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Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization

Brain (London, England : 1878), 2013-09, Vol.136 (Pt 9), p.2677-2696

2013

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Long-term effects of Aβ42 immunisation in Alzheimer's disease: follow-up of a randomised, placebo-controlled phase I trial

The Lancet (British edition), 2008-07, Vol.372 (9634), p.216-223

2008

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CD93 regulates central nervous system inflammation in two mouse models of autoimmune encephalomyelitis

Immunology, 2018-11, Vol.155 (3), p.346-355

2018

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Complement Activation and Up-Regulated Expression of Anaphylatoxin C3a/C3aR in Glioblastoma: Deciphering the Links with TGF-β and VEGF

Cancers, 2023-05, Vol.15 (9), p.2647

2023

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C3-dependent mechanism of microglial priming relevant to multiple sclerosis

Proceedings of the National Academy of Sciences - PNAS, 2012-01, Vol.109 (3), p.965-970

2012

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Complement activation and increased anaphylatoxin receptor expression are associated with cortical grey matter lesions and the compartmentalised inflammatory response of multiple sclerosis

Frontiers in cellular neuroscience, 2023-03, Vol.17, p.1094106-1094106

2023

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Complement activation in multiple sclerosis plaques: an immunohistochemical analysis

Acta neuropathologica communications, 2014-05, Vol.2 (1), p.53, Article 53

2014

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Persistent neuropathological effects 14 years following amyloid-β immunization in Alzheimer's disease

Brain (London, England : 1878), 2019-07, Vol.142 (7), p.2113-2126

2019

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Mixed pathologies including chronic traumatic encephalopathy account for dementia in retired association football (soccer) players

Acta neuropathologica, 2017-03, Vol.133 (3), p.337-352

2017

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Meningeal inflammation and cortical demyelination in acute multiple sclerosis

Annals of neurology, 2018-12, Vol.84 (6), p.829-842

2018

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The Multiple Roles of the Innate Immune System in the Regulation of Apoptosis and Inflammation in the Brain

Journal of neuropathology and experimental neurology, 2009-03, Vol.68 (3), p.217-226

2009

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Comparing the Pathology, Clinical, and Demographic Characteristics of Younger and Older‐Onset Multiple Sclerosis

Annals of neurology, 2024-03, Vol.95 (3), p.471-486

2024

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How Does the Brain Limit the Severity of Inflammation and Tissue Injury During Bacterial Meningitis?

Journal of neuropathology and experimental neurology, 2013-05, Vol.72 (5), p.370-385

2013

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The association between neurodegeneration and local complement activation in the thalamus to progressive multiple sclerosis outcome

Brain pathology (Zurich, Switzerland), 2022-09, Vol.32 (5), p.e13054-n/a

2022

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Complement is activated in progressive multiple sclerosis cortical grey matter lesions

Journal of neuroinflammation, 2016-06, Vol.13 (1), p.161-161, Article 161

2016

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Tissue microarray methodology identifies complement pathway activation and dysregulation in progressive multiple sclerosis

Brain pathology (Zurich, Switzerland), 2018-07, Vol.28 (4), p.507-520

2018

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Spontaneous Classical Pathway Activation and Deficiency of Membrane Regulators Render Human Neurons Susceptible to Complement Lysis

The American journal of pathology, 2000-09, Vol.157 (3), p.905-918

2000

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Recent Advances in the Genetics of the ALS-FTLD Complex

Current neurology and neuroscience reports, 2012-06, Vol.12 (3), p.243-250

2012

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The Membrane Attack Complex of Complement Causes Severe Demyelination Associated with Acute Axonal Injury

The Journal of immunology (1950), 2002-01, Vol.168 (1), p.458-465

2002

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Aberrant Interactions of Transcriptional Repressor Proteins with the Huntington's Disease Gene Product, Huntingtin

Human molecular genetics, 1999-09, Vol.8 (9), p.1647-1655

1999

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Familial early onset frontotemporal dementia caused by a novel S356T MAPT mutation, initially diagnosed as schizophrenia

Clinical neurology and neurosurgery, 2010-12, Vol.112 (10), p.917-920

2010

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Aβ42 immunization reduces both tau and Aβ deposits in Alzheimer's disease

Alzheimer's & dementia, 2010-07, Vol.6 (4), p.S144-S144

2010

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Expression of the receptor for complement C5a (CD88) is up-regulated on reactive astrocytes, microglia, and endothelial cells in the inflamed human central nervous system

The American journal of pathology, 1997-01, Vol.150 (1), p.31-41

1997

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C3d Binding to the Myelin Oligodendrocyte Glycoprotein Results in an Exacerbated Experimental Autoimmune Encephalomyelitis

Journal of Immunology, 2007-03, Vol.178 (5), p.3323-3331

2007

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C3-dependent microglial priming caused by deletion of the membrane complement regulator Crry

Molecular immunology, 2010-08, Vol.47 (13), p.2207-2208

2010

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Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization

Long-term effects of Aβ42 immunisation in Alzheimer's disease: follow-up of a randomised, placebo-controlled phase I trial

CD93 regulates central nervous system inflammation in two mouse models of autoimmune encephalomyelitis

Complement Activation and Up-Regulated Expression of Anaphylatoxin C3a/C3aR in Glioblastoma: Deciphering the Links with TGF-β and VEGF

C3-dependent mechanism of microglial priming relevant to multiple sclerosis

Complement activation and increased anaphylatoxin receptor expression are associated with cortical grey matter lesions and the compartmentalised inflammatory response of multiple sclerosis

Complement activation in multiple sclerosis plaques: an immunohistochemical analysis

Persistent neuropathological effects 14 years following amyloid-β immunization in Alzheimer's disease

Mixed pathologies including chronic traumatic encephalopathy account for dementia in retired association football (soccer) players

Meningeal inflammation and cortical demyelination in acute multiple sclerosis

The Multiple Roles of the Innate Immune System in the Regulation of Apoptosis and Inflammation in the Brain

Comparing the Pathology, Clinical, and Demographic Characteristics of Younger and Older‐Onset Multiple Sclerosis

How Does the Brain Limit the Severity of Inflammation and Tissue Injury During Bacterial Meningitis?

The association between neurodegeneration and local complement activation in the thalamus to progressive multiple sclerosis outcome

Complement is activated in progressive multiple sclerosis cortical grey matter lesions

Tissue microarray methodology identifies complement pathway activation and dysregulation in progressive multiple sclerosis

Spontaneous Classical Pathway Activation and Deficiency of Membrane Regulators Render Human Neurons Susceptible to Complement Lysis

Recent Advances in the Genetics of the ALS-FTLD Complex

The Membrane Attack Complex of Complement Causes Severe Demyelination Associated with Acute Axonal Injury

Aberrant Interactions of Transcriptional Repressor Proteins with the Huntington's Disease Gene Product, Huntingtin

Familial early onset frontotemporal dementia caused by a novel S356T MAPT mutation, initially diagnosed as schizophrenia

Aβ42 immunization reduces both tau and Aβ deposits in Alzheimer's disease

Expression of the receptor for complement C5a (CD88) is up-regulated on reactive astrocytes, microglia, and endothelial cells in the inflamed human central nervous system

C3d Binding to the Myelin Oligodendrocyte Glycoprotein Results in an Exacerbated Experimental Autoimmune Encephalomyelitis

C3-dependent microglial priming caused by deletion of the membrane complement regulator Crry

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