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Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells

PLoS pathogens, 2022-04, Vol.18 (4), p.e1010453-e1010453

2022

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EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice

PLoS pathogens, 2020-06, Vol.16 (6), p.e1008590

2020

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Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus (EBV) lytic reactivation through TEADs in epithelial cells

PLoS pathogens, 2021-08, Vol.17 (8), p.e1009783-e1009783

2021

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Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice

PLoS pathogens, 2024-04, Vol.20 (4), p.e1012132-e1012132

2024

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B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection

PLoS pathogens, 2020-02, Vol.16 (2), p.e1008365-e1008365

2020

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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868-e1010868

2022

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Plasma membrane proteoglycans syndecan-2 and syndecan-4 engage with EGFR and RON kinase to sustain carcinoma cell cycle progression

The Journal of biological chemistry, 2022-06, Vol.298 (6), p.102029-102029, Article 102029

2022

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Epstein-Barr virus LMP1 protein promotes proliferation and inhibits differentiation of epithelial cells via activation of YAP and TAZ

Proceedings of the National Academy of Sciences - PNAS, 2023-05, Vol.120 (20), p.e2219755120-e2219755120

2023

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An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse Model

Journal of virology, 2020-05, Vol.94 (10)

2020

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The gonadotropin releasing hormone (GnRH) receptor activating sequence (GRAS) is a composite regulatory element that interacts with multiple classes of transcription factors including Smads, AP-1 and a forkhead DNA binding protein

Molecular and cellular endocrinology, 2003-08, Vol.206 (1), p.93-111

2003

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Detemir as a once-daily basal insulin in type 2 diabetes

Clinical pharmacology: advances and applications, 2011-01, Vol.3 (default), p.27-37

Nelson, Scott E

2011

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Truncation of the C-terminus of human MLH1 blocks intracellular stabilization of PMS2 and disrupts DNA mismatch repair

DNA repair, 2006-03, Vol.5 (3), p.347-361

2006

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Binding of Agonist but Not Antagonist Leads to Fluorescence Resonance Energy Transfer between Intrinsically Fluorescent Gonadotropin-Releasing Hormone Receptors

Molecular endocrinology (Baltimore, Md.), 2001-05, Vol.15 (5), p.695-703

2001

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Addition of Insulin to Oral Therapy in Patients with Type 2 Diabetes

The American journal of the medical sciences, 2006-05, Vol.331 (5), p.257-263

2006

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Homer1a and 1bc levels in the rat somatosensory cortex vary with the time of day and sleep loss

Neuroscience letters, 2004-08, Vol.367 (1), p.105-108

2004

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Changes in the expression of the NR2B subunit during aging in macaque monkeys

Neurobiology of aging, 2004-02, Vol.25 (2), p.201-208

2004

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Beneficial effects of insulin on endothelial function, inflammation, and atherogenesis and their implications

The Journal of family practice, 2005-06, Vol.54 (6), p.S7-S9

2005

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Assessing the controversies of insulin therapy in patients with type 2 diabetes mellitus

The Journal of family practice, 2005-06, Vol.54 (6), p.S3

2005

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Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus

PLoS pathogens, 2021-08, Vol.17 (8)

2021

Volltextzugriff (PDF)

The Tripartite Basal Enhancer of the Gonadotropin-Releasing Hormone (GnRH) Receptor Gene Promoter Regulates Cell-Specific Expression Through a Novel GnRH Receptor Activating Sequence

Molecular endocrinology (Baltimore, Md.), 1997-11, Vol.11 (12), p.1814-1821

1997

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Insulin therapy in primary care: practical issues for clinicians

The Journal of family practice, 2005-06, Vol.54 (6), p.S10-S11

2005

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Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells

PLoS pathogens, 2022-04, Vol.18 (4)

2022

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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868

2022

Volltextzugriff (PDF)

B cells infected with Type 2 Epstein-Barr virus

PLoS pathogens, 2020-02, Vol.16 (2)

2020

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Confirmation of confocal microscopy diagnosis of Acanthamoeba keratitis using polymerase chain reaction analysis

Archives of ophthalmology (1960), 2000-02, Vol.118 (2), p.178

2000

Volltextzugriff (PDF)

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Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells

EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice

Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus (EBV) lytic reactivation through TEADs in epithelial cells

Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice

B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection

Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

Plasma membrane proteoglycans syndecan-2 and syndecan-4 engage with EGFR and RON kinase to sustain carcinoma cell cycle progression

Epstein-Barr virus LMP1 protein promotes proliferation and inhibits differentiation of epithelial cells via activation of YAP and TAZ

An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse Model

The gonadotropin releasing hormone (GnRH) receptor activating sequence (GRAS) is a composite regulatory element that interacts with multiple classes of transcription factors including Smads, AP-1 and a forkhead DNA binding protein

Detemir as a once-daily basal insulin in type 2 diabetes

Truncation of the C-terminus of human MLH1 blocks intracellular stabilization of PMS2 and disrupts DNA mismatch repair

Binding of Agonist but Not Antagonist Leads to Fluorescence Resonance Energy Transfer between Intrinsically Fluorescent Gonadotropin-Releasing Hormone Receptors

Addition of Insulin to Oral Therapy in Patients with Type 2 Diabetes

Homer1a and 1bc levels in the rat somatosensory cortex vary with the time of day and sleep loss

Changes in the expression of the NR2B subunit during aging in macaque monkeys

Beneficial effects of insulin on endothelial function, inflammation, and atherogenesis and their implications

Assessing the controversies of insulin therapy in patients with type 2 diabetes mellitus

Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus

The Tripartite Basal Enhancer of the Gonadotropin-Releasing Hormone (GnRH) Receptor Gene Promoter Regulates Cell-Specific Expression Through a Novel GnRH Receptor Activating Sequence

Insulin therapy in primary care: practical issues for clinicians

Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells

Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

B cells infected with Type 2 Epstein-Barr virus

Confirmation of confocal microscopy diagnosis of Acanthamoeba keratitis using polymerase chain reaction analysis

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