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RelB Expression Determines the Differential Effects of Ascorbic Acid in Normal and Cancer Cells

Cancer research (Chicago, Ill.), 2017-03, Vol.77 (6), p.1345-1356

2017

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Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna-A Randomized, Cross-Over Clinical Study

PloS one, 2015-04, Vol.10 (4), p.e0124988

2015

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p53 Regulates oxidative stress-mediated retrograde signaling: a novel mechanism for chemotherapy-induced cardiac injury

PloS one, 2011-03, Vol.6 (3), p.e18005

2011

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Pharmacokinetics of the potent redox-modulating manganese porphyrin, MnTE-2-PyP(5+), in plasma and major organs of B6C3F1 mice

Free radical biology & medicine, 2008-10, Vol.45 (7), p.943

2008

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Phenylbutyrate, a histone deacetylase inhibitor, protects against Adriamycin-induced cardiac injury

Free radical biology & medicine, 2007-06, Vol.42 (12), p.1818-1825

2007

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The Twentieth-Century Performance Reader

3rd edition, 2013

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Reflection & Self‐Assessment Cycles in a Medical School Anatomy Course

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Pharmacokinetics of the potent redox-modulating manganese porphyrin, MnTE-2-PyP5+, in plasma and major organs of B6C3F1 mice

Free radical biology & medicine, 2008-10, Vol.45 (7), p.943-949

2008

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Phospholipase C-δ1 Is a Critical Target for Tumor Necrosis Factor Receptor–Mediated Protection against Adriamycin-Induced Cardiac Injury

Cancer research (Chicago, Ill.), 2006-04, Vol.66 (8), p.4329-4338

2006

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Extracellular Vesicles as a Potential Mediator of Adaptive Redox State in Radiation Resistant Cancer

Free radical biology & medicine, 2022-02, Vol.180, p.s33

2022

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The triangle of death of neurons: Oxidative damage, mitochondrial dysfunction, and loss of choline-containing biomolecules in brains of mice treated with doxorubicin. Advanced insights into mechanisms of chemotherapy induced cognitive impairment (“chemobrain”) involving TNF-α

Free radical biology & medicine, 2019-04, Vol.134, p.1-8

2019

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Doxorubicin-induced elevated oxidative stress and neurochemical alterations in brain and cognitive decline: protection by MESNA and insights into mechanisms of chemotherapy-induced cognitive impairment ("chemobrain")

Oncotarget, 2018-07, Vol.9 (54), p.30324-30339

2018

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Tumor necrosis factor receptor deficiency exacerbated Adriamycin-induced cardiomyocytes apoptosis: an insight into the Fas connection

Molecular cancer therapeutics, 2006-02, Vol.5 (2), p.261-269

2006

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Extracellular Vesicles, Potential Non-invasive Markers of Cancer Therapy, Induce Neurotoxicity

Free radical biology & medicine, 2020-11, Vol.159, p.S54-S54

2020

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Adaptation of Redox State in Cancer Cells that Survive Radiation

Free radical biology & medicine, 2020-11, Vol.159, p.S57-S57

2020

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Targeting Mitochondrial Redox Capacity Coupled with Mitochondrial Protein Translation To Improve Radiation Efficacy

Free radical biology & medicine, 2022-02, Vol.180, p.s27

2022

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Overexpression of Brain-Derived Neurotrophic Factor Enhances Sensory Innervation and Selectively Increases Neuron Number

The Journal of neuroscience, 1999-07, Vol.19 (14), p.5919-5931

1999

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Mn porphyrin-based superoxide dismutase (SOD) mimic, MnIIITE-2-PyP5+, targets mouse heart mitochondria

Free radical biology & medicine, 2007-04, Vol.42 (8), p.1193-1200

2007

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Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury

Clinical cancer research, 2018-04, Vol.24 (7), p.1644-1653

2018

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Mitochondrial H2O2 Defines Redox Heterogeneity in Radiation Resistant Prostate Cancer

Free radical biology & medicine, 2020-11, Vol.159, p.S51-S51

2020

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Extracellular vesicles released after cranial radiation: An insight into an early mechanism of brain injury

Brain research, 2022-05, Vol.1782, p.147840-147840, Article 147840

2022

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2-Mercaptoethane sulfonate prevents doxorubicin-induced plasma protein oxidation and TNF-α release: Implications for the reactive oxygen species-mediated mechanisms of chemobrain

Free radical biology & medicine, 2011-06, Vol.50 (11), p.1630-1638

2011

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Acute treatment with doxorubicin affects glutamate neurotransmission in the mouse frontal cortex and hippocampus

Brain research, 2017-10, Vol.1672, p.10-17

2017

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Novel role of 4-hydroxy-2-nonenal in AIFm2-mediated mitochondrial stress signaling

Free radical biology & medicine, 2016-02, Vol.91, p.68-80

2016

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Exosomes Mediate Cardiac Injury in Doxorubicin-Induced Cardiomyopathy

Free radical biology & medicine, 2014-11, Vol.76, p.S56

2014

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RelB Expression Determines the Differential Effects of Ascorbic Acid in Normal and Cancer Cells

Plasma TNF-α and Soluble TNF Receptor Levels after Doxorubicin with or without Co-Administration of Mesna-A Randomized, Cross-Over Clinical Study

p53 Regulates oxidative stress-mediated retrograde signaling: a novel mechanism for chemotherapy-induced cardiac injury

Pharmacokinetics of the potent redox-modulating manganese porphyrin, MnTE-2-PyP(5+), in plasma and major organs of B6C3F1 mice

Phenylbutyrate, a histone deacetylase inhibitor, protects against Adriamycin-induced cardiac injury

The Twentieth-Century Performance Reader

Reflection & Self‐Assessment Cycles in a Medical School Anatomy Course

Pharmacokinetics of the potent redox-modulating manganese porphyrin, MnTE-2-PyP5+, in plasma and major organs of B6C3F1 mice

Phospholipase C-δ1 Is a Critical Target for Tumor Necrosis Factor Receptor–Mediated Protection against Adriamycin-Induced Cardiac Injury

Extracellular Vesicles as a Potential Mediator of Adaptive Redox State in Radiation Resistant Cancer

The triangle of death of neurons: Oxidative damage, mitochondrial dysfunction, and loss of choline-containing biomolecules in brains of mice treated with doxorubicin. Advanced insights into mechanisms of chemotherapy induced cognitive impairment (“chemobrain”) involving TNF-α

Doxorubicin-induced elevated oxidative stress and neurochemical alterations in brain and cognitive decline: protection by MESNA and insights into mechanisms of chemotherapy-induced cognitive impairment ("chemobrain")

Tumor necrosis factor receptor deficiency exacerbated Adriamycin-induced cardiomyocytes apoptosis: an insight into the Fas connection

Extracellular Vesicles, Potential Non-invasive Markers of Cancer Therapy, Induce Neurotoxicity

Adaptation of Redox State in Cancer Cells that Survive Radiation

Targeting Mitochondrial Redox Capacity Coupled with Mitochondrial Protein Translation To Improve Radiation Efficacy

Overexpression of Brain-Derived Neurotrophic Factor Enhances Sensory Innervation and Selectively Increases Neuron Number

Mn porphyrin-based superoxide dismutase (SOD) mimic, MnIIITE-2-PyP5+, targets mouse heart mitochondria

Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury

Mitochondrial H2O2 Defines Redox Heterogeneity in Radiation Resistant Prostate Cancer

Extracellular vesicles released after cranial radiation: An insight into an early mechanism of brain injury

2-Mercaptoethane sulfonate prevents doxorubicin-induced plasma protein oxidation and TNF-α release: Implications for the reactive oxygen species-mediated mechanisms of chemobrain

Acute treatment with doxorubicin affects glutamate neurotransmission in the mouse frontal cortex and hippocampus

Novel role of 4-hydroxy-2-nonenal in AIFm2-mediated mitochondrial stress signaling

Exosomes Mediate Cardiac Injury in Doxorubicin-Induced Cardiomyopathy

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