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Mechanisms of atrial structural changes caused by stretch occurring before and during early atrial fibrillation

Cardiovascular research, 2011-03, Vol.89 (4), p.754-765

2011

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Comparative pharmacology of human beta-adrenergic receptor subtypes--characterization of stably transfected receptors in CHO cells

Naunyn-Schmiedeberg's archives of pharmacology, 2004-02, Vol.369 (2), p.151-159

2004

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The transcriptional repressor Nab1 is a specific regulator of pathological cardiac hypertrophy

Nature medicine, 2005-08, Vol.11 (8), p.837-844

2005

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Combined SSCP and Heteroduplex Analysis of the Human Plasma Membrane Ca2+-ATPase Isoform 1 in Patients with Essential Hypertension

Biochemical and biophysical research communications, 1999-08, Vol.261 (2), p.515-520

1999

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Combined SSCP and heteroduplex analysis of the human plasma membrane Ca(2+)-ATPase isoform 1 in patients with essential hypertension

Biochemical and biophysical research communications, 1999-08, Vol.261 (2), p.515-520

1999

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Comparative pharmacology of human -adrenergic receptor subtypes?characterization of stably transfected receptors in CHO cells

Naunyn-Schmiedeberg's archives of pharmacology, 2004-02, Vol.369 (2), p.151-159

2004

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Hypertrophy, fibrosis, and sudden cardiac death in response to pathological stimuli in mice with mutations in cardiac troponin T

Circulation (New York, N.Y.), 2004-10, Vol.110 (15), p.2102-2109

2004

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Overexpression of the Sarcolemmal Calcium Pump in the Myocardium of Transgenic Rats

Circulation research, 1998-11, Vol.83 (9), p.877-888

1998

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Rational promoter selection for gene transfer into cardiac cells

Journal of molecular and cellular cardiology, 2003-07, Vol.35 (7), p.823-831

2003

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Expression of the Plasma Membrane Ca2+-ATPase in Myogenic Cells

The Journal of biological chemistry, 1996-11, Vol.271 (48), p.30816-30822

1996

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Expression of the plasma membrane Ca super(2+)-ATPase in myogenic cells

The Journal of biological chemistry, 1996-01, Vol.271 (48), p.80816-80822

1996

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Sodium–glucose co‐transporter 2 inhibition with empagliflozin improves cardiac function in non‐diabetic rats with left ventricular dysfunction after myocardial infarction

European journal of heart failure, 2019-07, Vol.21 (7), p.862-873

2019

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Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

International journal of molecular sciences, 2021-11, Vol.22 (21), p.11906

2021

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The role of cathepsin D in the pathophysiology of heart failure and its potentially beneficial properties: a translational approach

European journal of heart failure, 2020-11, Vol.22 (11), p.2102-2111

2020

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ATPase Inhibitory Factor-1 Disrupts Mitochondrial Ca2+ Handling and Promotes Pathological Cardiac Hypertrophy through CaMKIIδ

International journal of molecular sciences, 2021-05, Vol.22 (9), p.4427

2021

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Pirfenidone ameliorates pulmonary arterial pressure and neointimal remodeling in experimental pulmonary arterial hypertension by suppressing NLRP3 inflammasome activation

Pulmonary circulation, 2022-07, Vol.12 (3), p.e12101-n/a

2022

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A Kinase Interacting Protein 1 (AKIP1) promotes cardiomyocyte elongation and physiological cardiac remodelling

Scientific reports, 2023-03, Vol.13 (1), p.4046-4046, Article 4046

2023

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Cyclical stretch induces structural changes in atrial myocytes

Journal of cellular and molecular medicine, 2013-06, Vol.17 (6), p.743-753

2013

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Effects of Sodium–Glucose Co-transporter 2 Inhibition with Empaglifozin on Renal Structure and Function in Non-diabetic Rats with Left Ventricular Dysfunction After Myocardial Infarction

Cardiovascular drugs and therapy, 2020-06, Vol.34 (3), p.311-321

2020

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Atrial Structural Remodeling in a Model of Ventricular Pressure Overload

Heart rhythm, 2010, Vol.7 (11), p.1710-1711

2010

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Pirfenidon Prevents Experimental Flow-Associated PAH by Suppression of the NLRP3 Inflammasome

The Journal of heart and lung transplantation, 2022-04, Vol.41 (4), p.S27-S27

2022

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ATPase Inhibitory Factor-1 Disrupts Mitochondrial Ca 2+ Handling and Promotes Pathological Cardiac Hypertrophy through CaMKIIδ

International journal of molecular sciences, 2021-04, Vol.22 (9)

2021

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Mechanisms of atrial structural changes caused by stretch occurring before and during early atrial fibrillation: THE MOLECULAR BASIS OF ATRIAL FIBRILLATION

Cardiovascular research, 2011, Vol.89 (4), p.754-765

2011

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Abstract 14806: A Kinase Interacting Protein 1 (AKIP1) Promotes Physiological Cardiac Remodeling

Circulation (New York, N.Y.), 2019-11, Vol.140 (Suppl_1 Suppl 1), p.A14806-A14806

2019

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Mechanisms of atrial structural changes caused by stretch occurring before and during early atrial fibrillation

Comparative pharmacology of human beta-adrenergic receptor subtypes--characterization of stably transfected receptors in CHO cells

The transcriptional repressor Nab1 is a specific regulator of pathological cardiac hypertrophy

Combined SSCP and Heteroduplex Analysis of the Human Plasma Membrane Ca2+-ATPase Isoform 1 in Patients with Essential Hypertension

Combined SSCP and heteroduplex analysis of the human plasma membrane Ca(2+)-ATPase isoform 1 in patients with essential hypertension

Comparative pharmacology of human -adrenergic receptor subtypes?characterization of stably transfected receptors in CHO cells

Hypertrophy, fibrosis, and sudden cardiac death in response to pathological stimuli in mice with mutations in cardiac troponin T

Overexpression of the Sarcolemmal Calcium Pump in the Myocardium of Transgenic Rats

Rational promoter selection for gene transfer into cardiac cells

Expression of the Plasma Membrane Ca2+-ATPase in Myogenic Cells

Expression of the plasma membrane Ca super(2+)-ATPase in myogenic cells

Sodium–glucose co‐transporter 2 inhibition with empagliflozin improves cardiac function in non‐diabetic rats with left ventricular dysfunction after myocardial infarction

Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

The role of cathepsin D in the pathophysiology of heart failure and its potentially beneficial properties: a translational approach

ATPase Inhibitory Factor-1 Disrupts Mitochondrial Ca2+ Handling and Promotes Pathological Cardiac Hypertrophy through CaMKIIδ

Pirfenidone ameliorates pulmonary arterial pressure and neointimal remodeling in experimental pulmonary arterial hypertension by suppressing NLRP3 inflammasome activation

A Kinase Interacting Protein 1 (AKIP1) promotes cardiomyocyte elongation and physiological cardiac remodelling

Cyclical stretch induces structural changes in atrial myocytes

Effects of Sodium–Glucose Co-transporter 2 Inhibition with Empaglifozin on Renal Structure and Function in Non-diabetic Rats with Left Ventricular Dysfunction After Myocardial Infarction

Atrial Structural Remodeling in a Model of Ventricular Pressure Overload

Pirfenidon Prevents Experimental Flow-Associated PAH by Suppression of the NLRP3 Inflammasome

Disruption of TUFT1, a Desmosome-Associated Protein, Causes Skin Fragility, Woolly Hair, and Palmoplantar Keratoderma

ATPase Inhibitory Factor-1 Disrupts Mitochondrial Ca 2+ Handling and Promotes Pathological Cardiac Hypertrophy through CaMKIIδ

Mechanisms of atrial structural changes caused by stretch occurring before and during early atrial fibrillation: THE MOLECULAR BASIS OF ATRIAL FIBRILLATION

Abstract 14806: A Kinase Interacting Protein 1 (AKIP1) Promotes Physiological Cardiac Remodeling

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