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Deletion of the mouse Slc30a8 gene encoding zinc transporter-8 results in impaired insulin secretion

Biochemical journal, 2009-07, Vol.421 (3), p.371-376

2009

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Identification of structural motifs critical for human G6PC2 function informed by sequence analysis and an AlphaFold2-predicted model

Bioscience reports, 2024-01, Vol.44 (1), p.1

2024

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G6PC2: A Negative Regulator of Basal Glucose-Stimulated Insulin Secretion

Diabetes (New York, N.Y.), 2013-05, Vol.62 (5), p.1547-1556

2013

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Functional Analysis of Mouse G6pc1 Mutations Using a Novel In Situ Assay for Glucose-6-Phosphatase Activity and the Effect of Mutations in Conserved Human G6PC1/G6PC2 Amino Acids on G6PC2 Protein Expression

PloS one, 2016-09, Vol.11 (9), p.e0162439

2016

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Pancreatic islet beta cell-specific deletion of G6pc2 reduces fasting blood glucose

Journal of molecular endocrinology, 2020-05, Vol.64 (4), p.235-248

2020

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G6PC2 confers protection against hypoglycemia upon ketogenic diet feeding and prolonged fasting

Molecular metabolism (Germany), 2020-11, Vol.41, p.101043-101043, Article 101043

2020

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The Glucose-6-Phosphatase Catalytic Subunit Gene Promoter Contains Both Positive and Negative Glucocorticoid Response Elements

Molecular endocrinology (Baltimore, Md.), 2005-12, Vol.19 (12), p.3001-3022

2005

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The pancreatic islet β-cell-enriched transcription factor Pdx-1 regulates Slc30a8 gene transcription through an intronic enhancer

Biochemical journal, 2011-01, Vol.433 (1), p.95-105

2011

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Biophysical and functional properties of purified glucose-6-phosphatase catalytic subunit 1

The Journal of biological chemistry, 2022-01, Vol.298 (1), p.101520-101520, Article 101520

2022

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G6PC1 and G6PC2 influence G6P flux but not HSD11B1 activity

Journal of molecular endocrinology, 2023-11, Vol.71 (4)

2023

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Nonsynonymous single-nucleotide polymorphisms in the G6PC2 gene affect protein expression, enzyme activity, and fasting blood glucose

The Journal of biological chemistry, 2022-02, Vol.298 (2), p.101534-101534, Article 101534

2022

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Biochemical and metabolic characterization of a G6PC2 inhibitor

Biochimie, 2024-07, Vol.222, p.109-122

2024

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An Enhancer Within Abcb11 Regulates G6pc2 in C57BL/6 Mouse Pancreatic Islets

Diabetes (New York, N.Y.), 2023-11, Vol.72 (11), p.1621-1628

2023

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Glucose-6-phosphatase catalytic subunit 2 negatively regulates glucose oxidation and insulin secretion in pancreatic β-cells

The Journal of biological chemistry, 2022-04, Vol.298 (4), p.101729-101729, Article 101729

2022

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Characterization of the human SLC30A8 promoter and intronic enhancer

Journal of molecular endocrinology, 2011-12, Vol.47 (3), p.251-259

2011

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Potential positive and negative consequences of ZnT8 inhibition

Journal of endocrinology, 2020-08, Vol.246 (2), p.189-205

2020

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The Three Insulin Response Sequences in the Glucose-6-phosphatase Catalytic Subunit Gene Promoter Are Functionally Distinct

The Journal of biological chemistry, 2003-04, Vol.278 (14), p.11782-11793

2003

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The Diabetes Susceptibility Gene SLC30A8 that Encodes the Zinc Transporter ZnT8 is a Pseudogene in Guinea Pigs Potentially Contributing to Low Guinea Pig Islet Zinc Content

Journal of molecular evolution, 2018-12, Vol.86 (9), p.613-617

2018

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Long-Range Enhancers Are Required to Maintain Expression of the Autoantigen Islet-Specific Glucose-6-Phosphatase Catalytic Subunit–Related Protein in Adult Mouse Islets In Vivo

Diabetes (New York, N.Y.), 2008-01, Vol.57 (1), p.133-141

2008

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Evidence that Evolution of the Diabetes Susceptibility Gene SLC30A8 that Encodes the Zinc Transporter ZnT8 Drives Variations in Pancreatic Islet Zinc Content in Multiple Species

Journal of molecular evolution, 2019-07, Vol.87 (4-6), p.147-151

2019

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Foxa2 and MafA regulate islet-specific glucose-6-phosphatase catalytic subunit-related protein gene expression

Journal of molecular endocrinology, 2008-11, Vol.41 (5), p.315-328

2008

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G6PC2 Modulates the Effects of Dexamethasone on Fasting Blood Glucose and Glucose Tolerance

Endocrinology (Philadelphia), 2016-11, Vol.157 (11), p.4133-4145

2016

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G6PC2 Modulates Fasting Blood Glucose In Male Mice in Response to Stress

Endocrinology (Philadelphia), 2016-08, Vol.157 (8), p.3002-3008

2016

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Insulin and epidermal growth factor suppress basal glucose-6-phosphatase catalytic subunit gene transcription through overlapping but distinct mechanisms

Biochemical journal, 2009-01, Vol.417 (2), p.611-620

2009

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Upstream stimulatory factor (USF) and neurogenic differentiation/beta-cell E box transactivator 2 (NeuroD/BETA2) contribute to islet-specific glucose-6-phosphatase catalytic-subunit-related protein (IGRP) gene expression

Biochemical journal, 2003-05, Vol.371 (Pt 3), p.675-686

2003

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Deletion of the mouse Slc30a8 gene encoding zinc transporter-8 results in impaired insulin secretion

Identification of structural motifs critical for human G6PC2 function informed by sequence analysis and an AlphaFold2-predicted model

G6PC2: A Negative Regulator of Basal Glucose-Stimulated Insulin Secretion

Functional Analysis of Mouse G6pc1 Mutations Using a Novel In Situ Assay for Glucose-6-Phosphatase Activity and the Effect of Mutations in Conserved Human G6PC1/G6PC2 Amino Acids on G6PC2 Protein Expression

Pancreatic islet beta cell-specific deletion of G6pc2 reduces fasting blood glucose

G6PC2 confers protection against hypoglycemia upon ketogenic diet feeding and prolonged fasting

The Glucose-6-Phosphatase Catalytic Subunit Gene Promoter Contains Both Positive and Negative Glucocorticoid Response Elements

The pancreatic islet β-cell-enriched transcription factor Pdx-1 regulates Slc30a8 gene transcription through an intronic enhancer

Biophysical and functional properties of purified glucose-6-phosphatase catalytic subunit 1

G6PC1 and G6PC2 influence G6P flux but not HSD11B1 activity

Nonsynonymous single-nucleotide polymorphisms in the G6PC2 gene affect protein expression, enzyme activity, and fasting blood glucose

Biochemical and metabolic characterization of a G6PC2 inhibitor

An Enhancer Within Abcb11 Regulates G6pc2 in C57BL/6 Mouse Pancreatic Islets

Glucose-6-phosphatase catalytic subunit 2 negatively regulates glucose oxidation and insulin secretion in pancreatic β-cells

Characterization of the human SLC30A8 promoter and intronic enhancer

Potential positive and negative consequences of ZnT8 inhibition

The Three Insulin Response Sequences in the Glucose-6-phosphatase Catalytic Subunit Gene Promoter Are Functionally Distinct

The Diabetes Susceptibility Gene SLC30A8 that Encodes the Zinc Transporter ZnT8 is a Pseudogene in Guinea Pigs Potentially Contributing to Low Guinea Pig Islet Zinc Content

Long-Range Enhancers Are Required to Maintain Expression of the Autoantigen Islet-Specific Glucose-6-Phosphatase Catalytic Subunit–Related Protein in Adult Mouse Islets In Vivo

Evidence that Evolution of the Diabetes Susceptibility Gene SLC30A8 that Encodes the Zinc Transporter ZnT8 Drives Variations in Pancreatic Islet Zinc Content in Multiple Species

Foxa2 and MafA regulate islet-specific glucose-6-phosphatase catalytic subunit-related protein gene expression

G6PC2 Modulates the Effects of Dexamethasone on Fasting Blood Glucose and Glucose Tolerance

G6PC2 Modulates Fasting Blood Glucose In Male Mice in Response to Stress

Insulin and epidermal growth factor suppress basal glucose-6-phosphatase catalytic subunit gene transcription through overlapping but distinct mechanisms

Upstream stimulatory factor (USF) and neurogenic differentiation/beta-cell E box transactivator 2 (NeuroD/BETA2) contribute to islet-specific glucose-6-phosphatase catalytic-subunit-related protein (IGRP) gene expression

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