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Molecular mechanisms of varicella zoster virus pathogenesis

Nature reviews. Microbiology, 2014-03, Vol.12 (3), p.197-210

2014

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The cytoplasmic domain of varicella-zoster virus glycoprotein H regulates syncytia formation and skin pathogenesis

PLoS pathogens, 2014-05, Vol.10 (5), p.e1004173-e1004173

2014

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Calcineurin phosphatase activity regulates Varicella-Zoster Virus induced cell-cell fusion

PLoS pathogens, 2020-11, Vol.16 (11), p.e1009022-e1009022

2020

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Targeted mutagenesis of the herpesvirus fusogen central helix captures transition states

Nature communications, 2023-12, Vol.14 (1), p.7958-7958, Article 7958

2023

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A glycoprotein B-neutralizing antibody structure at 2.8 Å uncovers a critical domain for herpesvirus fusion initiation

Nature communications, 2020-08, Vol.11 (1), p.4141-15, Article 4141

2020

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The N-terminus of varicella-zoster virus glycoprotein B has a functional role in fusion

PLoS pathogens, 2021-01, Vol.17 (1), p.e1008961

2021

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Disruption of PML nuclear bodies is mediated by ORF61 SUMO-interacting motifs and required for varicella-zoster virus pathogenesis in skin

PLoS pathogens, 2011-08, Vol.7 (8), p.e1002157

2011

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The Structures and Functions of VZV Glycoproteins

Current topics in microbiology and immunology, 2023, Vol.438, p.25-58

2023

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site of varicella-zoster virus vulnerability identified by structural studies of neutralizing antibodies bound to the glycoprotein complex gHgL

Proceedings of the National Academy of Sciences - PNAS, 2015-05, Vol.112 (19), p.6056-6061

2015

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HIV-1 inhibitory properties of eCD4-Igmim2 determined using an Env-mediated membrane fusion assay

PloS one, 2018-10, Vol.13 (10), p.e0206365-e0206365

2018

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Publisher Correction: A glycoprotein B-neutralizing antibody structure at 2.8 Å uncovers a critical domain for herpesvirus fusion initiation

Nature communications, 2020-08, Vol.11 (1), p.4398-4398, Article 4398

2020

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immunoreceptor tyrosine-based inhibition motif in varicella-zoster virus glycoprotein B regulates cell fusion and skin pathogenesis

Proceedings of the National Academy of Sciences - PNAS, 2013-01, Vol.110 (5), p.1911-1916

2013

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Structure–function analysis of varicella-zoster virus glycoprotein H identifies domain-specific roles for fusion and skin tropism

Proceedings of the National Academy of Sciences - PNAS, 2011-11, Vol.108 (45), p.18412-18417

2011

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Mutagenesis of Varicella-Zoster Virus Glycoprotein B: Putative Fusion Loop Residues Are Essential for Viral Replication, and the Furin Cleavage Motif Contributes to Pathogenesis in Skin Tissue In Vivo

Journal of Virology, 2009-08, Vol.83 (15), p.7495-7506

2009

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Varicella-zoster virus: molecular controls of cell fusion-dependent pathogenesis

Biochemical Society transactions, 2020-12, Vol.48 (6), p.2415-2435

2020

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ORF11 Protein Interacts with the ORF9 Essential Tegument Protein in Varicella-Zoster Virus Infection

Journal of Virology, 2013-05, Vol.87 (9), p.5106-5117

2013

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Role for the αV Integrin Subunit in Varicella-Zoster Virus-Mediated Fusion and Infection

Journal of virology, 2016-08, Vol.90 (16), p.7567-7578

2016

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Dysregulated Glycoprotein B-Mediated Cell-Cell Fusion Disrupts Varicella-Zoster Virus and Host Gene Transcription during Infection

Journal of virology, 2017-01, Vol.91 (1)

2017

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Anti-Glycoprotein H Antibody Impairs the Pathogenicity of Varicella-Zoster Virus in Skin Xenografts in the SCID Mouse Model

Journal of Virology, 2010-01, Vol.84 (1), p.141-152

2010

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Human antibody responses to bovine (Newbury-2) norovirus (GIII.2) and association to histo-blood group antigens

Journal of medical virology, 2010-07, Vol.82 (7), p.1241-1246

2010

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The Glycoprotein B Cytoplasmic Domain Lysine Cluster Is Critical for Varicella-Zoster Virus Cell-Cell Fusion Regulation and Infection

Journal of virology, 2017-01, Vol.91 (1)

2017

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Target highlights in CASP14: Analysis of models by structure providers

Proteins, structure, function, and bioinformatics, 2021-12, Vol.89 (12), p.1647-1672

2021

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Mutagenesis of Varicella-Zoster Virus Glycoprotein I (gI) Identifies a Cysteine Residue Critical for gE/gI Heterodimer Formation, gI Structure, and Virulence in Skin Cells

Journal of Virology, 2011-05, Vol.85 (9), p.4095-4110

2011

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Varicella-Zoster Virus: Genetics, Pathogenesis and Immunity

1, 2022

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Varicella-Zoster Virus Glycoproteins: Entry, Replication, and Pathogenesis

Current clinical microbiology reports, 2016-12, Vol.3 (4), p.204-215

2016

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Molecular mechanisms of varicella zoster virus pathogenesis

The cytoplasmic domain of varicella-zoster virus glycoprotein H regulates syncytia formation and skin pathogenesis

Calcineurin phosphatase activity regulates Varicella-Zoster Virus induced cell-cell fusion

Targeted mutagenesis of the herpesvirus fusogen central helix captures transition states

A glycoprotein B-neutralizing antibody structure at 2.8 Å uncovers a critical domain for herpesvirus fusion initiation

The N-terminus of varicella-zoster virus glycoprotein B has a functional role in fusion

Disruption of PML nuclear bodies is mediated by ORF61 SUMO-interacting motifs and required for varicella-zoster virus pathogenesis in skin

The Structures and Functions of VZV Glycoproteins

site of varicella-zoster virus vulnerability identified by structural studies of neutralizing antibodies bound to the glycoprotein complex gHgL

HIV-1 inhibitory properties of eCD4-Igmim2 determined using an Env-mediated membrane fusion assay

Publisher Correction: A glycoprotein B-neutralizing antibody structure at 2.8 Å uncovers a critical domain for herpesvirus fusion initiation

immunoreceptor tyrosine-based inhibition motif in varicella-zoster virus glycoprotein B regulates cell fusion and skin pathogenesis

Structure–function analysis of varicella-zoster virus glycoprotein H identifies domain-specific roles for fusion and skin tropism

Mutagenesis of Varicella-Zoster Virus Glycoprotein B: Putative Fusion Loop Residues Are Essential for Viral Replication, and the Furin Cleavage Motif Contributes to Pathogenesis in Skin Tissue In Vivo

Varicella-zoster virus: molecular controls of cell fusion-dependent pathogenesis

ORF11 Protein Interacts with the ORF9 Essential Tegument Protein in Varicella-Zoster Virus Infection

Role for the αV Integrin Subunit in Varicella-Zoster Virus-Mediated Fusion and Infection

Dysregulated Glycoprotein B-Mediated Cell-Cell Fusion Disrupts Varicella-Zoster Virus and Host Gene Transcription during Infection

Anti-Glycoprotein H Antibody Impairs the Pathogenicity of Varicella-Zoster Virus in Skin Xenografts in the SCID Mouse Model

Human antibody responses to bovine (Newbury-2) norovirus (GIII.2) and association to histo-blood group antigens

The Glycoprotein B Cytoplasmic Domain Lysine Cluster Is Critical for Varicella-Zoster Virus Cell-Cell Fusion Regulation and Infection

Target highlights in CASP14: Analysis of models by structure providers

Mutagenesis of Varicella-Zoster Virus Glycoprotein I (gI) Identifies a Cysteine Residue Critical for gE/gI Heterodimer Formation, gI Structure, and Virulence in Skin Cells

Varicella-Zoster Virus: Genetics, Pathogenesis and Immunity

Varicella-Zoster Virus Glycoproteins: Entry, Replication, and Pathogenesis

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