H938-H944

1997

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Effects of h1-calponin ablation on the contractile properties of bladder versus vascular smooth muscle in mice lacking SM-B myosin

The Journal of physiology, 2006-12, Vol.577 (3), p.1033-1042

2006

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Prologue: vascular myogenic mechanisms

American journal of physiology. Heart and circulatory physiology, 2002-12, Vol.283 (6), p.H2157-H2159

2002

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Vascular smooth muscle cell stress as a determinant of cerebral artery myogenic tone

American journal of physiology. Heart and circulatory physiology, 2002-12, Vol.283 (6), p.H2210-H2216

2002

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Structural remodeling of rat uterine veins in pregnancy

American journal of obstetrics and gynecology, 2002-12, Vol.187 (6), p.1647-1652

2002

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Myoendometrial versus placental uterine arteries: Structural, mechanical, and functional differences in late-pregnant rabbits

American journal of obstetrics and gynecology, 1997-07, Vol.177 (1), p.215-221

1997

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Increased Ca2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries

American journal of physiology. Heart and circulatory physiology, 1999-09, Vol.277 (3), p.H1178

1999

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Isoform switching from SM-B to SM-A myosin results in decreased contractility and altered expression of thin filament regulatory proteins

American Journal of Physiology: Cell Physiology, 2004-09, Vol.56 (3), p.C723-C729

2004

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Increased Ca2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries

American journal of physiology. Heart and circulatory physiology, 1999-09, Vol.46 (3), p.H1178-H1188

1999

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Vascular smooth muscle actin cytoskeleton in cerebral artery forced dilatation / Editorial comment

Stroke (1970), 1998-06, Vol.29 (6), p.1223

1998

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Increased Ca 2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries

American journal of physiology. Heart and circulatory physiology, 1999-09, Vol.277 (3), p.H1178-H1188

1999

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Development affects in vitro vascular tone and calcium sensitivity in ovine cerebral arteries

N-terminal chromogranin-derived peptides as dilators of bovine coronary resistance arteries

Augmented Expression of Piezo1 in the Uterine Vasculature During Pregnancy

Pressure‐induced actin polymerization in vascular smooth muscle as a mechanism underlying myogenic behavior

Myogenic tone, reactivity, and forced dilatation: a three-phase model of in vitro arterial myogenic behavior

Loss of SM-B myosin affects muscle shortening velocity and maximal force development

Gestation increases nitric oxide–mediated vasodilation in rat uterine arteries

Vascular smooth muscle actin cytoskeleton in cerebral artery forced dilatation

Estrogen augments the vasodilatory effects of vascular endothelial growth factor in the uterine circulation of the rat

Sildenafil Increases Uterine Blood Flow in Nonpregnant Nulliparous Women

High glucose concentrations dilate cerebral arteries and diminish myogenic tone through an endothelial mechanism

Isoform switching from SM-B to SM-A myosin results in decreased contractility and altered expression of thin filament regulatory proteins

Pregnancy augments uteroplacental vascular endothelial growth factor gene expression and vasodilator effects

Effects of h1-calponin ablation on the contractile properties of bladder versus vascular smooth muscle in mice lacking SM-B myosin

Prologue: vascular myogenic mechanisms

Vascular smooth muscle cell stress as a determinant of cerebral artery myogenic tone

Structural remodeling of rat uterine veins in pregnancy

Myoendometrial versus placental uterine arteries: Structural, mechanical, and functional differences in late-pregnant rabbits

Increased Ca2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries

Isoform switching from SM-B to SM-A myosin results in decreased contractility and altered expression of thin filament regulatory proteins

Increased Ca2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries

Vascular smooth muscle actin cytoskeleton in cerebral artery forced dilatation / Editorial comment

Increased Ca 2+ sensitivity as a key mechanism of PKC-induced constriction in pressurized cerebral arteries

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