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Sirtuins: To Be or Not To Be in Diabetic Cardiomyopathy

Trends in molecular medicine, 2021-06, Vol.27 (6), p.554-571

2021

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SIRT3-mediated inhibition of FOS through histone H3 deacetylation prevents cardiac fibrosis and inflammation

Signal transduction and targeted therapy, 2020-02, Vol.5 (1), p.14, Article 14

2020

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Aortic disease in Marfan syndrome is caused by overactivation of sGC-PRKG signaling by NO

Nature communications, 2021-05, Vol.12 (1), p.2628-2628, Article 2628

2021

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Neuron-derived orphan receptor-1 modulates cardiac gene expression and exacerbates angiotensin II-induced cardiac hypertrophy

Clinical science (1979), 2020-02, Vol.134 (3), p.359-377

2020

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Myocardial and circulating levels of microRNA-21 reflect left ventricular fibrosis in aortic stenosis patients

International journal of cardiology, 2013-09, Vol.167 (6), p.2875-2881

2013

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Experimental modelling of cardiac pressure overload hypertrophy: Modified technique for precise, reproducible, safe and easy aortic arch banding-debanding in mice

Scientific reports, 2018-02, Vol.8 (1), p.3167-10, Article 3167

2018

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Conditional deletion of Rcan1 predisposes to hypertension-mediated intramural hematoma and subsequent aneurysm and aortic rupture

Nature communications, 2018-11, Vol.9 (1), p.4795-16, Article 4795

2018

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Extracellular Tuning of Mitochondrial Respiration Leads to Aortic Aneurysm

Circulation (New York, N.Y.), 2021-05, Vol.143 (21), p.2091-2109

2021

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Plasma Levels of Transforming Growth Factor-β1 Reflect Left Ventricular Remodeling in Aortic Stenosis

PloS one, 2009-12, Vol.4 (12), p.e8476

2009

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Nitric oxide mediates aortic disease in mice deficient in the metalloprotease Adamts1 and in a mouse model of Marfan syndrome

Nature medicine, 2017-02, Vol.23 (2), p.200-212

2017

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BMP7-based peptide agonists of BMPR1A protect the left ventricle against pathological remodeling induced by pressure overload

Biomedicine & pharmacotherapy, 2022-05, Vol.149, p.112910-112910, Article 112910

2022

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Androgens contribute to sex differences in myocardial remodeling under pressure overload by a mechanism involving TGF-β

PloS one, 2012, Vol.7 (4), p.e35635

2012

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BAMBI (BMP and activin membrane-bound inhibitor) protects the murine heart from pressure-overload biomechanical stress by restraining TGF-β signaling

Biochimica et biophysica acta, 2013-02, Vol.1832 (2), p.323-335

2013

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Long-term results of the augmented PFNA: a prospective multicenter trial

Archives of orthopaedic and trauma surgery, 2014-03, Vol.134 (3), p.343-349

2014

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Extracellular heat shock protein 90 binding to TGFβ receptor I participates in TGFβ-mediated collagen production in myocardial fibroblasts

Cellular signalling, 2016-10, Vol.28 (10), p.1563-1579

2016

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p-SMAD2/3 and DICER promote pre-miR-21 processing during pressure overload-associated myocardial remodeling

Biochimica et biophysica acta, 2015-07, Vol.1852 (7), p.1520-1530

2015

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Knockout mutants of Arabidopsis thaliana β-galactosidase. Modifications in the cell wall saccharides and enzymatic activities

Biologia plantarum, 2018, Vol.62 (1), p.80-88

2018

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Sex-Specific Regulation of miR-29b in the Myocardium Under Pressure Overload is Associated with Differential Molecular, Structural and Functional Remodeling Patterns in Mice and Patients with Aortic Stenosis

Cells (Basel, Switzerland), 2020-03, Vol.9 (4), p.833

2020

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Myocardial gene expression of microRNA-133a and myosin heavy and light chains, in conjunction with clinical parameters, predict regression of left ventricular hypertrophy after valve replacement in patients with aortic stenosis

Heart (British Cardiac Society), 2011-07, Vol.97 (14), p.1132-1137

2011

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Versican accumulation drives Nos2 induction and aortic disease in Marfan syndrome via Akt activation

EMBO molecular medicine, 2024-01, Vol.16 (1), p.132-157

2024

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Coordinated action of β-galactosidases in the cell wall of embryonic axes during chickpea germination and seedling growth

Plant biology (Stuttgart, Germany), 2014-03, Vol.16 (2), p.404-410

2014

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Circulating Levels of miR‐133a Predict the Regression Potential of Left Ventricular Hypertrophy After Valve Replacement Surgery in Patients With Aortic Stenosis

Journal of the American Heart Association, 2013-08, Vol.2 (4), p.e000211-n/a

2013

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Selective inhibition of plasma membrane calcium ATPase 4 improves angiogenesis and vascular reperfusion

Journal of molecular and cellular cardiology, 2017-08, Vol.109, p.38-47

2017

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Gender differences of echocardiographic and gene expression patterns in human pressure overload left ventricular hypertrophy

Journal of molecular and cellular cardiology, 2009-04, Vol.46 (4), p.526-535

2009

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Infestation of a diabetic foot by Wohlfahrtia magnifica

Journal of vascular surgery cases and innovative techniques, 2016-09, Vol.2 (3), p.119-122

2016

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Sirtuins: To Be or Not To Be in Diabetic Cardiomyopathy

SIRT3-mediated inhibition of FOS through histone H3 deacetylation prevents cardiac fibrosis and inflammation

Aortic disease in Marfan syndrome is caused by overactivation of sGC-PRKG signaling by NO

Neuron-derived orphan receptor-1 modulates cardiac gene expression and exacerbates angiotensin II-induced cardiac hypertrophy

Myocardial and circulating levels of microRNA-21 reflect left ventricular fibrosis in aortic stenosis patients

Experimental modelling of cardiac pressure overload hypertrophy: Modified technique for precise, reproducible, safe and easy aortic arch banding-debanding in mice

Conditional deletion of Rcan1 predisposes to hypertension-mediated intramural hematoma and subsequent aneurysm and aortic rupture

Extracellular Tuning of Mitochondrial Respiration Leads to Aortic Aneurysm

Plasma Levels of Transforming Growth Factor-β1 Reflect Left Ventricular Remodeling in Aortic Stenosis

Nitric oxide mediates aortic disease in mice deficient in the metalloprotease Adamts1 and in a mouse model of Marfan syndrome

BMP7-based peptide agonists of BMPR1A protect the left ventricle against pathological remodeling induced by pressure overload

Androgens contribute to sex differences in myocardial remodeling under pressure overload by a mechanism involving TGF-β

BAMBI (BMP and activin membrane-bound inhibitor) protects the murine heart from pressure-overload biomechanical stress by restraining TGF-β signaling

Long-term results of the augmented PFNA: a prospective multicenter trial

Extracellular heat shock protein 90 binding to TGFβ receptor I participates in TGFβ-mediated collagen production in myocardial fibroblasts

p-SMAD2/3 and DICER promote pre-miR-21 processing during pressure overload-associated myocardial remodeling

Knockout mutants of Arabidopsis thaliana β-galactosidase. Modifications in the cell wall saccharides and enzymatic activities

Sex-Specific Regulation of miR-29b in the Myocardium Under Pressure Overload is Associated with Differential Molecular, Structural and Functional Remodeling Patterns in Mice and Patients with Aortic Stenosis

Myocardial gene expression of microRNA-133a and myosin heavy and light chains, in conjunction with clinical parameters, predict regression of left ventricular hypertrophy after valve replacement in patients with aortic stenosis

Versican accumulation drives Nos2 induction and aortic disease in Marfan syndrome via Akt activation

Coordinated action of β-galactosidases in the cell wall of embryonic axes during chickpea germination and seedling growth

Circulating Levels of miR‐133a Predict the Regression Potential of Left Ventricular Hypertrophy After Valve Replacement Surgery in Patients With Aortic Stenosis

Selective inhibition of plasma membrane calcium ATPase 4 improves angiogenesis and vascular reperfusion

Gender differences of echocardiographic and gene expression patterns in human pressure overload left ventricular hypertrophy

Infestation of a diabetic foot by Wohlfahrtia magnifica

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