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Severe acute respiratory syndrome coronavirus envelope protein ion channel activity promotes virus fitness and pathogenesis

PLoS pathogens, 2014-05, Vol.10 (5), p.e1004077-e1004077

2014

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Inhibition of NF-κB-Mediated Inflammation in Severe Acute Respiratory Syndrome Coronavirus-Infected Mice Increases Survival

Journal of Virology, 2014-01, Vol.88 (2), p.913-924

2014

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The PDZ-binding motif of severe acute respiratory syndrome coronavirus envelope protein is a determinant of viral pathogenesis

PLoS pathogens, 2014-08, Vol.10 (8), p.e1004320-e1004320

2014

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Identification of the Mechanisms Causing Reversion to Virulence in an Attenuated SARS-CoV for the Design of a Genetically Stable Vaccine

PLoS pathogens, 2015-10, Vol.11 (10), p.e1005215-e1005215

2015

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LC3B phosphorylation regulates FYCO1 binding and directional transport of autophagosomes

Current biology, 2021-08, Vol.31 (15), p.3440-3449.e7

2021

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Engineering a replication-competent, propagation-defective Middle East respiratory syndrome coronavirus as a vaccine candidate

mBio, 2013-09, Vol.4 (5), p.e00650-e00613

2013

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Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis

PLoS pathogens, 2011-10, Vol.7 (10), p.e1002315

2011

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Macroautophagy and aging: The impact of cellular recycling on health and longevity

Molecular aspects of medicine, 2021-12, Vol.82, p.101020-101020, Article 101020

2021

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Relevance of Viroporin Ion Channel Activity on Viral Replication and Pathogenesis

Viruses, 2015-07, Vol.7 (7), p.3552-3573

2015

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Correction for Castaño-Rodriguez et al., "Role of Severe Acute Respiratory Syndrome Coronavirus Viroporins E, 3a, and 8a in Replication and Pathogenesis"

mBio, 2023-12, Vol.14 (6), p.e0248523

2023

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Beyond Autophagy: The Expanding Roles of ATG8 Proteins

Trends in biochemical sciences (Amsterdam. Regular ed.), 2021-08, Vol.46 (8), p.673-686

2021

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Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease

Virology (New York, N.Y.), 2010-03, Vol.399 (1), p.120-128

2010

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Severe acute respiratory syndrome coronavirus E protein transports calcium ions and activates the NLRP3 inflammasome

Virology (New York, N.Y.), 2015-11, Vol.485, p.330-339

2015

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Post-translational modifications of ATG8 proteins - an emerging mechanism of autophagy control

Journal of cell science, 2023-08, Vol.136 (16)

2023

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LC3B phosphorylation: autophagosome's ticket for a ride toward the cell nucleus

Autophagy, 2021-10, Vol.17 (10), p.3266-3268

2021

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Severe acute respiratory syndrome coronaviruses with mutations in the E protein are attenuated and promising vaccine candidates

Journal of virology, 2015-04, Vol.89 (7), p.3870-3887

2015

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Coronavirus E protein forms ion channels with functionally and structurally-involved membrane lipids

Virology (New York, N.Y.), 2012-10, Vol.432 (2), p.485-494

2012

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Coronavirus virulence genes with main focus on SARS-CoV envelope gene

Virus research, 2014-12, Vol.194, p.124-137

2014

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Subcellular location and topology of severe acute respiratory syndrome coronavirus envelope protein

Virology (New York, N.Y.), 2011-07, Vol.415 (2), p.69-82

2011

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Analysis of SARS-CoV E protein ion channel activity by tuning the protein and lipid charge

Biochimica et biophysica acta, 2013-09, Vol.1828 (9), p.2026-2031

2013

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The Autophagy, Inflammation and Metabolism Center international eSymposium - an early-career investigators' seminar series during the COVID-19 pandemic

Journal of cell science, 2021-10, Vol.134 (19)

2021

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The envelope protein of severe acute respiratory syndrome coronavirus interacts with the non-structural protein 3 and is ubiquitinated

Virology (New York, N.Y.), 2010-07, Vol.402 (2), p.281-291

2010

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The replication of a mouse adapted SARS-CoV in a mouse cell line stably expressing the murine SARS-CoV receptor mACE2 efficiently induces the expression of proinflammatory cytokines

Journal of virological methods, 2013-11, Vol.193 (2), p.639-646

2013

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Abstract B20: STK3/4-mediated phosphorylation of LC3B regulates directional intracellular transport of autophagic vesicles

Molecular cancer research, 2020-08, Vol.18 (8_Supplement), p.B20-B20

2020

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Severe Acute Respiratory Syndrome - Virology
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Viroporin Proteins
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Severe acute respiratory syndrome coronavirus envelope protein ion channel activity promotes virus fitness and pathogenesis

Inhibition of NF-κB-Mediated Inflammation in Severe Acute Respiratory Syndrome Coronavirus-Infected Mice Increases Survival

Role of Severe Acute Respiratory Syndrome Coronavirus Viroporins E, 3a, and 8a in Replication and Pathogenesis

The PDZ-binding motif of severe acute respiratory syndrome coronavirus envelope protein is a determinant of viral pathogenesis

Identification of the Mechanisms Causing Reversion to Virulence in an Attenuated SARS-CoV for the Design of a Genetically Stable Vaccine

LC3B phosphorylation regulates FYCO1 binding and directional transport of autophagosomes

Engineering a replication-competent, propagation-defective Middle East respiratory syndrome coronavirus as a vaccine candidate

Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis

Macroautophagy and aging: The impact of cellular recycling on health and longevity

Relevance of Viroporin Ion Channel Activity on Viral Replication and Pathogenesis

Correction for Castaño-Rodriguez et al., "Role of Severe Acute Respiratory Syndrome Coronavirus Viroporins E, 3a, and 8a in Replication and Pathogenesis"

Beyond Autophagy: The Expanding Roles of ATG8 Proteins

Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease

Severe acute respiratory syndrome coronavirus E protein transports calcium ions and activates the NLRP3 inflammasome

Post-translational modifications of ATG8 proteins - an emerging mechanism of autophagy control

LC3B phosphorylation: autophagosome's ticket for a ride toward the cell nucleus

Severe acute respiratory syndrome coronaviruses with mutations in the E protein are attenuated and promising vaccine candidates

Coronavirus E protein forms ion channels with functionally and structurally-involved membrane lipids

Coronavirus virulence genes with main focus on SARS-CoV envelope gene

Subcellular location and topology of severe acute respiratory syndrome coronavirus envelope protein

Analysis of SARS-CoV E protein ion channel activity by tuning the protein and lipid charge

The Autophagy, Inflammation and Metabolism Center international eSymposium - an early-career investigators' seminar series during the COVID-19 pandemic

The envelope protein of severe acute respiratory syndrome coronavirus interacts with the non-structural protein 3 and is ubiquitinated

The replication of a mouse adapted SARS-CoV in a mouse cell line stably expressing the murine SARS-CoV receptor mACE2 efficiently induces the expression of proinflammatory cytokines

Abstract B20: STK3/4-mediated phosphorylation of LC3B regulates directional intracellular transport of autophagic vesicles

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