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Can Exposure to Environmental Chemicals Increase the Risk of Diabetes Type 1 Development?

BioMed research international, 2015-01, Vol.2015, p.208947-19

2015

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Prenatal exposure to polychlorinated biphenyls and dioxins from the maternal diet may be associated with immunosuppressive effects that persist into early childhood

Food and chemical toxicology, 2013-01, Vol.51, p.165-172

2013

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Suspected gut barrier disruptors and development of food allergy: Adjuvant effects and early immune responses

Frontiers in allergy, 2022-11, Vol.3, p.1029125-1029125

2022

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Exposure to perfluoroundecanoic acid (PFUnDA) accelerates insulitis development in a mouse model of type 1 diabetes

Toxicology reports, 2016-01, Vol.3, p.664-672

2016

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Exposure to bisphenol A, but not phthalates, increases spontaneous diabetes type 1 development in NOD mice

Toxicology reports, 2015, Vol.2 (C), p.99-110

2015

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Investigations of immunogenic, allergenic and adjuvant properties of Cry1Ab protein after intragastric exposure in a food allergy model in mice

BMC immunology, 2016-05, Vol.17 (1), p.10-10, Article 10

2016

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Carbon Nanofibers Have IgE Adjuvant Capacity but Are Less Potent Than Nanotubes in Promoting Allergic Airway Responses

BioMed research international, 2013-01, Vol.2013, p.476010-12

2013

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Genetic and Diet-Induced Obesity Increased Intestinal Tumorigenesis in the Double Mutant Mouse Model Multiple Intestinal Neoplasia X Obese via Disturbed Glucose Regulation and Inflammation

Journal of Obesity, 2015-01, Vol.2015, p.343479-21

2015

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Corrigendum to “Genetic and Diet-Induced Obesity Increased Intestinal Tumorigenesis in the Double Mutant Mouse Model Multiple Intestinal Neoplasia X Obese via Disturbed Glucose Regulation and Inflammation”

Journal of obesity, 2016-01, Vol.2016, p.2361290-2

2016

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Particles from wood smoke and road traffic differently affect the innate immune system of the lung

Inhalation toxicology, 2009-09, Vol.21 (11), p.943-951

2009

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Immune responses in Omicron SARS-CoV-2 breakthrough infection in vaccinated adults

Nature communications, 2022-07, Vol.13 (1), p.4165-12, Article 4165

2022

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Toxic effects of mineral oil saturated hydrocarbons (MOSH) and relation to accumulation in rat liver

Food and chemical toxicology, 2023-07, Vol.177, p.113847-113847, Article 113847

2023

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Corrigendum to <Toxic effects of mineral oil saturated hydrocarbons (MOSH) and relation to accumulation in rat liver [Food Chem. Toxicol. 123 (2019) 431–442]>

Food and chemical toxicology, 2023-07, Vol.177, p.113848, Article 113848

2023

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Transmaternal bisphenol A exposure accelerates diabetes type 1 development in NOD mice

Toxicological sciences, 2014-02, Vol.137 (2), p.311-323

2014

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Gut bacteria at 6months of age are associated with immune cell status in 1‐year‐old children

Scandinavian journal of immunology, 2024-04, Vol.99 (4)

2024

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Gut bacteria at 6 months of age are associated with immune cell status in 1‐year‐old children

Scandinavian journal of immunology, 2024-04, Vol.99 (4), p.e13346-n/a

2024

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A Simple Pharmacokinetic Model of Prenatal and Postnatal Exposure to Perfluoroalkyl Substances (PFASs)

Environmental science & technology, 2016-01, Vol.50 (2), p.978-986

2016

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Toxic effects of mineral oil saturated hydrocarbons (MOSH) and relation to accumulation in rat liver

Food and chemical toxicology, 2019-01, Vol.123, p.431-442

2019

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Perfluoroalkyl substances, airways infections, allergy and asthma related health outcomes – implications of gender, exposure period and study design

Environment international, 2019

2019

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Particles Influence Allergic Responses in Mice-Role of Gender and Particle Size

Journal of Toxicology and Environmental Health, Part A, 2014-03, Vol.77 (5), p.281-292

2014

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Reduced polyfunctional T cells and increased cellular activation markers in adult allergy patients reporting adverse reactions to food

BMC immunology, 2020-07, Vol.21 (1), p.1-14, Article 43

2020

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Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice

Immunopharmacology and immunotoxicology, 2013-06, Vol.35 (3), p.349-358

2013

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Decreased macrophage phagocytic function due to xenobiotic exposures in vitro, difference in sensitivity between various macrophage models

Food and chemical toxicology, 2018-02, Vol.112, p.86-96

2018

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Exposure to endocrine disruptors accelerates diabetes type 1 development in NOD mice via impaired macrophage function

Toxicology letters, 2016-09, Vol.258, p.S25-S25

2016

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Can Exposure to Environmental Chemicals Increase the Risk of Diabetes Type 1 Development?

Prenatal exposure to polychlorinated biphenyls and dioxins from the maternal diet may be associated with immunosuppressive effects that persist into early childhood

Suspected gut barrier disruptors and development of food allergy: Adjuvant effects and early immune responses

Exposure to perfluoroundecanoic acid (PFUnDA) accelerates insulitis development in a mouse model of type 1 diabetes

Exposure to bisphenol A, but not phthalates, increases spontaneous diabetes type 1 development in NOD mice

Investigations of immunogenic, allergenic and adjuvant properties of Cry1Ab protein after intragastric exposure in a food allergy model in mice

Carbon Nanofibers Have IgE Adjuvant Capacity but Are Less Potent Than Nanotubes in Promoting Allergic Airway Responses

Genetic and Diet-Induced Obesity Increased Intestinal Tumorigenesis in the Double Mutant Mouse Model Multiple Intestinal Neoplasia X Obese via Disturbed Glucose Regulation and Inflammation

Corrigendum to “Genetic and Diet-Induced Obesity Increased Intestinal Tumorigenesis in the Double Mutant Mouse Model Multiple Intestinal Neoplasia X Obese via Disturbed Glucose Regulation and Inflammation”

Particles from wood smoke and road traffic differently affect the innate immune system of the lung

Immune responses in Omicron SARS-CoV-2 breakthrough infection in vaccinated adults

Toxic effects of mineral oil saturated hydrocarbons (MOSH) and relation to accumulation in rat liver

Corrigendum to <Toxic effects of mineral oil saturated hydrocarbons (MOSH) and relation to accumulation in rat liver [Food Chem. Toxicol. 123 (2019) 431–442]>

Transmaternal bisphenol A exposure accelerates diabetes type 1 development in NOD mice

Gut bacteria at 6months of age are associated with immune cell status in 1‐year‐old children

Gut bacteria at 6 months of age are associated with immune cell status in 1‐year‐old children

Gut bacteria at 6 months of age are associated with immune cell status in 1-year-old children

A Simple Pharmacokinetic Model of Prenatal and Postnatal Exposure to Perfluoroalkyl Substances (PFASs)

Toxic effects of mineral oil saturated hydrocarbons (MOSH) and relation to accumulation in rat liver

Perfluoroalkyl substances, airways infections, allergy and asthma related health outcomes – implications of gender, exposure period and study design

Particles Influence Allergic Responses in Mice-Role of Gender and Particle Size

Reduced polyfunctional T cells and increased cellular activation markers in adult allergy patients reporting adverse reactions to food

Long-term bisphenol A exposure accelerates insulitis development in diabetes-prone NOD mice

Decreased macrophage phagocytic function due to xenobiotic exposures in vitro, difference in sensitivity between various macrophage models

Exposure to endocrine disruptors accelerates diabetes type 1 development in NOD mice via impaired macrophage function

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