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Identification of Pathological Conditions in Human Skeletal Remains

Ortner, Donald J

2, 2003

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Voltage-Gated Ca2+ Channels in Dopaminergic Substantia Nigra Neurons: Therapeutic Targets for Neuroprotection in Parkinson's Disease?

Frontiers in synaptic neuroscience, 2021-02, Vol.13

Ortner, Nadine J.

2021

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Pharmacology of L-type Calcium Channels: Novel Drugs for Old Targets?

Current molecular pharmacology, 2015-09, Vol.8 (2), p.110

2015

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Lower Affinity of Isradipine for L-Type Ca2+ Channels during Substantia Nigra Dopamine Neuron-Like Activity: Implications for Neuroprotection in Parkinson's Disease

The Journal of neuroscience, 2017-07, Vol.37 (28), p.6761-6777

2017

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Novel protocol for multiple-dose oral administration of the L-type Ca2+ channel blocker isradipine in mice: A dose-finding pharmacokinetic study

Channels (Austin, Tex.), 2024-12, Vol.18 (1), p.2335469-2335469

2024

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De novo CACAN1D Ca2+ channelopathies: clinical phenotypes and molecular mechanism

Pflügers Archiv, 2020-07, Vol.472 (7), p.755-773

2020

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CACNA1D-Related Channelopathies: From Hypertension to Autism

Voltage-gated Ca2+ Channels: Pharmacology, Modulation and their Role in Human Disease, p.183-225

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Biophysical classification of a CACNA1D de novo mutation as a high-risk mutation for a severe neurodevelopmental disorder

Molecular autism, 2020-01, Vol.11 (1), p.4-18, Article 4

2020

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Childhood scurvy in a medieval burial from Mačvanska Mitrovica, Serbia

International journal of osteoarchaeology, 2011-03, Vol.21 (2), p.197-207

2011

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L-type calcium channels as drug targets in CNS disorders

Channels (Austin, Tex.), 2016-01, Vol.10 (1), p.7-13

2016

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Evidence of probable scurvy in subadults from archeological sites in North America

American journal of physical anthropology, 2001-04, Vol.114 (4), p.343-351

2001

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Ca v 1.3-selective inhibitors of voltage-gated L-type Ca 2+ channels: Fact or (still) fiction?

British journal of pharmacology, 2023-05, Vol.180 (10), p.1289-1303

2023

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Cav1.3‐selective inhibitors of voltage‐gated L‐type Ca2+ channels: Fact or (still) fiction?

British journal of pharmacology, 2023-05, Vol.180 (10), p.1289-1303

2023

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Anemia or scurvy: A pilot study on differential diagnosis of porous and hyperostotic lesions using differential cranial vault thickness in subadult humans

International journal of paleopathology, 2014-06, Vol.5, p.27-33

2014

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Novel protocol for multiple-dose oral administration of the L-type Ca 2+ channel blocker isradipine in mice: A dose-finding pharmacokinetic study

Channels (Austin, Tex.), 2024-12, Vol.18 (1), p.2335469

2024

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Frequency of outpatient care adverse events in an occupational mutual insurance company in Spain

Journal of healthcare quality research, 2021-11, Vol.36 (6), p.340

2021

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A case of treponematosis from pre-Columbian Chaco Canyon, New Mexico

International journal of osteoarchaeology, 2011-01, Vol.21 (1), p.19-31

2011

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Germline de novo variant F747S extends the phenotypic spectrum of CACNA1D Ca2+ channelopathies

Human molecular genetics, 2023-02, Vol.32 (5), p.847-859

2023

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STAC proteins associate to the IQ domain of CaV1.2 and inhibit calcium-dependent inactivation

Proceedings of the National Academy of Sciences - PNAS, 2018-02, Vol.115 (6), p.1376-1381

2018

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Pyrimidine-2,4,6-triones are a new class of voltage-gated L-type Ca2+ channel activators

Nature communications, 2014-06, Vol.5 (1), Article 3897

2014

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Pathogenicity of de novo CACNA1D Ca2+ channel variants predicted from sequence co-variation

European journal of human genetics : EJHG, 2024-03

2024

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Novel CACNA1A Variant p.Cys256Phe Disrupts Disulfide Bonds and Causes Spinocerebellar Ataxia

Movement disorders, 2022-02, Vol.37 (2), p.401-404

2022

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What skeletons tell us. The story of human paleopathology

Virchows Archiv : an international journal of pathology, 2011-09, Vol.459 (3), p.247-254

Ortner, Donald J.

2011

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Correction to: De novo CACNA1D Ca2+ channelopathies: clinical phenotypes and molecular mechanism

Pflügers Archiv, 2020-08, Vol.472 (8), p.1105-1105

2020

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Identification of Pathological Conditions in Human Skeletal Remains

Voltage-Gated Ca2+ Channels in Dopaminergic Substantia Nigra Neurons: Therapeutic Targets for Neuroprotection in Parkinson's Disease?

Pharmacology of L-type Calcium Channels: Novel Drugs for Old Targets?

Lower Affinity of Isradipine for L-Type Ca2+ Channels during Substantia Nigra Dopamine Neuron-Like Activity: Implications for Neuroprotection in Parkinson's Disease

Novel protocol for multiple-dose oral administration of the L-type Ca2+ channel blocker isradipine in mice: A dose-finding pharmacokinetic study

De novo CACAN1D Ca2+ channelopathies: clinical phenotypes and molecular mechanism

CACNA1D-Related Channelopathies: From Hypertension to Autism

Biophysical classification of a CACNA1D de novo mutation as a high-risk mutation for a severe neurodevelopmental disorder

Childhood scurvy in a medieval burial from Mačvanska Mitrovica, Serbia

L-type calcium channels as drug targets in CNS disorders

Evidence of probable scurvy in subadults from archeological sites in North America

Ca v 1.3-selective inhibitors of voltage-gated L-type Ca 2+ channels: Fact or (still) fiction?

Cav1.3‐selective inhibitors of voltage‐gated L‐type Ca2+ channels: Fact or (still) fiction?

The human channel gating-modifying A749G CACNA1D (Cav1.3) variant induces a neurodevelopmental syndrome-like phenotype in mice

Anemia or scurvy: A pilot study on differential diagnosis of porous and hyperostotic lesions using differential cranial vault thickness in subadult humans

Novel protocol for multiple-dose oral administration of the L-type Ca 2+ channel blocker isradipine in mice: A dose-finding pharmacokinetic study

Frequency of outpatient care adverse events in an occupational mutual insurance company in Spain

A case of treponematosis from pre-Columbian Chaco Canyon, New Mexico

Germline de novo variant F747S extends the phenotypic spectrum of CACNA1D Ca2+ channelopathies

STAC proteins associate to the IQ domain of CaV1.2 and inhibit calcium-dependent inactivation

Pyrimidine-2,4,6-triones are a new class of voltage-gated L-type Ca2+ channel activators

Pathogenicity of de novo CACNA1D Ca2+ channel variants predicted from sequence co-variation

Novel CACNA1A Variant p.Cys256Phe Disrupts Disulfide Bonds and Causes Spinocerebellar Ataxia

What skeletons tell us. The story of human paleopathology

Correction to: De novo CACNA1D Ca2+ channelopathies: clinical phenotypes and molecular mechanism

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