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N-acetylaspartylglutamate (NAAG) and glutamate carboxypeptidase II: An abundant peptide neurotransmitter-enzyme system with multiple clinical applications

Progress in neurobiology, 2020-01, Vol.184, p.101722, Article 101722

2020

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A role for N-acetylaspartylglutamate (NAAG) and mGluR3 in cognition

Neurobiology of learning and memory, 2019-02, Vol.158, p.9-13

2019

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N‐Acetylaspartylglutamate is an agonist at mGluR3 in vivo and in vitro

Journal of neurochemistry, 2011-12, Vol.119 (5), p.891-895

Neale, Joseph H.

2011

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Predation yields greater population performance: what are the contributions of density‐ and trait‐mediated effects?

Ecological entomology, 2021-02, Vol.46 (1), p.56-65

2021

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Endogenous N-Acetylaspartylglutamate (NAAG) Inhibits Synaptic Plasticity/Transmission in the Amygdala in a Mouse Inflammatory Pain Model

Molecular pain, 2010-09, Vol.6 (1), p.60-60

2010

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Mice lacking glutamate carboxypeptidase II develop normally, but are less susceptible to traumatic brain injury

Journal of neurochemistry, 2015-07, Vol.134 (2), p.340-353

2015

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Advances in understanding the peptide neurotransmitter NAAG and appearance of a new member of the NAAG neuropeptide family

Journal of neurochemistry, 2011-08, Vol.118 (4), p.490-498

2011

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Finding the sweet spot: What levels of larval mortality lead to compensation or overcompensation in adult production?

Ecosphere (Washington, D.C), 2019-09, Vol.10 (9), p.n/a

2019

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Glutamate carboxypeptidase II gene knockout attenuates oxidative stress and cortical apoptosis after traumatic brain injury

BMC neuroscience, 2016-04, Vol.17 (15), p.15-15, Article 15

2016

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Effects of N-Acetylaspartylglutamate (NAAG) Peptidase Inhibition on Release of Glutamate and Dopamine in Prefrontal Cortex and Nucleus Accumbens in Phencyclidine Model of Schizophrenia

The Journal of biological chemistry, 2012-06, Vol.287 (26), p.21773-21782

2012

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Parkin-mediated reduction of nuclear and soluble TDP-43 reverses behavioral decline in symptomatic mice

Human molecular genetics, 2014-09, Vol.23 (18), p.4960-4969

2014

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NAAG peptidase inhibitors and their potential for diagnosis and therapy

Nature reviews. Drug discovery, 2005-12, Vol.4 (12), p.1015-1026

2005

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mGluR3 and not mGluR2 receptors mediate the efficacy of NAAG peptidase inhibitor in validated model of schizophrenia

Schizophrenia research, 2012-04, Vol.136 (1), p.160-161

2012

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NAAG Peptidase Inhibition in the Periaqueductal Gray and Rostral Ventromedial Medulla Reduces Flinching in the Formalin Model of Inflammation

Molecular pain, 2012-09, Vol.8 (1), p.67-67

2012

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N-acetylaspartylglutamate : The most abundant peptide neurotransmitter in the mammalian central nervous system

Journal of neurochemistry, 2000-08, Vol.75 (2), p.443-452

2000

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NAAG Peptidase Inhibitors Act via mGluR3: Animal Models of Memory, Alzheimer’s, and Ethanol Intoxication

Neurochemical research, 2017-09, Vol.42 (9), p.2646-2657

2017

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Synthesis of Urea-Based Inhibitors as Active Site Probes of Glutamate Carboxypeptidase II: Efficacy as Analgesic Agents

Journal of medicinal chemistry, 2004-03, Vol.47 (7), p.1729-1738

2004

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A role for the locus coeruleus in the analgesic efficacy of N-acetylaspartylglutamate peptidase (GCPII) inhibitors ZJ43 and 2-PMPA

Molecular pain, 2017, Vol.13, p.1744806917697008

2017

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The neurotransmitter N-acetylaspartylglutamate in models of pain, ALS, diabetic neuropathy, CNS injury and schizophrenia

Trends in pharmacological sciences (Regular ed.), 2005-09, Vol.26 (9), p.477-484

2005

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Immunohistological and electrophysiological evidence that N-acetylaspartylglutamate is a co-transmitter at the vertebrate neuromuscular junction

The European journal of neuroscience, 2013-01, Vol.37 (1), p.118-129

2013

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Intracerebroventricular Administration of N-Acetylaspartylglutamate (NAAG) Peptidase Inhibitors is Analgesic in Inflammatory Pain

Molecular pain, 2008-08, Vol.4 (1), p.31-31

2008

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Design of Remarkably Simple, Yet Potent Urea-Based Inhibitors of Glutamate Carboxypeptidase II (NAALADase)

Journal of medicinal chemistry, 2001-02, Vol.44 (3), p.298-301

2001

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NAAG peptidase inhibitor increases dialysate NAAG and reduces glutamate, aspartate and GABA levels in the dorsal hippocampus following fluid percussion injury in the rat

Journal of neurochemistry, 2006-05, Vol.97 (4), p.1015-1025

2006

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NAAG peptidase inhibitors and deletion of NAAG peptidase gene enhance memory in novel object recognition test

European journal of pharmacology, 2013-02, Vol.701 (1-3), p.27-32

2013

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Antinociceptive effects of N-acetylaspartylglutamate (NAAG) peptidase inhibitors ZJ-11, ZJ-17 and ZJ-43 in the rat formalin test and in the rat neuropathic pain model

The European journal of neuroscience, 2004-07, Vol.20 (2), p.483-494

2004

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N-acetylaspartylglutamate (NAAG) and glutamate carboxypeptidase II: An abundant peptide neurotransmitter-enzyme system with multiple clinical applications

A role for N-acetylaspartylglutamate (NAAG) and mGluR3 in cognition

N‐Acetylaspartylglutamate is an agonist at mGluR3 in vivo and in vitro

Predation yields greater population performance: what are the contributions of density‐ and trait‐mediated effects?

Endogenous N-Acetylaspartylglutamate (NAAG) Inhibits Synaptic Plasticity/Transmission in the Amygdala in a Mouse Inflammatory Pain Model

Mice lacking glutamate carboxypeptidase II develop normally, but are less susceptible to traumatic brain injury

Advances in understanding the peptide neurotransmitter NAAG and appearance of a new member of the NAAG neuropeptide family

Finding the sweet spot: What levels of larval mortality lead to compensation or overcompensation in adult production?

Glutamate carboxypeptidase II gene knockout attenuates oxidative stress and cortical apoptosis after traumatic brain injury

Effects of N-Acetylaspartylglutamate (NAAG) Peptidase Inhibition on Release of Glutamate and Dopamine in Prefrontal Cortex and Nucleus Accumbens in Phencyclidine Model of Schizophrenia

Parkin-mediated reduction of nuclear and soluble TDP-43 reverses behavioral decline in symptomatic mice

NAAG peptidase inhibitors and their potential for diagnosis and therapy

mGluR3 and not mGluR2 receptors mediate the efficacy of NAAG peptidase inhibitor in validated model of schizophrenia

NAAG Peptidase Inhibition in the Periaqueductal Gray and Rostral Ventromedial Medulla Reduces Flinching in the Formalin Model of Inflammation

N-acetylaspartylglutamate : The most abundant peptide neurotransmitter in the mammalian central nervous system

NAAG Peptidase Inhibitors Act via mGluR3: Animal Models of Memory, Alzheimer’s, and Ethanol Intoxication

Synthesis of Urea-Based Inhibitors as Active Site Probes of Glutamate Carboxypeptidase II: Efficacy as Analgesic Agents

A role for the locus coeruleus in the analgesic efficacy of N-acetylaspartylglutamate peptidase (GCPII) inhibitors ZJ43 and 2-PMPA

The neurotransmitter N-acetylaspartylglutamate in models of pain, ALS, diabetic neuropathy, CNS injury and schizophrenia

Immunohistological and electrophysiological evidence that N-acetylaspartylglutamate is a co-transmitter at the vertebrate neuromuscular junction

Intracerebroventricular Administration of N-Acetylaspartylglutamate (NAAG) Peptidase Inhibitors is Analgesic in Inflammatory Pain

Design of Remarkably Simple, Yet Potent Urea-Based Inhibitors of Glutamate Carboxypeptidase II (NAALADase)

NAAG peptidase inhibitor increases dialysate NAAG and reduces glutamate, aspartate and GABA levels in the dorsal hippocampus following fluid percussion injury in the rat

NAAG peptidase inhibitors and deletion of NAAG peptidase gene enhance memory in novel object recognition test

Antinociceptive effects of N-acetylaspartylglutamate (NAAG) peptidase inhibitors ZJ-11, ZJ-17 and ZJ-43 in the rat formalin test and in the rat neuropathic pain model

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