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Blebs produced by actin–myosin contraction during apoptosis release damage-associated molecular pattern proteins before secondary necrosis occurs

Cell death and differentiation, 2013-10, Vol.20 (10), p.1293-1305

2013

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Activating ROCK1 somatic mutations in human cancer

Oncogene, 2010-04, Vol.29 (17), p.2591-2598

2010

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Guanine nucleotide exchange factors for the Rho GTPases: a role in human disease?

Journal of molecular medicine (Berlin, Germany), 1996, Vol.74 (10), p.563-571

Olson, M. F.

1996

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How apoptotic cells aid in the removal of their own cold dead bodies

Cell death and differentiation, 2012-05, Vol.19 (5), p.735-742

2012

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Rho GTPase signalling pathways in the morphological changes associated with apoptosis

Cell death and differentiation, 2002-05, Vol.9 (5), p.493-504

2002

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Cross-talk between Ras and Rho signalling pathways in transformation favours proliferation and increased motility

The EMBO journal, 2001-02, Vol.20 (4), p.755-766

2001

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An essential role of Rho, Rac, and Cdc42 GTPases in cell cycle progression through G1

Science (American Association for the Advancement of Science), 1995-09, Vol.269 (5228), p.1270-1272

1995

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Requirement of Ras-GTP-Raf Complexes for Activation of Raf-1 by Protein Kinase C

Science (American Association for the Advancement of Science), 1998-04, Vol.280 (5360), p.109-112

1998

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Membrane blebbing during apoptosis results from caspase-mediated activation of ROCK I

Nature cell biology, 2001-04, Vol.3 (4), p.339-345

2001

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Stability of p21Waf1/Cip1 CDK inhibitor protein is responsive to RhoA-mediated regulation of the actin cytoskeleton

Oncogene, 2006-05, Vol.25 (19), p.2708-2716

2006

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Distinct roles for DH and PH domains in the Lbc oncogene

Oncogene, 1997-12, Vol.15 (23), p.2827-2831

1997

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Guanine nucleotide exchange factors for the Rho GTPases : a role in human disease ?

Journal of molecular medicine (Berlin, Germany), 1996-10, Vol.74 (10), p.563-571

OLSON, M. F

1996

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Stability of p21 CDK inhibitor protein is responsive to RhoA-mediated regulation of the actin cytoskeleton

Oncogene, 2006-05, Vol.25 (19), p.2708-2716

2006

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Ras protein signalling

Seminars in immunology, 2000-02, Vol.12 (1), p.63-73

2000

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Faciogenital dysplasia protein (FGD1) and Vav, two related proteins required for normal embryonic development, are upstream regulators of Rho GTPases

Current biology, 1996-12, Vol.6 (12), p.1628-1633

1996

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Molecular basis for the 3',5'-cyclic adenosine monophosphate resistance of Kin mutant Y1 adrenocortical tumor cells

Molecular endocrinology (Baltimore, Md.), 1993-04, Vol.7 (4), p.477-487

1993

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Ras signalling is required for inactivation of the tumour suppressor pRb cell-cycle control protein

Current biology, 1997-03, Vol.7 (3), p.219-221

1997

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ROCK2/rasHa co-operation induces malignant conversion via p53 loss, elevated NF-[kappa]B and tenascin C-associated rigidity, but p21 inhibits ROCK2/NF-[kappa]B-mediated progression

Oncogene, 2017-05, Vol.36 (18), p.2529

2017

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ROCK2/rasHa co-operation induces malignant conversion via p53 loss, elevated NF-κB and tenascin C-associated rigidity, but p21 inhibits ROCK2/NF-κB-mediated progression

Oncogene, 2017-05, Vol.36 (18), p.2529-2542

2017

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ROCK2/ras super(Ha) co-operation induces malignant conversion via p53 loss, elevated NF- Kappa B and tenascin C-associated rigidity, but p21 inhibits ROCK2/NF- Kappa B-mediated progression

Oncogene, 2017-05, Vol.36 (18), p.2529-2542

2017

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The endocytic protein intersectin is a major binding partner for the Ras exchange factor mSos1 in rat brain

The EMBO journal, 2000-03, Vol.19 (6), p.1263-1271

2000

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Direct Involvement of the Small GTP-binding Protein Rho in lbc Oncogene Function

The Journal of biological chemistry, 1995-04, Vol.270 (16), p.9031-9034

1995

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Heterologous desensitization of the human dopamine D1 receptor in Y1 adrenal cells and in a desensitization-resistant Y1 mutant

Molecular endocrinology (Baltimore, Md.), 1992-07, Vol.6 (7), p.1095-1102

1992

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RAS and RHO GTPases in G1-phase cell-cycle regulation

Nature reviews. Molecular cell biology, 2004-05, Vol.5 (5), p.355-366

2004

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Targeting Ras and Rho GTPases as opportunities for cancer therapeutics

Current opinion in genetics & development, 2005-02, Vol.15 (1), p.62-68

2005

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Blebs produced by actin–myosin contraction during apoptosis release damage-associated molecular pattern proteins before secondary necrosis occurs

Activating ROCK1 somatic mutations in human cancer

Guanine nucleotide exchange factors for the Rho GTPases: a role in human disease?

How apoptotic cells aid in the removal of their own cold dead bodies

Rho GTPase signalling pathways in the morphological changes associated with apoptosis

Cross-talk between Ras and Rho signalling pathways in transformation favours proliferation and increased motility

An essential role of Rho, Rac, and Cdc42 GTPases in cell cycle progression through G1

Requirement of Ras-GTP-Raf Complexes for Activation of Raf-1 by Protein Kinase C

Membrane blebbing during apoptosis results from caspase-mediated activation of ROCK I

Stability of p21Waf1/Cip1 CDK inhibitor protein is responsive to RhoA-mediated regulation of the actin cytoskeleton

Distinct roles for DH and PH domains in the Lbc oncogene

Guanine nucleotide exchange factors for the Rho GTPases : a role in human disease ?

Stability of p21 CDK inhibitor protein is responsive to RhoA-mediated regulation of the actin cytoskeleton

Ras protein signalling

Faciogenital dysplasia protein (FGD1) and Vav, two related proteins required for normal embryonic development, are upstream regulators of Rho GTPases

Molecular basis for the 3',5'-cyclic adenosine monophosphate resistance of Kin mutant Y1 adrenocortical tumor cells

Ras signalling is required for inactivation of the tumour suppressor pRb cell-cycle control protein

ROCK2/rasHa co-operation induces malignant conversion via p53 loss, elevated NF-[kappa]B and tenascin C-associated rigidity, but p21 inhibits ROCK2/NF-[kappa]B-mediated progression

ROCK2/rasHa co-operation induces malignant conversion via p53 loss, elevated NF-κB and tenascin C-associated rigidity, but p21 inhibits ROCK2/NF-κB-mediated progression

ROCK2/ras super(Ha) co-operation induces malignant conversion via p53 loss, elevated NF- Kappa B and tenascin C-associated rigidity, but p21 inhibits ROCK2/NF- Kappa B-mediated progression

The endocytic protein intersectin is a major binding partner for the Ras exchange factor mSos1 in rat brain

Direct Involvement of the Small GTP-binding Protein Rho in lbc Oncogene Function

Heterologous desensitization of the human dopamine D1 receptor in Y1 adrenal cells and in a desensitization-resistant Y1 mutant

RAS and RHO GTPases in G1-phase cell-cycle regulation

Targeting Ras and Rho GTPases as opportunities for cancer therapeutics

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