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Update on the pathogenesis of bullous pemphigoid: An autoantibody-mediated blistering disease targeting collagen XVII

Journal of dermatological science, 2014-03, Vol.73 (3), p.179-186

Nishie, Wataru

2014

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Collagen XVII Processing and Blistering Skin Diseases

Acta dermato-venereologica, 2020-02, Vol.100 (5), p.adv00054-107

Nishie, Wataru

2020

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Dipeptidyl peptidase IV inhibitor-associated bullous pemphigoid: a recently recognized autoimmune blistering disease with unique clinical, immunological and genetic characteristics

Immunological medicine, 2019-01, Vol.42 (1), p.22-28

Nishie, Wataru

2019

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Dipeptidyl Peptidase-4 Inhibitor-Associated Bullous Pemphigoid

Frontiers in immunology, 2019-06, Vol.10, p.1238

2019

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Autoantibody Profile Differentiates between Inflammatory and Noninflammatory Bullous Pemphigoid

Journal of investigative dermatology, 2016-11, Vol.136 (11), p.2201-2210

2016

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Life before and beyond blistering: The role of collagen XVII in epidermal physiology

Experimental dermatology, 2019-10, Vol.28 (10), p.1135-1141

2019

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Granzyme B inhibition reduces disease severity in autoimmune blistering diseases

Nature communications, 2021-01, Vol.12 (1), p.302-14, Article 302

2021

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In vitro and in vivo models to investigate the pathomechanisms and novel treatments for pemphigoid diseases

Experimental dermatology, 2017-12, Vol.26 (12), p.1163-1170

2017

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Japanese guidelines for the management of pemphigoid (including epidermolysis bullosa acquisita)

Journal of dermatology, 2019-12, Vol.46 (12), p.1102-1135

2019

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HLA-DQB103:01 as a Biomarker for Genetic Susceptibility to Bullous Pemphigoid Induced by DPP-4 Inhibitors

Journal of investigative dermatology, 2018-05, Vol.138 (5), p.1201-1204

2018

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Type XVII collagen coordinates proliferation in the interfollicular epidermis

eLife, 2017-07, Vol.6

2017

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In vivo analysis of IgE autoantibodies in bullous pemphigoid: A study of 100 cases

Journal of dermatological science, 2015-04, Vol.78 (1), p.21-25

2015

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Bullous pemphigoid autoantibodies directly induce blister formation without complement activation

The Journal of immunology (1950), 2014-11, Vol.193 (9), p.4415-4428

2014

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Preferential Reactivity of Dipeptidyl Peptidase-IV Inhibitor-Associated Bullous Pemphigoid Autoantibodies to the Processed Extracellular Domains of BP180

Frontiers in immunology, 2019-05, Vol.10, p.1224

2019

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A Cross-Sectional Study Comparing the Prevalence of Bullous Pemphigoid Autoantibodies in 275 Cases of Type II Diabetes Mellitus Treated With or Without Dipeptidyl Peptidase-IV Inhibitors

Frontiers in immunology, 2019-06, Vol.10, p.1439

2019

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Dipeptidyl peptidase‐4 inhibitors‐associated bullous pemphigoid: A retrospective study of 168 pemphigoid and 9,304 diabetes mellitus patients

Journal of diabetes investigation, 2019-03, Vol.10 (2), p.392-398

2019

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Case of hereditary leiomyomatosis and renal cell cancer showing multiple cutaneous leiomyomas harboring a recurrent nonsense mutation in the fumarate hydratase gene

Journal of dermatology, 2022-01, Vol.49 (1), p.e42-e43

2022

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Dipeptidyl peptidase 4 inhibitor‐associated mucous membrane pemphigoid

Journal of dermatology, 2021-10, Vol.48 (10), p.1584-1587

2021

Volltextzugriff (PDF)

High Expression of Collagen XVII Compensates for its Depletion Induced by Pemphigoid IgG in the Oral Mucosa

Journal of investigative dermatology, 2018-08, Vol.138 (8), p.1707-1715

2018

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The direct binding of collagen XVII and collagen IV is disrupted by pemphigoid autoantibodies

Laboratory investigation, 2019-01, Vol.99 (1), p.48-57

2019

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Antibodies to pathogenic epitopes on type XVII collagen cause skin fragility in a complement-dependent and -independent manner

The Journal of immunology (1950), 2012-06, Vol.188 (11), p.5792-5799

2012

Volltextzugriff (PDF)

Bullous Pemphigoid Triggered by Thermal Burn Under Medication With a Dipeptidyl Peptidase-IV Inhibitor: A Case Report and Review of the Literature

Frontiers in immunology, 2018-04, Vol.9, p.542

2018

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Neonatal Linear IgA Bullous Dermatosis Mediated by Breast Milk-Borne Maternal IgA

JAMA dermatology (Chicago, Ill.), 2021-09, Vol.157 (9), p.1107

2021

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Regression of acquired reactive perforating collagenosis after dipeptidyl peptidase‐4 inhibitor cessation

Journal of dermatology, 2022-07, Vol.49 (7), p.e238-e240

2022

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Fc‐binding proteins enhance autoantibody‐induced BP180 depletion in pemphigoid

The Journal of pathology, 2019-03, Vol.247 (3), p.371-380

2019

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Update on the pathogenesis of bullous pemphigoid: An autoantibody-mediated blistering disease targeting collagen XVII

Collagen XVII Processing and Blistering Skin Diseases

Dipeptidyl peptidase IV inhibitor-associated bullous pemphigoid: a recently recognized autoimmune blistering disease with unique clinical, immunological and genetic characteristics

Dipeptidyl Peptidase-4 Inhibitor-Associated Bullous Pemphigoid

Autoantibody Profile Differentiates between Inflammatory and Noninflammatory Bullous Pemphigoid

Life before and beyond blistering: The role of collagen XVII in epidermal physiology

Granzyme B inhibition reduces disease severity in autoimmune blistering diseases

In vitro and in vivo models to investigate the pathomechanisms and novel treatments for pemphigoid diseases

Japanese guidelines for the management of pemphigoid (including epidermolysis bullosa acquisita)

HLA-DQB103:01 as a Biomarker for Genetic Susceptibility to Bullous Pemphigoid Induced by DPP-4 Inhibitors

Type XVII collagen coordinates proliferation in the interfollicular epidermis

In vivo analysis of IgE autoantibodies in bullous pemphigoid: A study of 100 cases

Bullous pemphigoid autoantibodies directly induce blister formation without complement activation

Preferential Reactivity of Dipeptidyl Peptidase-IV Inhibitor-Associated Bullous Pemphigoid Autoantibodies to the Processed Extracellular Domains of BP180

A Cross-Sectional Study Comparing the Prevalence of Bullous Pemphigoid Autoantibodies in 275 Cases of Type II Diabetes Mellitus Treated With or Without Dipeptidyl Peptidase-IV Inhibitors

Dipeptidyl peptidase‐4 inhibitors‐associated bullous pemphigoid: A retrospective study of 168 pemphigoid and 9,304 diabetes mellitus patients

Case of hereditary leiomyomatosis and renal cell cancer showing multiple cutaneous leiomyomas harboring a recurrent nonsense mutation in the fumarate hydratase gene

Dipeptidyl peptidase 4 inhibitor‐associated mucous membrane pemphigoid

High Expression of Collagen XVII Compensates for its Depletion Induced by Pemphigoid IgG in the Oral Mucosa

The direct binding of collagen XVII and collagen IV is disrupted by pemphigoid autoantibodies

Antibodies to pathogenic epitopes on type XVII collagen cause skin fragility in a complement-dependent and -independent manner

Bullous Pemphigoid Triggered by Thermal Burn Under Medication With a Dipeptidyl Peptidase-IV Inhibitor: A Case Report and Review of the Literature

Neonatal Linear IgA Bullous Dermatosis Mediated by Breast Milk-Borne Maternal IgA

Regression of acquired reactive perforating collagenosis after dipeptidyl peptidase‐4 inhibitor cessation

Fc‐binding proteins enhance autoantibody‐induced BP180 depletion in pemphigoid

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