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Enduring glucocorticoid-evoked exacerbation of synaptic plasticity disruption in male rats modelling early Alzheimer's disease amyloidosis

Neuropsychopharmacology (New York, N.Y.), 2021-11, Vol.46 (12), p.2170-2179

2021

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Soluble tau aggregates inhibit synaptic long-term depression and amyloid β-facilitated LTD in vivo

Neurobiology of disease, 2019-07, Vol.127, p.582-590

2019

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PATIENT BRAIN-DERIVED SOLUBLE TAU RAPIDLY AND PERSISTENTLY DISRUPTS SYNAPTIC PLASTICITY IN VIVO

IBRO neuroscience reports, 2023-10, Vol.15, p.S468-S468

2023

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Removal of synaptic Ca²+-permeable AMPA receptors during sleep

The Journal of neuroscience, 2011-03, Vol.31 (11), p.3953-3961

2011

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Pre-plaque Aß-Mediated Impairment of Synaptic Depotentiation in a Transgenic Rat Model of Alzheimer's Disease Amyloidosis

Frontiers in neuroscience, 2019-08, Vol.13, p.861-861

2019

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Do tau-synaptic long-term depression interactions in the hippocampus play a pivotal role in the progression of Alzheimer's disease?

Neural regeneration research, 2023-06, Vol.18 (6), p.1213-1219

2023

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Rapidly reversible persistent long-term potentiation inhibition by patient-derived brain tau and amyloid ß proteins

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2024-07, Vol.379 (1906)

2024

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Cellular Prion Protein Mediates the Disruption of Hippocampal Synaptic Plasticity by Soluble Tau In Vivo

The Journal of neuroscience, 2018-12, Vol.38 (50), p.10595-10606

2018

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Glutamate receptors in preclinical research on Alzheimer's disease: Update on recent advances

Pharmacology, biochemistry and behavior, 2012-02, Vol.100 (4), p.855-862

2012

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Gamma‐patterned sensory stimulation reverses synaptic plasticity deficits in rat models of early Alzheimer's disease

The European journal of neuroscience, 2023-09, Vol.58 (6), p.3402-3411

2023

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Tau and Amyloid β Protein in Patient-Derived Aqueous Brain Extracts Act Concomitantly to Disrupt Long-Term Potentiation in Vivo

The Journal of neuroscience, 2023-08, Vol.43 (32), p.5870-5879

2023

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Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system on the inhibition of hippocampal LTP by Aß

Hippocampus, 2016-12, Vol.26 (12), p.1655-1665

2016

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Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system x c - on the inhibition of hippocampal LTP by Aß

Hippocampus, 2016-12, Vol.26 (12), p.1655

2016

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Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system [Formulaomitted] on the inhibition of hippocampal LTP by As

Hippocampus, 2016-12, Vol.26 (12), p.1655-1665

2016

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Targeting glutamatergic and cellular prion protein mechanisms of amyloid β-mediated persistent synaptic plasticity disruption: Longitudinal studies

Neuropharmacology, 2017-07, Vol.121, p.231-246

2017

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Glucocorticoids, synaptic plasticity and Alzheimer's disease

Current opinion in endocrine and metabolic research, 2022-08, Vol.25, p.100365, Article 100365

2022

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Long-Term Depression-Inducing Low Frequency Stimulation Enhances p-Tau181 and p-Tau217 in an Age-Dependent Manner in Live Rats

Journal of Alzheimer's disease, 2022-01, Vol.89 (1), p.335-350

2022

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Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system xc− on the inhibition of hippocampal LTP by Aß

Hippocampus, 2016-12, Vol.26 (12), p.1655-1665

2016

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Alzheimer’s Disease Amyloid β-Protein and Synaptic Function

Neuromolecular medicine, 2010-03, Vol.12 (1), p.13-26

2010

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Neurotransmitter receptor and time dependence of the synaptic plasticity disrupting actions of Alzheimer's disease Aβ in vivo

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2014-01, Vol.369 (1633), p.20130147

2014

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A human monoclonal IgG that binds aβ assemblies and diverse amyloids exhibits anti-amyloid activities in vitro and in vivo

The Journal of neuroscience, 2015-04, Vol.35 (16), p.6265-6276

2015

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MEDI1814, A HIGH-AFFINITY ANTIBODY DIRECTED TO THE C-TERMINUS OF AβX-42, ABROGATES SYNAPTIC PLASTICITY DISRUPTION CAUSED BY SYNTHETIC OR AD BRAIN-DERIVED Aβ SOLUBLE ASSEMBLIES IN VIVO

Alzheimer's & dementia, 2017-07, Vol.13 (7), p.P943-P944

2017

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Glutamate receptors in preclinical research on Alzheimer's disease: Update on recent advances: Glutamate Receptors

Pharmacology, biochemistry and behavior, 2012, Vol.100 (4), p.855-862

2012

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Activation of α7 nicotinic acetylcholine receptors persistently enhances hippocampal synaptic transmission and prevents Aß-mediated inhibition of LTP in the rat hippocampus

European journal of pharmacology, 2012-02, Vol.677 (1-3), p.63-70

2012

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S 44858, a positive allosteric modulator of ampa receptors and GluN2B selective nmda receptors antagonist, prevents the disruption of synaptic plasticity induced by Aβ oligomers

Alzheimer's & dementia, 2015-07, Vol.11 (7), p.P845-P845

2015

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Enduring glucocorticoid-evoked exacerbation of synaptic plasticity disruption in male rats modelling early Alzheimer's disease amyloidosis

Soluble tau aggregates inhibit synaptic long-term depression and amyloid β-facilitated LTD in vivo

PATIENT BRAIN-DERIVED SOLUBLE TAU RAPIDLY AND PERSISTENTLY DISRUPTS SYNAPTIC PLASTICITY IN VIVO

Removal of synaptic Ca²+-permeable AMPA receptors during sleep

Pre-plaque Aß-Mediated Impairment of Synaptic Depotentiation in a Transgenic Rat Model of Alzheimer's Disease Amyloidosis

Do tau-synaptic long-term depression interactions in the hippocampus play a pivotal role in the progression of Alzheimer's disease?

Rapidly reversible persistent long-term potentiation inhibition by patient-derived brain tau and amyloid ß proteins

Cellular Prion Protein Mediates the Disruption of Hippocampal Synaptic Plasticity by Soluble Tau In Vivo

Glutamate receptors in preclinical research on Alzheimer's disease: Update on recent advances

Gamma‐patterned sensory stimulation reverses synaptic plasticity deficits in rat models of early Alzheimer's disease

Tau and Amyloid β Protein in Patient-Derived Aqueous Brain Extracts Act Concomitantly to Disrupt Long-Term Potentiation in Vivo

Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system on the inhibition of hippocampal LTP by Aß

Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system x c - on the inhibition of hippocampal LTP by Aß

Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system [Formulaomitted] on the inhibition of hippocampal LTP by As

Targeting glutamatergic and cellular prion protein mechanisms of amyloid β-mediated persistent synaptic plasticity disruption: Longitudinal studies

Glucocorticoids, synaptic plasticity and Alzheimer's disease

Long-Term Depression-Inducing Low Frequency Stimulation Enhances p-Tau181 and p-Tau217 in an Age-Dependent Manner in Live Rats

Opposite in vivo effects of agents that stimulate or inhibit the glutamate/cysteine exchanger system xc− on the inhibition of hippocampal LTP by Aß

Alzheimer’s Disease Amyloid β-Protein and Synaptic Function

Neurotransmitter receptor and time dependence of the synaptic plasticity disrupting actions of Alzheimer's disease Aβ in vivo

A human monoclonal IgG that binds aβ assemblies and diverse amyloids exhibits anti-amyloid activities in vitro and in vivo

MEDI1814, A HIGH-AFFINITY ANTIBODY DIRECTED TO THE C-TERMINUS OF AβX-42, ABROGATES SYNAPTIC PLASTICITY DISRUPTION CAUSED BY SYNTHETIC OR AD BRAIN-DERIVED Aβ SOLUBLE ASSEMBLIES IN VIVO

Glutamate receptors in preclinical research on Alzheimer's disease: Update on recent advances: Glutamate Receptors

Activation of α7 nicotinic acetylcholine receptors persistently enhances hippocampal synaptic transmission and prevents Aß-mediated inhibition of LTP in the rat hippocampus

S 44858, a positive allosteric modulator of ampa receptors and GluN2B selective nmda receptors antagonist, prevents the disruption of synaptic plasticity induced by Aβ oligomers

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