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Influenza virus targets the mRNA export machinery and the nuclear pore complex

Proceedings of the National Academy of Sciences - PNAS, 2007-02, Vol.104 (6), p.1853-1858

2007

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A rare cause of melena

Clinical journal of gastroenterology, 2020-12, Vol.13 (6), p.1157-1159

2020

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Nucleoporin Seh1 Interacts with Olig2/Brd7 to Promote Oligodendrocyte Differentiation and Myelination

Neuron (Cambridge, Mass.), 2019-05, Vol.102 (3), p.587-601.e7

2019

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NFAR-1 and -2 modulate translation and are required for efficient host defense

Proceedings of the National Academy of Sciences - PNAS, 2008-03, Vol.105 (11), p.4173-4178

2008

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The Nucleoporin Nup96 Is Required for Proper Expression of Interferon-Regulated Proteins and Functions

Immunity (Cambridge, Mass.), 2006-03, Vol.24 (3), p.295-304

2006

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Vesicular stomatitis virus inhibits mitotic progression and triggers cell death

EMBO reports, 2009-10, Vol.10 (10), p.1154-1160

2009

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Scurvy and Tinea Corporis Simulating Leukocytoclastic Vasculitis

The American journal of dermatopathology, 2021-12, Vol.43 (12), p.e230-e233

2021

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Sec13 Regulates Expression of Specific Immune Factors Involved in Inflammation In Vivo

Scientific reports, 2015-12, Vol.5 (1), p.17655-17655, Article 17655

2015

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Glycosylation Is Important for Binding to Human Calcitonin Receptors

Biochemistry (Easton), 1999-02, Vol.38 (6), p.1866-1872

1999

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S2880 A Rare Cause of Melena

The American journal of gastroenterology, 2020-10, Vol.115 (1), p.S1510-S1510

2020

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Viral Interactions with the Nuclear Transport Machinery: Discovering and Disrupting Pathways

IUBMB life, 2005-03, Vol.57 (2), p.65-72

2005

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The Diverse Clinical Presentations of Adrenal Lymphoma

AACE clinical case reports, 2017-01, Vol.3 (4), p.307-312

2017

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A Hydrophobic Cluster between Transmembrane Helices 5 and 6 Constrains the Thyrotropin-Releasing Hormone Receptor in an Inactive Conformation

Molecular pharmacology, 1998-12, Vol.54 (6), p.968-978

1998

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A Disulfide Bond between Conserved Extracellular Cysteines in the Thyrotropin-releasing Hormone Receptor Is Critical for Binding (∗)

The Journal of biological chemistry, 1995-10, Vol.270 (42), p.24682-24685

1995

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Truncation of the Thyrotropin-releasing Hormone Receptor Carboxyl Tail Causes Constitutive Activity and Leads to Impaired Responsiveness in Xenopus Oocytes and AtT20 Cells (∗)

The Journal of biological chemistry, 1995-01, Vol.270 (3), p.1041-1047

1995

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Human calcitonin receptor is directly targeted to and retained in the basolateral surface of MDCK cells

American Journal of Physiology: Cell Physiology, 1998-11, Vol.275 (5), p.C1264-C1276

1998

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Human Calcitonin Receptors Exhibit Agonist-Independent (Constitutive) Signaling Activity1

Endocrinology (Philadelphia), 1997-04, Vol.138 (4), p.1400-1405

1997

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Cellular mechanisms of the clearance function of type C receptors of atrial natriuretic factor

The Journal of biological chemistry, 1990-12, Vol.265 (34), p.20952-20958

1990

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Agonist-stimulated internalization of the thyrotropin-releasing hormone receptor is dependent on two domains in the receptor carboxyl terminus

The Journal of biological chemistry, 1993-02, Vol.268 (4), p.2389-2392

1993

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Inhibition of inositol phosphate second messenger formation by intracellular loop one of a human calcitonin receptor. Expression and mutational analysis of synthetic receptor genes

The Journal of biological chemistry, 1994-11, Vol.269 (45), p.28123-28129

1994

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Dynamics of atrial natriuretic factor-guanylate cyclase receptors and receptor-ligand complexes in cultured glomerular mesangial and renomedullary interstitial cells

The Journal of biological chemistry, 1992-06, Vol.267 (17), p.11987-11994

1992

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Decreased levels of internalized thyrotropin-releasing hormone receptors after uncoupling from guanine nucleotide-binding protein and phospholipase-C

Molecular endocrinology (Baltimore, Md.), 1993-09, Vol.7 (9), p.1105-1111

1993

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Thyrotropin-releasing hormone binding to the mouse pituitary receptor does not involve ionic interactions. A model for neutral peptide binding to G protein-coupled receptors

The Journal of biological chemistry, 1992-12, Vol.267 (34), p.24413-24417

1992

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Expression of thyrotropin-releasing hormone receptors by adenovirus-mediated gene transfer reveals that thyrotropin-releasing hormone desensitization is cell specific

Molecular pharmacology, 1994-04, Vol.45 (4), p.684

1994

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A constitutively active mutant thyrotropin-releasing hormone receptor is chronically down-regulated in pituitary cells: evidence using chlordiazepoxide as a negative antagonist

Molecular endocrinology (Baltimore, Md.), 1995-11, Vol.9 (11), p.1455-1460

1995

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Influenza virus targets the mRNA export machinery and the nuclear pore complex

A rare cause of melena

Nucleoporin Seh1 Interacts with Olig2/Brd7 to Promote Oligodendrocyte Differentiation and Myelination

NFAR-1 and -2 modulate translation and are required for efficient host defense

The Nucleoporin Nup96 Is Required for Proper Expression of Interferon-Regulated Proteins and Functions

Vesicular stomatitis virus inhibits mitotic progression and triggers cell death

Scurvy and Tinea Corporis Simulating Leukocytoclastic Vasculitis

Sec13 Regulates Expression of Specific Immune Factors Involved in Inflammation In Vivo

Glycosylation Is Important for Binding to Human Calcitonin Receptors

S2880 A Rare Cause of Melena

Viral Interactions with the Nuclear Transport Machinery: Discovering and Disrupting Pathways

The Diverse Clinical Presentations of Adrenal Lymphoma

A Hydrophobic Cluster between Transmembrane Helices 5 and 6 Constrains the Thyrotropin-Releasing Hormone Receptor in an Inactive Conformation

A Disulfide Bond between Conserved Extracellular Cysteines in the Thyrotropin-releasing Hormone Receptor Is Critical for Binding (∗)

Truncation of the Thyrotropin-releasing Hormone Receptor Carboxyl Tail Causes Constitutive Activity and Leads to Impaired Responsiveness in Xenopus Oocytes and AtT20 Cells (∗)

Human calcitonin receptor is directly targeted to and retained in the basolateral surface of MDCK cells

Human Calcitonin Receptors Exhibit Agonist-Independent (Constitutive) Signaling Activity1

Cellular mechanisms of the clearance function of type C receptors of atrial natriuretic factor

Agonist-stimulated internalization of the thyrotropin-releasing hormone receptor is dependent on two domains in the receptor carboxyl terminus

Inhibition of inositol phosphate second messenger formation by intracellular loop one of a human calcitonin receptor. Expression and mutational analysis of synthetic receptor genes

Dynamics of atrial natriuretic factor-guanylate cyclase receptors and receptor-ligand complexes in cultured glomerular mesangial and renomedullary interstitial cells

Decreased levels of internalized thyrotropin-releasing hormone receptors after uncoupling from guanine nucleotide-binding protein and phospholipase-C

Thyrotropin-releasing hormone binding to the mouse pituitary receptor does not involve ionic interactions. A model for neutral peptide binding to G protein-coupled receptors

Expression of thyrotropin-releasing hormone receptors by adenovirus-mediated gene transfer reveals that thyrotropin-releasing hormone desensitization is cell specific

A constitutively active mutant thyrotropin-releasing hormone receptor is chronically down-regulated in pituitary cells: evidence using chlordiazepoxide as a negative antagonist

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