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Notch activation inhibits AML growth and survival: a potential therapeutic approach

The Journal of experimental medicine, 2013-02, Vol.210 (2), p.321-337

2013

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The bantam MicroRNA Is a Target of the Hippo Tumor-Suppressor Pathway

Current biology, 2006-10, Vol.16 (19), p.1895-1904

2006

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Hippo promotes proliferation arrest and apoptosis in the Salvador/Warts pathway

Nature cell biology, 2003-10, Vol.5 (10), p.914-920

2003

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Characterization of TRKA signaling in acute myeloid leukemia

Oncotarget, 2018-07, Vol.9 (53), p.30092-30105

2018

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Shar-pei mediates cell proliferation arrest during imaginal disc growth in Drosophila

Development (Cambridge), 2002-12, Vol.129 (24), p.5719-5730

2002

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Senseless, a Zn Finger Transcription Factor, Is Necessary and Sufficient for Sensory Organ Development in Drosophila

Cell, 2000-08, Vol.102 (3), p.349-362

2000

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Notch pathway activation induces neuroblastoma tumor cell growth arrest

Pediatric blood & cancer, 2012-05, Vol.58 (5), p.682-689

2012

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senseless Repression of rough Is Required for R8 Photoreceptor Differentiation in the Developing Drosophila Eye

Neuron (Cambridge, Mass.), 2001-11, Vol.32 (3), p.403-414

2001

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Regulation of cell migration by amphoterin

Journal of cell science, 2000-02, Vol.113 ( Pt 4) (4), p.611-620

2000

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The tumour-suppressor genes NF2/Merlin and Expanded act through Hippo signalling to regulate cell proliferation and apoptosis

Nature cell biology, 2006-01, Vol.8 (1), p.27-36

2006

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Neurite Outgrowth in Brain Neurons Induced by Heparin-binding Growth-associated Molecule (HB-GAM) Depends on the Specific Interaction of HB-GAM with Heparan Sulfate at the Cell Surface

The Journal of biological chemistry, 1996-01, Vol.271 (4), p.2243-2248

1996

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Targeting P-selectin blocks neuroblastoma growth

Oncotarget, 2017-10, Vol.8 (49), p.86657-86670

2017

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Regulation of NOTCH signaling by reciprocal inhibition of HES1 and Deltex 1 and its role in osteosarcoma invasiveness

Oncogene, 2010-05, Vol.29 (20), p.2916-2926

2010

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Senseless acts as a binary switch during sensory organ precursor selection

Genes & development, 2003-12, Vol.17 (23), p.2966-2978

2003

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Expression of HB-GAM (heparin-binding growth-associated molecules) in the pathways of developing axonal processes in vivo and neurite outgrowth in vitro induced by HB-GAM

Brain research. Developmental brain research, 1994-06, Vol.79 (2), p.157-176

1994

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Isolation of a neuronal cell surface receptor of heparin binding growth-associated molecule (HB-GAM). Identification as N-syndecan (syndecan-3)

The Journal of biological chemistry, 1994-04, Vol.269 (17), p.12999-13004

1994

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Understanding the Role of TRKA Receptor in Acute Myeloid Leukemias: From Proliferation and Pro-Survival Signals to a Novel Therapeutic Approach

Blood, 2008-11, Vol.112 (11), p.3789-3789

2008

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HB-GAM is a cytokine present in Alzheimerʼs and Downʼs syndrome lesions

Neuroreport, 1996-01, Vol.7 (2), p.667-671

1996

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Amphoterin, the 30-kDa protein in a family of HMG1-type polypeptides. Enhanced expression in transformed cells, leading edge localization, and interactions with plasminogen activation

The Journal of biological chemistry, 1993-09, Vol.268 (26), p.19726-19738

1993

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N-Syndecan and HB-GAM (Heparin-Binding Growth-Associated Molecule) associate with early axonal tracts in the rat brain

The European journal of neuroscience, 1998-02, Vol.10 (2), p.635-648

1998

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Heparan sulphate and HB-GAM (heparin-binding rowth-associated molecule) in the development of the thalamocortical pathway of rat brain

The European journal of neuroscience, 1999-02, Vol.11 (2), p.491-502

1999

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Drosophila Lyra Mutations Are Gain-of-Function Mutations of senseless

Genetics (Austin), 2001-01, Vol.157 (1), p.307-315

2001

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Co-expression of heparin-binding growth-associated molecule (HB-GAM) and N-syndecan (syndecan-3) in developing rat brain

Neuroscience letters, 1995-01, Vol.191 (1), p.39-42

1995

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Developmentally Regulated Neurite Outgrowth Response from Dorsal Root Ganglion Neurons to Heparin-binding Growth-associated Molecule (HB-GAM) and the Expression of HB-GAM in the Targets of the Developing Dorsal Root Ganglion Neurites

The European journal of neuroscience, 1996-08, Vol.8 (8), p.1658-1665

1996

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HB-GAM is a cytokine present in Alzheimerʼs and Downʼs syndrome lesions

Neuroreport, 1996-01, Vol.7 (2), p.667-671

1996

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Notch activation inhibits AML growth and survival: a potential therapeutic approach

The bantam MicroRNA Is a Target of the Hippo Tumor-Suppressor Pathway

Hippo promotes proliferation arrest and apoptosis in the Salvador/Warts pathway

Characterization of TRKA signaling in acute myeloid leukemia

Shar-pei mediates cell proliferation arrest during imaginal disc growth in Drosophila

Senseless, a Zn Finger Transcription Factor, Is Necessary and Sufficient for Sensory Organ Development in Drosophila

Notch pathway activation induces neuroblastoma tumor cell growth arrest

senseless Repression of rough Is Required for R8 Photoreceptor Differentiation in the Developing Drosophila Eye

Regulation of cell migration by amphoterin

The tumour-suppressor genes NF2/Merlin and Expanded act through Hippo signalling to regulate cell proliferation and apoptosis

Neurite Outgrowth in Brain Neurons Induced by Heparin-binding Growth-associated Molecule (HB-GAM) Depends on the Specific Interaction of HB-GAM with Heparan Sulfate at the Cell Surface

Targeting P-selectin blocks neuroblastoma growth

Regulation of NOTCH signaling by reciprocal inhibition of HES1 and Deltex 1 and its role in osteosarcoma invasiveness

Senseless acts as a binary switch during sensory organ precursor selection

Expression of HB-GAM (heparin-binding growth-associated molecules) in the pathways of developing axonal processes in vivo and neurite outgrowth in vitro induced by HB-GAM

Isolation of a neuronal cell surface receptor of heparin binding growth-associated molecule (HB-GAM). Identification as N-syndecan (syndecan-3)

Understanding the Role of TRKA Receptor in Acute Myeloid Leukemias: From Proliferation and Pro-Survival Signals to a Novel Therapeutic Approach

HB-GAM is a cytokine present in Alzheimerʼs and Downʼs syndrome lesions

Amphoterin, the 30-kDa protein in a family of HMG1-type polypeptides. Enhanced expression in transformed cells, leading edge localization, and interactions with plasminogen activation

N-Syndecan and HB-GAM (Heparin-Binding Growth-Associated Molecule) associate with early axonal tracts in the rat brain

Heparan sulphate and HB-GAM (heparin-binding rowth-associated molecule) in the development of the thalamocortical pathway of rat brain

Drosophila Lyra Mutations Are Gain-of-Function Mutations of senseless

Co-expression of heparin-binding growth-associated molecule (HB-GAM) and N-syndecan (syndecan-3) in developing rat brain

Developmentally Regulated Neurite Outgrowth Response from Dorsal Root Ganglion Neurons to Heparin-binding Growth-associated Molecule (HB-GAM) and the Expression of HB-GAM in the Targets of the Developing Dorsal Root Ganglion Neurites

HB-GAM is a cytokine present in Alzheimerʼs and Downʼs syndrome lesions

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