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The updated biology of hypoxia-inducible factor

The EMBO journal, 2012-05, Vol.31 (11), p.2448-2460

2012

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Cathepsins L and B target HIF1α for oxygen-independent proteolytic cleavage

Scientific reports, 2024-06, Vol.14 (1), p.14799-9, Article 14799

2024

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Inhibition of SHP2-mediated dephosphorylation of Ras suppresses oncogenesis

Nature communications, 2015-11, Vol.6 (1), p.8859-8859, Article 8859

2015

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Evolution of metazoan oxygen-sensing involved a conserved divergence of VHL affinity for HIF1α and HIF2α

Nature communications, 2019-07, Vol.10 (1), p.3293-12, Article 3293

2019

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NRAS Status Determines Sensitivity to SHP2 Inhibitor Combination Therapies Targeting the RAS-MAPK Pathway in Neuroblastoma

Cancer research (Chicago, Ill.), 2020-08, Vol.80 (16), p.3413-3423

2020

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Ubiquitin Pathway in VHL Cancer Syndrome

Neoplasia (New York, N.Y.), 2006-08, Vol.8 (8), p.623-629

Ohh, Michael

2006

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Hypoxia-inducible factor underlies von Hippel-Lindau disease stigmata

eLife, 2022-08, Vol.11

2022

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HIF-2α-pVHL complex reveals broad genotype-phenotype correlations in HIF-2α-driven disease

Nature communications, 2018-08, Vol.9 (1), p.3359-11, Article 3359

2018

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Tyrosyl phosphorylation of KRAS stalls GTPase cycle via alteration of switch I and II conformation

Nature communications, 2019-01, Vol.10 (1), p.224-224, Article 224

2019

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PD-1 blockade delays tumor growth by inhibiting an intrinsic SHP2/Ras/MAPK signalling in thyroid cancer cells

Journal of experimental & clinical cancer research, 2021-01, Vol.40 (1), p.22-22, Article 22

2021

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Src promotes GTPase activity of Ras via tyrosine 32 phosphorylation

Proceedings of the National Academy of Sciences - PNAS, 2014-09, Vol.111 (36), p.E3785-E3794

2014

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The Q61H mutation decouples KRAS from upstream regulation and renders cancer cells resistant to SHP2 inhibitors

Nature communications, 2021-11, Vol.12 (1), p.6274-6274, Article 6274

2021

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Hypoxia promotes ligand-independent EGF receptor signaling via hypoxia-inducible factor–mediated upregulation of caveolin-1

Proceedings of the National Academy of Sciences - PNAS, 2012-03, Vol.109 (13), p.4892-4897

2012

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Functional Importance of Dicer Protein in the Adaptive Cellular Response to Hypoxia

The Journal of biological chemistry, 2012-08, Vol.287 (34), p.29003-29020

2012

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SOCS-1 mediates ubiquitylation and degradation of GM-CSF receptor

PloS one, 2013-09, Vol.8 (9), p.e76370-e76370

2013

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Hypoxia and viral infectious diseases

JCI insight, 2021-04, Vol.6 (7)

2021

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Human HIF‐3α4 is a dominant‐negative regulator of HIF‐1 and is down‐regulated in renal cell carcinoma

The FASEB journal, 2005-09, Vol.19 (11), p.1396-1406

2005

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Consequences of VHL Loss on Global DNA Methylome

Scientific reports, 2018-02, Vol.8 (1), p.3313-11, Article 3313

2018

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Characterization of a von hippel lindau pathway involved in extracellular matrix remodeling, cell invasion, and angiogenesis

Cancer research (Chicago, Ill.), 2006-02, Vol.66 (3), p.1313-1319

2006

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VHL Promotes E2 Box-Dependent E-Cadherin Transcription by HIF-Mediated Regulation of SIP1 and Snail

Molecular and Cellular Biology, 2007-01, Vol.27 (1), p.157-169

2007

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Hypoxia-inducible Expression of a Natural cis-Antisense Transcript Inhibits Endothelial Nitric-oxide Synthase

The Journal of biological chemistry, 2007-05, Vol.282 (21), p.15652-15666

2007

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NEDD8 acts as a 'molecular switch' defining the functional selectivity of VHL

EMBO reports, 2008-05, Vol.9 (5), p.486-491

2008

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Deficiency in PHD2-mediated hydroxylation of HIF2α underlies Pacak-Zhuang syndrome

Communications biology, 2024-02, Vol.7 (1), p.240-240, Article 240

2024

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Loss of JAK2 regulation via a heterodimeric VHL-SOCS1 E3 ubiquitin ligase underlies Chuvash polycythemia

Nature medicine, 2011-07, Vol.17 (7), p.845-853

2011

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Dominant-Negative HIF-3α4 Suppresses VHL-Null Renal Cell Carcinoma Progression

Cell cycle (Georgetown, Tex.), 2007-11, Vol.6 (22), p.2810-2816

2007

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The updated biology of hypoxia-inducible factor

Cathepsins L and B target HIF1α for oxygen-independent proteolytic cleavage

Inhibition of SHP2-mediated dephosphorylation of Ras suppresses oncogenesis

Evolution of metazoan oxygen-sensing involved a conserved divergence of VHL affinity for HIF1α and HIF2α

NRAS Status Determines Sensitivity to SHP2 Inhibitor Combination Therapies Targeting the RAS-MAPK Pathway in Neuroblastoma

Ubiquitin Pathway in VHL Cancer Syndrome

Hypoxia-inducible factor underlies von Hippel-Lindau disease stigmata

HIF-2α-pVHL complex reveals broad genotype-phenotype correlations in HIF-2α-driven disease

Tyrosyl phosphorylation of KRAS stalls GTPase cycle via alteration of switch I and II conformation

PD-1 blockade delays tumor growth by inhibiting an intrinsic SHP2/Ras/MAPK signalling in thyroid cancer cells

Src promotes GTPase activity of Ras via tyrosine 32 phosphorylation

The Q61H mutation decouples KRAS from upstream regulation and renders cancer cells resistant to SHP2 inhibitors

Hypoxia promotes ligand-independent EGF receptor signaling via hypoxia-inducible factor–mediated upregulation of caveolin-1

Functional Importance of Dicer Protein in the Adaptive Cellular Response to Hypoxia

SOCS-1 mediates ubiquitylation and degradation of GM-CSF receptor

Hypoxia and viral infectious diseases

Human HIF‐3α4 is a dominant‐negative regulator of HIF‐1 and is down‐regulated in renal cell carcinoma

Consequences of VHL Loss on Global DNA Methylome

Characterization of a von hippel lindau pathway involved in extracellular matrix remodeling, cell invasion, and angiogenesis

VHL Promotes E2 Box-Dependent E-Cadherin Transcription by HIF-Mediated Regulation of SIP1 and Snail

Hypoxia-inducible Expression of a Natural cis-Antisense Transcript Inhibits Endothelial Nitric-oxide Synthase

NEDD8 acts as a 'molecular switch' defining the functional selectivity of VHL

Deficiency in PHD2-mediated hydroxylation of HIF2α underlies Pacak-Zhuang syndrome

Loss of JAK2 regulation via a heterodimeric VHL-SOCS1 E3 ubiquitin ligase underlies Chuvash polycythemia

Dominant-Negative HIF-3α4 Suppresses VHL-Null Renal Cell Carcinoma Progression

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