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Early alteration of epigenetic-related transcription in Huntington's disease mouse models

Scientific reports, 2018-07, Vol.8 (1), p.9925-14, Article 9925

2018

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CBP and SRF co-regulate dendritic growth and synaptic maturation

Cell death and differentiation, 2019-11, Vol.26 (11), p.2208-2222

2019

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The Epigenetic Factor CBP Is Required for the Differentiation and Function of Medial Ganglionic Eminence-Derived Interneurons

Molecular neurobiology, 2019-06, Vol.56 (6), p.4440-4454

2019

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CBP and SRF co-regulate dendritic growth and synaptic maturation

Cell death and differentiation, 2019-11, Vol.26 (11), p.2208-2222

2019

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Plasmalemmal sodium-calcium exchanger shapes the calcium and exocytotic signals of chromaffin cells at physiological temperature

American Journal of Physiology: Cell Physiology, 2013-07, Vol.305 (2), p.C160-C172

2013

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Loss of Kdm5c Causes Spurious Transcription and Prevents the Fine-Tuning of Activity-Regulated Enhancers in Neurons

Cell reports (Cambridge), 2017-10, Vol.21 (1), p.47-59

2017

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KAT3-dependent acetylation of cell type-specific genes maintains neuronal identity in the adult mouse brain

Nature communications, 2020-05, Vol.11 (1), p.2588-2588, Article 2588

2020

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Histone acetylation and CREB binding protein are required for neuronal resistance against ischemic injury

PloS one, 2014-04, Vol.9 (4), p.e95465

2014

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cAMP Response Element-Binding Protein-Mediated Gene Expression Increases the Intrinsic Excitability of CA1 Pyramidal Neurons

The Journal of neuroscience, 2007-12, Vol.27 (50), p.13909-13918

2007

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Unmasking the functions of the chromaffin cell α7 nicotinic receptor by using short pulses of acetylcholine and selective blockers

Proceedings of the National Academy of Sciences - PNAS, 1998-11, Vol.95 (24), p.14184-14189

1998

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Syndromic features and mild cognitive impairment in mice with genetic reduction on p300 activity: Differential contribution of p300 and CBP to Rubinstein–Taybi syndrome etiology

Neurobiology of disease, 2010-01, Vol.37 (1), p.186-194

2010

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Blockade of nicotinic receptors of bovine adrenal chromaffin cells by nanomolar concentrations of atropine

European journal of pharmacology, 2006-03, Vol.535 (1), p.13-24

2006

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Acetylcholine and potassium elicit different patterns of exocytosis in chromaffin cells when the intracellular calcium handling is disturbed

Pflügers Archiv, 2002-05, Vol.444 (1-2), p.133-142

2002

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Control of secretion by mitochondria depends on the size of the local [Ca2+] after chromaffin cell stimulation

The European journal of neuroscience, 2001-06, Vol.13 (12), p.2247-2254

2001

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Blockade by agmatine of catecholamine release from chromaffin cells is unrelated to imidazoline receptors

European journal of pharmacology, 2001-04, Vol.417 (1), p.99-109

2001

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Blockade of Ca2+-activated K+ channels by galantamine can also contribute to the potentiation of catecholamine secretion from chromaffin cells

European journal of pharmacology, 2006-10, Vol.548 (1-3), p.45-52

2006

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Ablation of CBP in forebrain principal neurons causes modest memory and transcriptional defects and a dramatic reduction of histone acetylation but does not affect cell viability

The Journal of neuroscience, 2011-02, Vol.31 (5), p.1652-1663

2011

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Inhibition of cAMP Response Element-Binding Protein Reduces Neuronal Excitability and Plasticity, and Triggers Neurodegeneration

Cerebral cortex (New York, N.Y. 1991), 2009-11, Vol.19 (11), p.2535-2547

2009

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Actuación de un servicio de prevención de riesgos laborales para la atención a casos sospechosos de enfermedad respiratoria por coronavirus (MERS-CoV): A propósito de un caso

Medicina y Seguridad del Trabajo, 2017-06, Vol.63 (247), p.93-102

2017

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Presentación

Boletín ICE económico, 2018-03 (3097)

Escolano Olivares, Román

2018

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Presentación

Boletín ICE económico, 2018-03, Vol.3097, p.3-4

Escolano Olivares, Román

2018

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Loss of neuronal 3D chromatin organization causes transcriptional and behavioural deficits related to serotonergic dysfunction

Nature communications, 2014-07, Vol.5 (1), p.4450-4450, Article 4450

2014

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A choline‐evoked [Ca2+]C signal causes catecholamine release and hyperpolarization of chromaffin cells

The FASEB journal, 2004-09, Vol.18 (12), p.1468-1470

2004

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Blockade of Ca 2+-activated K + channels by galantamine can also contribute to the potentiation of catecholamine secretion from chromaffin cells

European journal of pharmacology, 2006, Vol.548 (1), p.45-52

2006

Volltextzugriff (PDF)

Public banks during the Great Recession: the case of Instituto de Crédito Oficial in Spain

Información comercial española, 2014-07, Vol.879, p.65-74

Escolano Olivares, Román

2014

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Early alteration of epigenetic-related transcription in Huntington's disease mouse models

CBP and SRF co-regulate dendritic growth and synaptic maturation

The Epigenetic Factor CBP Is Required for the Differentiation and Function of Medial Ganglionic Eminence-Derived Interneurons

CBP and SRF co-regulate dendritic growth and synaptic maturation

Plasmalemmal sodium-calcium exchanger shapes the calcium and exocytotic signals of chromaffin cells at physiological temperature

Loss of Kdm5c Causes Spurious Transcription and Prevents the Fine-Tuning of Activity-Regulated Enhancers in Neurons

KAT3-dependent acetylation of cell type-specific genes maintains neuronal identity in the adult mouse brain

Histone acetylation and CREB binding protein are required for neuronal resistance against ischemic injury

cAMP Response Element-Binding Protein-Mediated Gene Expression Increases the Intrinsic Excitability of CA1 Pyramidal Neurons

Unmasking the functions of the chromaffin cell α7 nicotinic receptor by using short pulses of acetylcholine and selective blockers

Syndromic features and mild cognitive impairment in mice with genetic reduction on p300 activity: Differential contribution of p300 and CBP to Rubinstein–Taybi syndrome etiology

Blockade of nicotinic receptors of bovine adrenal chromaffin cells by nanomolar concentrations of atropine

Acetylcholine and potassium elicit different patterns of exocytosis in chromaffin cells when the intracellular calcium handling is disturbed

Control of secretion by mitochondria depends on the size of the local [Ca2+] after chromaffin cell stimulation

Blockade by agmatine of catecholamine release from chromaffin cells is unrelated to imidazoline receptors

Blockade of Ca2+-activated K+ channels by galantamine can also contribute to the potentiation of catecholamine secretion from chromaffin cells

Ablation of CBP in forebrain principal neurons causes modest memory and transcriptional defects and a dramatic reduction of histone acetylation but does not affect cell viability

Inhibition of cAMP Response Element-Binding Protein Reduces Neuronal Excitability and Plasticity, and Triggers Neurodegeneration

Actuación de un servicio de prevención de riesgos laborales para la atención a casos sospechosos de enfermedad respiratoria por coronavirus (MERS-CoV): A propósito de un caso

Presentación

Presentación

Loss of neuronal 3D chromatin organization causes transcriptional and behavioural deficits related to serotonergic dysfunction

A choline‐evoked [Ca2+]C signal causes catecholamine release and hyperpolarization of chromaffin cells

Blockade of Ca 2+-activated K + channels by galantamine can also contribute to the potentiation of catecholamine secretion from chromaffin cells

Public banks during the Great Recession: the case of Instituto de Crédito Oficial in Spain

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