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Coordinate β-adrenergic inhibition of mitochondrial activity and angiogenesis arrest tumor growth

Nature communications, 2020-07, Vol.11 (1), p.3606-3606, Article 3606

2020

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Dysfunctional oxidative phosphorylation shunts branched‐chain amino acid catabolism onto lipogenesis in skeletal muscle

The EMBO journal, 2020-07, Vol.39 (14), p.e103812-n/a

2020

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MiR‐9‐5p protects from kidney fibrosis by metabolic reprogramming

The FASEB journal, 2020-01, Vol.34 (1), p.410-431

2020

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Analysis of the metabolic proteome of lung adenocarcinomas by reverse-phase protein arrays (RPPA) emphasizes mitochondria as targets for therapy

Oncogenesis (New York, NY), 2022-05, Vol.11 (1), p.24-24, Article 24

2022

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The role of KRAS splice variants in cancer biology

Frontiers in cell and developmental biology, 2022-11, Vol.10, p.1033348-1033348

2022

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Critical requirement of SOS1 RAS-GEF function for mitochondrial dynamics, metabolism, and redox homeostasis

Oncogene, 2021-07, Vol.40 (27), p.4538-4551

2021

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PKA Phosphorylates the ATPase Inhibitory Factor 1 and Inactivates Its Capacity to Bind and Inhibit the Mitochondrial H+-ATP Synthase

Cell reports (Cambridge), 2015-09, Vol.12 (12), p.2143-2155

2015

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PD-1 signaling affects cristae morphology and leads to mitochondrial dysfunction in human CD8 + T lymphocytes

Journal for immunotherapy of cancer, 2019-06, Vol.7 (1), p.151-151, Article 151

2019

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IF1 promotes oligomeric assemblies of sluggish ATP synthase and outlines the heterogeneity of the mitochondrial membrane potential

Communications biology, 2023-08, Vol.6 (1), p.836-836, Article 836

2023

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ATPase Inhibitory Factor 1 (IF1): A main driver of metabolic reprogramming and nuclear signaling in cancer

Biochimica et biophysica acta. Bioenergetics, 2016-08, Vol.1857, p.e9-e9

2016

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Determination of the H+-ATP Synthase and Hydrolytic Activities

Bio-protocol, 2016-08, Vol.6 (16)

2016

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Metabolic reprogramming and disease progression in cancer patients

Biochimica et biophysica acta. Molecular basis of disease, 2020-05, Vol.1866 (5), p.165721-165721, Article 165721

2020

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Differential functions of the KRAS splice variants

Biochemical Society transactions, 2023-06, Vol.51 (3), p.1191-1199

2023

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Regulation of the H+-ATP synthase by IF1: a role in mitohormesis

Cellular and molecular life sciences : CMLS, 2017-06, Vol.74 (12), p.2151-2166

2017

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UPF1 deficiency enhances mitochondrial ROS which promotes an immunosuppressive microenvironment in pancreatic ductal adenocarcinoma

Proceedings of the National Academy of Sciences - PNAS, 2024-08, Vol.121 (33)

2024

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Regulation of the H^sup +^-ATP synthase by IF1: a role in mitohormesis

Cellular and molecular life sciences : CMLS, 2017-06, Vol.74 (12), p.2151

2017

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Tissue‐specific expression and post‐transcriptional regulation of the ATPase inhibitory factor 1 (IF1) in human and mouse tissues

The FASEB journal, 2019-02, Vol.33 (2), p.1836-1851

2019

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Effective therapeutic strategies in a preclinical mouse model of Charcot–Marie–Tooth disease

Human molecular genetics, 2021-11, Vol.30 (24), p.2441-2455

2021

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Dysfunction of the immune system in conditional IF1 (Atp5if1-/-) knockout mice in colon

Biochimica et biophysica acta. Bioenergetics, 2018-09, Vol.1859, p.e63-e63

2018

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Overexpression of Mitochondrial IF1 Prevents Metastatic Disease of Colorectal Cancer by Enhancing Anoikis and Tumor Infiltration of NK Cells

Cancers, 2019-12, Vol.12 (1), p.22

2019

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Metformin as an Adjuvant to Photodynamic Therapy in Resistant Basal Cell Carcinoma Cells

Cancers, 2020-03, Vol.12 (3), p.668

2020

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Overexpression of the ATPase Inhibitory Factor 1 Favors a Non-metastatic Phenotype in Breast Cancer

Frontiers in oncology, 2017-04, Vol.7, p.69-69

2017

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Specific Effects of Trabectedin and Lurbinectedin on Human Macrophage Function and Fate—Novel Insights

Cancers, 2020-10, Vol.12 (10), p.3060

2020

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Coordinate β-adrenergic inhibition of mitochondrial activity and angiogenesis arrest tumor growth

Dysfunctional oxidative phosphorylation shunts branched‐chain amino acid catabolism onto lipogenesis in skeletal muscle

MiR‐9‐5p protects from kidney fibrosis by metabolic reprogramming

Analysis of the metabolic proteome of lung adenocarcinomas by reverse-phase protein arrays (RPPA) emphasizes mitochondria as targets for therapy

The role of KRAS splice variants in cancer biology

Critical requirement of SOS1 RAS-GEF function for mitochondrial dynamics, metabolism, and redox homeostasis

PKA Phosphorylates the ATPase Inhibitory Factor 1 and Inactivates Its Capacity to Bind and Inhibit the Mitochondrial H+-ATP Synthase

PD-1 signaling affects cristae morphology and leads to mitochondrial dysfunction in human CD8 + T lymphocytes

IF1 promotes oligomeric assemblies of sluggish ATP synthase and outlines the heterogeneity of the mitochondrial membrane potential

ATPase Inhibitory Factor 1 (IF1): A main driver of metabolic reprogramming and nuclear signaling in cancer

Determination of the H+-ATP Synthase and Hydrolytic Activities

Metabolic reprogramming and disease progression in cancer patients

Differential functions of the KRAS splice variants

Regulation of the H+-ATP synthase by IF1: a role in mitohormesis

UPF1 deficiency enhances mitochondrial ROS which promotes an immunosuppressive microenvironment in pancreatic ductal adenocarcinoma

Regulation of the H^sup +^-ATP synthase by IF1: a role in mitohormesis

Tissue‐specific expression and post‐transcriptional regulation of the ATPase inhibitory factor 1 (IF1) in human and mouse tissues

Effective therapeutic strategies in a preclinical mouse model of Charcot–Marie–Tooth disease

Dysfunction of the immune system in conditional IF1 (Atp5if1-/-) knockout mice in colon

Overexpression of Mitochondrial IF1 Prevents Metastatic Disease of Colorectal Cancer by Enhancing Anoikis and Tumor Infiltration of NK Cells

Metformin as an Adjuvant to Photodynamic Therapy in Resistant Basal Cell Carcinoma Cells

Overexpression of the ATPase Inhibitory Factor 1 Favors a Non-metastatic Phenotype in Breast Cancer

Specific Effects of Trabectedin and Lurbinectedin on Human Macrophage Function and Fate—Novel Insights

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