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CHOP is a multifunctional transcription factor in the ER stress response

Journal of biochemistry (Tokyo), 2012-03, Vol.151 (3), p.217-219

Nishitoh, Hideki

2012

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Paradigm shift from 'Compartment' to 'Zone' in the understanding of organelles

Journal of biochemistry (Tokyo), 2019-02, Vol.165 (2), p.97-99

Nishitoh, Hideki

2019

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Endoplasmic reticulum quality control by garbage disposal

The FEBS journal, 2019-01, Vol.286 (2), p.232-240

2019

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Role of the unfolded protein response in the development of central nervous system

Journal of biochemistry (Tokyo), 2017-09, Vol.162 (3), p.155-162

2017

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Signaling pathways from the endoplasmic reticulum and their roles in disease

Genes, 2013-07, Vol.4 (3), p.306-333

2013

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Chemical chaperones ameliorate neurodegenerative disorders in Derlin-1-deficient mice via improvement of cholesterol biosynthesis

Scientific reports, 2022-12, Vol.12 (1), p.21840-21840, Article 21840

2022

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Molecular mechanism of ER stress-induced pre-emptive quality control involving association of the translocon, Derlin-1, and HRD1

Scientific reports, 2018-05, Vol.8 (1), p.7317-11, Article 7317

2018

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ALS-linked mutant SOD1 induces ER stress- and ASK1-dependent motor neuron death by targeting Derlin-1

Genes & development, 2008-06, Vol.22 (11), p.1451-1464

2008

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SOD1 as a Molecular Switch for Initiating the Homeostatic ER Stress Response under Zinc Deficiency

Molecular cell, 2013-10, Vol.52 (1), p.75-86

2013

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ROS-dependent activation of the TRAF6-ASK1-p38 pathway is selectively required for TLR4-mediated innate immunity

Nature immunology, 2005-06, Vol.6 (6), p.587-592

2005

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Role of cytoplasmic acetyltransferases, NAA60 and HAT1, in cellular protection against genotoxic agents

Fundamental toxicological sciences, 2022, Vol.9 (6), p.179-186

2022

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ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats

Genes & development, 2002-06, Vol.16 (11), p.1345-1355

2002

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Roles of MAPKKK ASK1 in Stress-Induced Cell Death

Cell Structure and Function, 2003, Vol.28(1), pp.23-29

2003

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The ASK1-specific inhibitors K811 and K812 prolong survival in a mouse model of amyotrophic lateral sclerosis

Human molecular genetics, 2016-01, Vol.25 (2), p.245-253

2016

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Pre-emptive Quality Control Protects the ER from Protein Overload via the Proximity of ERAD Components and SRP

Cell reports (Cambridge), 2015-11, Vol.13 (5), p.944-956

2015

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Neurodegenerative diseases associated with the disruption of proteostasis and their therapeutic strategies using chemical chaperones

Journal of biochemistry (Tokyo), 2024-09, Vol.176 (3), p.179-186

2024

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A novel monoclonal antibody reveals a conformational alteration shared by amyotrophic lateral sclerosis-linked SOD1 mutants

Annals of neurology, 2012-11, Vol.72 (5), p.739-749

2012

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The Expression of Fn14 via Mechanical Stress-activated JNK Contributes to Apoptosis Induction in Osteoblasts

The Journal of biological chemistry, 2014-03, Vol.289 (10), p.6438-6450

2014

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ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

EMBO reports, 2001-03, Vol.2 (3), p.222-228

2001

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STT3B-Dependent Posttranslational N-Glycosylation as a Surveillance System for Secretory Protein

Molecular cell, 2012-07, Vol.47 (1), p.99-110

2012

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Activation of Apoptosis Signal-Regulating Kinase 1 (ASK1) by Tumor Necrosis Factor Receptor-Associated Factor 2 Requires Prior Dissociation of the ASK1 Inhibitor Thioredoxin

Molecular and Cellular Biology, 2000-03, Vol.20 (6), p.2198-2208

2000

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ASK1 and ASK2 differentially regulate the counteracting roles of apoptosis and inflammation in tumorigenesis

The EMBO journal, 2009-04, Vol.28 (7), p.843-853

2009

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Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression

Biochemical and biophysical research communications, 2015-08, Vol.463 (4), p.870-875

2015

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Physiological roles of ASK1-mediated signal transduction in oxidative stress- and endoplasmic reticulum stress-induced apoptosis: advanced findings from ASK1 knockout mice

Antioxidants & redox signaling, 2002-06, Vol.4 (3), p.415-425

2002

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CHOP is a multifunctional transcription factor in the ER stress response

Paradigm shift from 'Compartment' to 'Zone' in the understanding of organelles

Endoplasmic reticulum quality control by garbage disposal

Role of the unfolded protein response in the development of central nervous system

Signaling pathways from the endoplasmic reticulum and their roles in disease

Chemical chaperones ameliorate neurodegenerative disorders in Derlin-1-deficient mice via improvement of cholesterol biosynthesis

Molecular mechanism of ER stress-induced pre-emptive quality control involving association of the translocon, Derlin-1, and HRD1

ALS-linked mutant SOD1 induces ER stress- and ASK1-dependent motor neuron death by targeting Derlin-1

The Src/c-Abl pathway is a potential therapeutic target in amyotrophic lateral sclerosis

SOD1 as a Molecular Switch for Initiating the Homeostatic ER Stress Response under Zinc Deficiency

ROS-dependent activation of the TRAF6-ASK1-p38 pathway is selectively required for TLR4-mediated innate immunity

Role of cytoplasmic acetyltransferases, NAA60 and HAT1, in cellular protection against genotoxic agents

ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats

Roles of MAPKKK ASK1 in Stress-Induced Cell Death

The ASK1-specific inhibitors K811 and K812 prolong survival in a mouse model of amyotrophic lateral sclerosis

Pre-emptive Quality Control Protects the ER from Protein Overload via the Proximity of ERAD Components and SRP

Neurodegenerative diseases associated with the disruption of proteostasis and their therapeutic strategies using chemical chaperones

A novel monoclonal antibody reveals a conformational alteration shared by amyotrophic lateral sclerosis-linked SOD1 mutants

The Expression of Fn14 via Mechanical Stress-activated JNK Contributes to Apoptosis Induction in Osteoblasts

ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

STT3B-Dependent Posttranslational N-Glycosylation as a Surveillance System for Secretory Protein

Activation of Apoptosis Signal-Regulating Kinase 1 (ASK1) by Tumor Necrosis Factor Receptor-Associated Factor 2 Requires Prior Dissociation of the ASK1 Inhibitor Thioredoxin

ASK1 and ASK2 differentially regulate the counteracting roles of apoptosis and inflammation in tumorigenesis

Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression

Physiological roles of ASK1-mediated signal transduction in oxidative stress- and endoplasmic reticulum stress-induced apoptosis: advanced findings from ASK1 knockout mice

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