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Acute transient cognitive dysfunction and acute brain injury induced by systemic inflammation occur by dissociable IL-1-dependent mechanisms

Molecular psychiatry, 2019-10, Vol.24 (10), p.1533-1548

2019

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RIG-I mediates innate immune response in mouse neurons following Japanese encephalitis virus infection

PloS one, 2011-06, Vol.6 (6), p.e21761-e21761

2011

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Role of pattern recognition receptors in flavivirus infections

Virus research, 2014-06, Vol.185, p.32-40

2014

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Chronic neurodegeneration induces type I interferon synthesis via STING, shaping microglial phenotype and accelerating disease progression

Glia, 2019-07, Vol.67 (7), p.1254-1276

2019

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Glioprotective Effects of Ashwagandha Leaf Extract against Lead Induced Toxicity

BioMed research international, 2014-01, Vol.2014, p.182029-15

2014

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A common carcinogen benzo[a]pyrene causes neuronal death in mouse via microglial activation

PloS one, 2010-04, Vol.5 (4), p.e9984

2010

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TLR7 is a key regulator of innate immunity against Japanese encephalitis virus infection

Neurobiology of disease, 2014-09, Vol.69, p.235-247

2014

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Hyperthermia elevates brain temperature and improves behavioural signs in animal models of autism spectrum disorder

Molecular autism, 2023-11, Vol.14 (1), p.43-43, Article 43

2023

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Correction: Acute transient cognitive dysfunction and acute brain injury induced by systemic inflammation occur by dissociable IL-1-dependent mechanisms

Molecular psychiatry, 2019-10, Vol.24 (10), p.1566-1566

2019

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Antiviral and neuroprotective role of octaguanidinium dendrimer-conjugated morpholino oligomers in Japanese encephalitis

PLoS neglected tropical diseases, 2010-11, Vol.4 (11), p.e892

2010

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γδT cells but not αβT cells contribute to sepsis-induced white matter injury and motor abnormalities in mice

Journal of neuroinflammation, 2017-12, Vol.14 (1), p.255-255, Article 255

2017

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Type 2 Innate Lymphoid Cells Accumulate in the Brain After Hypoxia-Ischemia but Do Not Contribute to the Development of Preterm Brain Injury

Frontiers in cellular neuroscience, 2020-08, Vol.14, p.249-249

2020

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Lymphocytes Contribute to the Pathophysiology of Neonatal Brain Injury

Frontiers in neurology, 2018-03, Vol.9, p.159-159

2018

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Minocycline differentially modulates macrophage mediated peripheral immune response following Japanese encephalitis virus infection

Immunobiology (1979), 2010-11, Vol.215 (11), p.884-893

2010

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STING mediates neuronal innate immune response following Japanese encephalitis virus infection

Scientific reports, 2012-04, Vol.2 (1), p.347-347, Article 347

2012

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Acute systemic inflammation exacerbates neuroinflammation in Alzheimer's disease: IL‐1β drives amplified responses in primed astrocytes and neuronal network dysfunction

Alzheimer's & dementia, 2021-10, Vol.17 (10), p.1735-1755

2021

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Japanese encephalitis virus infection modulates the expression of suppressors of cytokine signaling (SOCS) in macrophages: Implications for the hosts’ innate immune response

Cellular immunology, 2013-09, Vol.285 (1-2), p.100-110

2013

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γδ T Cells Contribute to Injury in the Developing Brain

The American journal of pathology, 2018-03, Vol.188 (3), p.757-767

2018

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Neurons under viral attack: Victims or warriors?

Neurochemistry international, 2010-05, Vol.56 (6), p.727-735

2010

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RIG-I knockdown impedes neurogenesis in a murine model of Japanese encephalitis

Cell biology international, 2015-02, Vol.39 (2), p.224-229

2015

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Japanese Encephalitis Virus-Infected Macrophages Induce Neuronal Death

Journal of neuroimmune pharmacology, 2011-09, Vol.6 (3), p.420-433

2011

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Neural stem/progenitor cells induce conversion of encephalitogenic T cells into CD4+-CD25+- FOXP3+ regulatory T cells

Viral immunology, 2014-03, Vol.27 (2), p.48

2014

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[delta]T cells but not [alpha][beta]T cells contribute to sepsis-induced white matter injury and motor abnormalities in mice

Journal of neuroinflammation, 2017-12, Vol.14 (1)

2017

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Minocycline Differentially Modulates Viral Infection and Persistence in an Experimental Model of Japanese Encephalitis

Journal of neuroimmune pharmacology, 2010-12, Vol.5 (4), p.553-565

2010

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Chemotherapy in Japanese encephalitis: are we there yet?

Infectious disorders drug targets, 2011-06, Vol.11 (3), p.300

2011

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Acute transient cognitive dysfunction and acute brain injury induced by systemic inflammation occur by dissociable IL-1-dependent mechanisms

RIG-I mediates innate immune response in mouse neurons following Japanese encephalitis virus infection

Role of pattern recognition receptors in flavivirus infections

Chronic neurodegeneration induces type I interferon synthesis via STING, shaping microglial phenotype and accelerating disease progression

Glioprotective Effects of Ashwagandha Leaf Extract against Lead Induced Toxicity

A common carcinogen benzo[a]pyrene causes neuronal death in mouse via microglial activation

TLR7 is a key regulator of innate immunity against Japanese encephalitis virus infection

Hyperthermia elevates brain temperature and improves behavioural signs in animal models of autism spectrum disorder

Correction: Acute transient cognitive dysfunction and acute brain injury induced by systemic inflammation occur by dissociable IL-1-dependent mechanisms

Antiviral and neuroprotective role of octaguanidinium dendrimer-conjugated morpholino oligomers in Japanese encephalitis

γδT cells but not αβT cells contribute to sepsis-induced white matter injury and motor abnormalities in mice

Type 2 Innate Lymphoid Cells Accumulate in the Brain After Hypoxia-Ischemia but Do Not Contribute to the Development of Preterm Brain Injury

Lymphocytes Contribute to the Pathophysiology of Neonatal Brain Injury

Minocycline differentially modulates macrophage mediated peripheral immune response following Japanese encephalitis virus infection

STING mediates neuronal innate immune response following Japanese encephalitis virus infection

Acute systemic inflammation exacerbates neuroinflammation in Alzheimer's disease: IL‐1β drives amplified responses in primed astrocytes and neuronal network dysfunction

Japanese encephalitis virus infection modulates the expression of suppressors of cytokine signaling (SOCS) in macrophages: Implications for the hosts’ innate immune response

γδ T Cells Contribute to Injury in the Developing Brain

Neurons under viral attack: Victims or warriors?

RIG-I knockdown impedes neurogenesis in a murine model of Japanese encephalitis

Japanese Encephalitis Virus-Infected Macrophages Induce Neuronal Death

Neural stem/progenitor cells induce conversion of encephalitogenic T cells into CD4+-CD25+- FOXP3+ regulatory T cells

[delta]T cells but not [alpha][beta]T cells contribute to sepsis-induced white matter injury and motor abnormalities in mice

Minocycline Differentially Modulates Viral Infection and Persistence in an Experimental Model of Japanese Encephalitis

Chemotherapy in Japanese encephalitis: are we there yet?

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