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Sensitive detection of Aβ protofibrils by proximity ligation--relevance for Alzheimer's disease

BMC neuroscience, 2010-10, Vol.11 (1), p.124-124, Article 124

2010

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Impaired astrocytic Ca2+ signaling in awake-behaving Alzheimer’s disease transgenic mice

eLife, 2022-07, Vol.11

2022

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The CCAAT/enhancer binding protein (C/EBP) δ is differently regulated by fibrillar and oligomeric forms of the Alzheimer amyloid-β peptide

Journal of neuroinflammation, 2011-04, Vol.8 (1), p.34-34, Article 34

2011

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The Uppsala APP deletion causes early onset autosomal dominant Alzheimer's disease by altering APP processing and increasing amyloid β fibril formation

Science translational medicine, 2021-08, Vol.13 (606)

2021

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Sensitive ELISA detection of amyloid-β protofibrils in biological samples

Journal of neurochemistry, 2007-10, Vol.103 (1), p.334-345

2007

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CSF sTREM2 and Tau Work Together in Predicting Increased Temporal Lobe Atrophy in Older Adults

Cerebral cortex (New York, N.Y. 1991), 2020-04, Vol.30 (4), p.2295-2306

2020

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Overexpression of Heparanase Lowers the Amyloid Burden in Amyloid-β Precursor Protein Transgenic Mice

The Journal of biological chemistry, 2015-02, Vol.290 (8), p.5053-5064

2015

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An amyloid-β protofibril-selective antibody prevents amyloid formation in a mouse model of Alzheimer's disease

Neurobiology of disease, 2009-12, Vol.36 (3), p.425-434

2009

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Amyloid-β protofibril levels correlate with spatial learning in Arctic Alzheimer's disease transgenic mice

The FEBS journal, 2009-02, Vol.276 (4), p.995-1006

2009

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Heparanase overexpression impairs inflammatory response and macrophage-mediated clearance of amyloid-β in murine brain

Acta neuropathologica, 2012-10, Vol.124 (4), p.465-478

2012

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Lack of exon 10 in the murine tau gene results in mild sensorimotor defects with aging

BMC neuroscience, 2013-11, Vol.14 (1), p.148-148, Article 148

2013

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The Arctic amyloid-β precursor protein (AβPP) mutation results in distinct plaques and accumulation of N- and C-truncated Aβ

Neurobiology of aging, 2012-05, Vol.33 (5), p.1010.e1-1010.e13

2012

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Appearance of Cxcl10-expressing cell clusters is common for traumatic brain injury and neurodegenerative disorders

The European journal of neuroscience, 2010-03, Vol.31 (5), p.852-863

2010

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Impaired behavior of female tg-ArcSwe APP mice in the IntelliCage: A longitudinal study

Behavioural brain research, 2010, Vol.215 (1), p.83-94

2010

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Genetic deletion and pharmacological inhibition of Nogo-66 receptor impairs cognitive outcome after traumatic brain injury in mice

Journal of neurotrauma, 2010-07, Vol.27 (7), p.1297

2010

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Amyloid-β oligomers are inefficiently measured by enzyme-linked immunosorbent assay

Annals of neurology, 2005-07, Vol.58 (1), p.147-150

2005

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The Arctic Alzheimer mutation favors intracellular amyloid‐β production by making amyloid precursor protein less available to α‐secretase

Journal of neurochemistry, 2007-05, Vol.101 (3), p.854-862

2007

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Heparan Sulfate Accumulation with Aβ Deposits in Alzheimer's Disease and Tg2576 Mice is Contributed by Glial Cells

Brain pathology (Zurich, Switzerland), 2008-10, Vol.18 (4), p.548-561

2008

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The Arctic AβPP mutation leads to Alzheimer's disease pathology with highly variable topographic deposition of differentially truncated Aβ

Acta neuropathologica communications, 2013-01, Vol.1 (1), p.60-60, Article 60

2013

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Contrast Enhanced Magnetic Resonance Imaging of Amyloid-β Plaques in a Murine Alzheimer's Disease Model

Journal of Alzheimer's disease, 2023-01, Vol.93 (2), p.411

2023

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Translating research on brain aging into public health: a new type of immunotherapy for Alzheimer's disease: Nutrition Reviews©, Vol. 68, No. s2

Nutrition reviews, 2010-12, Vol.68, p.S128-S134

2010

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Translating research on brain aging into public health: a new type of immunotherapy for Alzheimer's disease

Nutrition reviews, 2010-12, Vol.68 (-), p.S128

2010

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Translating research on brain aging into public health: a new type of immunotherapy for Alzheimer's disease

Nutrition reviews, 2010-12, Vol.68 (s2), p.S128-S134

2010

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Animal models of amyloid-[beta]-related pathologies in Alzheimer's disease

The FEBS journal, 2010-03, Vol.277 (6), p.1389

2010

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Animal models of amyloid-b-related pathologies in Alzheimer's disease

The FEBS journal, 2010-03, Vol.277 (6), p.1389-1409

2010

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Sensitive detection of Aβ protofibrils by proximity ligation--relevance for Alzheimer's disease

Impaired astrocytic Ca2+ signaling in awake-behaving Alzheimer’s disease transgenic mice

The CCAAT/enhancer binding protein (C/EBP) δ is differently regulated by fibrillar and oligomeric forms of the Alzheimer amyloid-β peptide

The Uppsala APP deletion causes early onset autosomal dominant Alzheimer's disease by altering APP processing and increasing amyloid β fibril formation

Sensitive ELISA detection of amyloid-β protofibrils in biological samples

CSF sTREM2 and Tau Work Together in Predicting Increased Temporal Lobe Atrophy in Older Adults

Overexpression of Heparanase Lowers the Amyloid Burden in Amyloid-β Precursor Protein Transgenic Mice

An amyloid-β protofibril-selective antibody prevents amyloid formation in a mouse model of Alzheimer's disease

Amyloid-β protofibril levels correlate with spatial learning in Arctic Alzheimer's disease transgenic mice

Heparanase overexpression impairs inflammatory response and macrophage-mediated clearance of amyloid-β in murine brain

Lack of exon 10 in the murine tau gene results in mild sensorimotor defects with aging

The Arctic amyloid-β precursor protein (AβPP) mutation results in distinct plaques and accumulation of N- and C-truncated Aβ

Appearance of Cxcl10-expressing cell clusters is common for traumatic brain injury and neurodegenerative disorders

Impaired behavior of female tg-ArcSwe APP mice in the IntelliCage: A longitudinal study

Genetic deletion and pharmacological inhibition of Nogo-66 receptor impairs cognitive outcome after traumatic brain injury in mice

Amyloid-β oligomers are inefficiently measured by enzyme-linked immunosorbent assay

The Arctic Alzheimer mutation favors intracellular amyloid‐β production by making amyloid precursor protein less available to α‐secretase

Heparan Sulfate Accumulation with Aβ Deposits in Alzheimer's Disease and Tg2576 Mice is Contributed by Glial Cells

The Arctic AβPP mutation leads to Alzheimer's disease pathology with highly variable topographic deposition of differentially truncated Aβ

Contrast Enhanced Magnetic Resonance Imaging of Amyloid-β Plaques in a Murine Alzheimer's Disease Model

Translating research on brain aging into public health: a new type of immunotherapy for Alzheimer's disease: Nutrition Reviews©, Vol. 68, No. s2

Translating research on brain aging into public health: a new type of immunotherapy for Alzheimer's disease

Translating research on brain aging into public health: a new type of immunotherapy for Alzheimer's disease

Animal models of amyloid-[beta]-related pathologies in Alzheimer's disease

Animal models of amyloid-b-related pathologies in Alzheimer's disease

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