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Cellular Hypertrophy and Increased Susceptibility to Spontaneous Calcium-Release of Rat Left Atrial Myocytes Due to Elevated Afterload

PloS one, 2015-12, Vol.10 (12), p.e0144309-e0144309

2015

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Consecutive Isoproterenol and Adenosine Treatment Confers Marked Protection against Reperfusion Injury in Adult but Not in Immature Heart: A Role for Glycogen

International journal of molecular sciences, 2018-02, Vol.19 (2), p.494

2018

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Density and sub-cellular distribution of cardiac and neuronal sodium channel isoforms in rat ventricular myocytes

Biochemical and biophysical research communications, 2006-09, Vol.348 (3), p.1163-1166

2006

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Characterization and influence of cardiac background sodium current in the atrioventricular node

Journal of molecular and cellular cardiology, 2016-08, Vol.97, p.114-124

2016

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A model of the guinea-pig ventricular cardiac myocyte incorporating a transverse–axial tubular system

Progress in biophysics and molecular biology, 2008, Vol.96 (1), p.258-280

2008

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Excitation-contraction coupling in rat ventricular myocytes after formamide-induced detubulation

American journal of physiology. Heart and circulatory physiology, 1999-08, Vol.277 (2), p.H603-H609

1999

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Acidosis Impairs the Protective Role of hERG K+ Channels Against Premature Stimulation

Journal of cardiovascular electrophysiology, 2010-10, Vol.21 (10), p.1160-1169

2010

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Caveolin-3 KO disrupts t-tubule structure and decreases t-tubular I Ca density in mouse ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2018-11, Vol.315 (5), p.H1101-H1111

2018

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Caveolin-3 KO disrupts t-tubule structure and decreases t-tubular ICa density in mouse ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2018-11, Vol.315 (5), p.H1101-H1111

2018

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Altered distribution of ICa impairs Ca release at the t-tubules of ventricular myocytes from failing hearts

Journal of molecular and cellular cardiology, 2015-09, Vol.86, p.23-31

2015

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Ca efflux via the sarcolemmal Ca ATPase occurs only in the t-tubules of rat ventricular myocytes

Journal of molecular and cellular cardiology, 2011-01, Vol.50 (1), p.187-193

2011

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Solute movement in the t-tubule system of rabbit and mouse cardiomyocytes

Proceedings of the National Academy of Sciences - PNAS, 2018-07, Vol.115 (30), p.E7073-E7080

2018

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Acidosis in models of cardiac ventricular myocytes

Philosophical transactions of the Royal Society of London. Series A: Mathematical, physical, and engineering sciences, 2006-05, Vol.364 (1842), p.1171-1186

2006

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Sub-microscopic analysis of t-tubule geometry in living cardiac ventricular myocytes using a shape-based analysis method

Journal of molecular and cellular cardiology, 2017-07, Vol.108, p.1-7

2017

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Loss of caveolin-3-dependent regulation of ICa in rat ventricular myocytes in heart failure

American journal of physiology. Heart and circulatory physiology, 2018-03, Vol.314 (3), p.H521-H529

2018

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Loss of caveolin-3-dependent regulation of I Ca in rat ventricular myocytes in heart failure

American journal of physiology. Heart and circulatory physiology, 2018-03, Vol.314 (3), p.H521-H529

2018

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Altered Na/Ca exchange distribution in ventricular myocytes from failing hearts

American journal of physiology. Heart and circulatory physiology, 2016-01, Vol.310 (2), p.H262-H268

2016

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Caveolin 3-dependent loss of t-tubular I Ca during hypertrophy and heart failure in mice

Experimental physiology, 2018-05, Vol.103 (5), p.652-665

2018

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Caveolin 3‐dependent loss of t‐tubular ICa during hypertrophy and heart failure in mice

Experimental physiology, 2018-05, Vol.103 (5), p.652-665

2018

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Localised Ca channel phosphorylation modulates the distribution of L-type Ca current in cardiac myocytes

Journal of molecular and cellular cardiology, 2010-07, Vol.49 (1), p.121-131

2010

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Reduced density and altered regulation of rat atrial L-type Ca2+ current in heart failure

American journal of physiology. Heart and circulatory physiology, 2017-03, Vol.312 (3), p.H384-H391

2017

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The role of mammalian cardiac t‐tubules in excitation–contraction coupling: experimental and computational approaches

Experimental physiology, 2009-05, Vol.94 (5), p.509-519

2009

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Electrophysiological response of rat ventricular myocytes to acidosis

American journal of physiology. Heart and circulatory physiology, 2002-07, Vol.283 (1), p.H412-H422

2002

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Cardiac-specific overexpression of caveolin-3 preserves t-tubular I Ca during heart failure in mice

Experimental physiology, 2019-05, Vol.104 (5), p.654-666

2019

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Cardiac‐specific overexpression of caveolin‐3 preserves t‐tubular ICa during heart failure in mice

Experimental physiology, 2019-05, Vol.104 (5), p.654-666

2019

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Cellular Hypertrophy and Increased Susceptibility to Spontaneous Calcium-Release of Rat Left Atrial Myocytes Due to Elevated Afterload

Consecutive Isoproterenol and Adenosine Treatment Confers Marked Protection against Reperfusion Injury in Adult but Not in Immature Heart: A Role for Glycogen

Density and sub-cellular distribution of cardiac and neuronal sodium channel isoforms in rat ventricular myocytes

Characterization and influence of cardiac background sodium current in the atrioventricular node

A model of the guinea-pig ventricular cardiac myocyte incorporating a transverse–axial tubular system

Excitation-contraction coupling in rat ventricular myocytes after formamide-induced detubulation

Acidosis Impairs the Protective Role of hERG K+ Channels Against Premature Stimulation

Caveolin-3 KO disrupts t-tubule structure and decreases t-tubular I Ca density in mouse ventricular myocytes

Caveolin-3 KO disrupts t-tubule structure and decreases t-tubular ICa density in mouse ventricular myocytes

Altered distribution of ICa impairs Ca release at the t-tubules of ventricular myocytes from failing hearts

Ca efflux via the sarcolemmal Ca ATPase occurs only in the t-tubules of rat ventricular myocytes

Solute movement in the t-tubule system of rabbit and mouse cardiomyocytes

Acidosis in models of cardiac ventricular myocytes

Sub-microscopic analysis of t-tubule geometry in living cardiac ventricular myocytes using a shape-based analysis method

Loss of caveolin-3-dependent regulation of ICa in rat ventricular myocytes in heart failure

Loss of caveolin-3-dependent regulation of I Ca in rat ventricular myocytes in heart failure

Altered Na/Ca exchange distribution in ventricular myocytes from failing hearts

Caveolin 3-dependent loss of t-tubular I Ca during hypertrophy and heart failure in mice

Caveolin 3‐dependent loss of t‐tubular ICa during hypertrophy and heart failure in mice

Localised Ca channel phosphorylation modulates the distribution of L-type Ca current in cardiac myocytes

Reduced density and altered regulation of rat atrial L-type Ca2+ current in heart failure

The role of mammalian cardiac t‐tubules in excitation–contraction coupling: experimental and computational approaches

Electrophysiological response of rat ventricular myocytes to acidosis

Cardiac-specific overexpression of caveolin-3 preserves t-tubular I Ca during heart failure in mice

Cardiac‐specific overexpression of caveolin‐3 preserves t‐tubular ICa during heart failure in mice

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