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Glucose Oxidation Modulates Anoikis and Tumor Metastasis

Molecular and Cellular Biology, 2012-05, Vol.32 (10), p.1893-1907

2012

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Synergy between rapamycin and FLT3 ligand enhances plasmacytoid dendritic cell–dependent induction of CD4+CD25+FoxP3+ Treg

Blood, 2015-05, Vol.125 (19), p.2937-2947

2015

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B-Cell Depletion and Immunomodulation before Initiation of Enzyme Replacement Therapy Blocks the Immune Response to Acid Alpha-Glucosidase in Infantile-Onset Pompe Disease

The Journal of pediatrics, 2013-09, Vol.163 (3), p.847-854.e1

2013

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Sustained Correction of Motoneuron Histopathology Following Intramuscular Delivery of AAV in Pompe Mice

Molecular therapy, 2014-04, Vol.22 (4), p.702-712

2014

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GITR engagement preferentially enhances proliferation of functionally competent CD4+CD25+FoxP3+ regulatory T cells

International immunology, 2010-04, Vol.22 (4), p.259-270

2010

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Impact of the Underlying Mutation and the Route of Vector Administration on Immune Responses to Factor IX in Gene Therapy for Hemophilia B

Molecular therapy, 2009-10, Vol.17 (10), p.1733-1742

2009

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Targeted approaches to induce immune tolerance for Pompe disease therapy

Molecular therapy. Methods & clinical development, 2016-01, Vol.3 (C), p.15053-15053, Article 15053

2016

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Immune responses and hypercoagulation in ERT for Pompe disease are mutation and rhGAA dose dependent

PloS one, 2014-06, Vol.9 (6), p.e98336

2014

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Tolerance induction to cytoplasmic beta-galactosidase by hepatic AAV gene transfer: implications for antigen presentation and immunotoxicity

PloS one, 2009-08, Vol.4 (8), p.e6376-e6376

2009

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Progress and prospects: immune responses to viral vectors

Gene therapy, 2010-03, Vol.17 (3), p.295-304

2010

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Induction and role of regulatory CD4+CD25+ T cells in tolerance to the transgene product following hepatic in vivo gene transfer

Blood, 2007-08, Vol.110 (4), p.1132-1140

2007

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Effective gene therapy for haemophilic mice with pathogenic factor IX antibodies

EMBO molecular medicine, 2013-11, Vol.5 (11), p.1698-1709

2013

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Copackaged AAV9 Vectors Promote Simultaneous Immune Tolerance and Phenotypic Correction of Pompe Disease

Human gene therapy, 2016-01, Vol.27 (1), p.43-59

2016

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Strategies to Prevent or Reverse Immune Responses Against Factor In Gene Replacement for Hemophilia B

Blood, 2010-11, Vol.116 (21), p.3761-3761

2010

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Humoral and Anaphylactic Responses to Factor IX In Murine Hemophilia B Are Genotype Dependent and Can Be Reversed by Hepatic Gene Transfer

Blood, 2010-11, Vol.116 (21), p.2202-2202

2010

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Improved induction of immune tolerance to factor IX by hepatic AAV-8 gene transfer

Human gene therapy, 2009-07, Vol.20 (7), p.767-776

2009

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BAFF blockade prevents anti-drug antibody formation in a mouse model of Pompe disease

Clinical immunology (Orlando, Fla.), 2015-06, Vol.158 (2), p.140-147

2015

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MAIT cells in the human oral mucosa are activated and perforinlow

The Journal of immunology (1950), 2017-05, Vol.198 (1_Supplement), p.149-149.8

2017

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Pompe disease gene therapy: Gene Therapy

Human molecular genetics, 2011-04, Vol.20 (1), p.R61-R68

2011

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Mapping the T helper cell response to acid α-glucosidase in Pompe mice

Molecular genetics and metabolism, 2012-06, Vol.106 (2), p.189-195

2012

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293. Optimal In Vivo Treg Induction and Suppression of Immune Responses By Synergistic Use of Rapamycin and FLT3 Ligand

Molecular therapy, 2015-05, Vol.23, p.S118-S118

2015

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Pharmacological chaperones prevent the precipitation of rhGAA by anti-GAA antibodies during enzyme replacement therapy

Molecular genetics and metabolism, 2014-02, Vol.111 (2), p.S38-S38

2014

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Prevention and Reversal of Antibody Responses Against Factor IX in Gene Therapy for Hemophilia B

Frontiers in microbiology, 2011-01, Vol.2, p.244-244

2011

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Tolerance Induction to Factor VIII by Transient Co-administration with Rapamycin

Journal of thrombosis and haemostasis, 2011-08, Vol.9 (8), p.1524-1533

2011

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B-cell Depletion and Immunomodulation Prior to Initiation of Enzyme Replacement Therapy Blocks the Immune Response to Acid Alpha Glucosidase in Infantile Onset Pompe Disease

The Journal of pediatrics, 2013-04, Vol.163 (3), p.847-54.e1

2013

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Glucose Oxidation Modulates Anoikis and Tumor Metastasis

Synergy between rapamycin and FLT3 ligand enhances plasmacytoid dendritic cell–dependent induction of CD4+CD25+FoxP3+ Treg

B-Cell Depletion and Immunomodulation before Initiation of Enzyme Replacement Therapy Blocks the Immune Response to Acid Alpha-Glucosidase in Infantile-Onset Pompe Disease

Sustained Correction of Motoneuron Histopathology Following Intramuscular Delivery of AAV in Pompe Mice

GITR engagement preferentially enhances proliferation of functionally competent CD4+CD25+FoxP3+ regulatory T cells

Impact of the Underlying Mutation and the Route of Vector Administration on Immune Responses to Factor IX in Gene Therapy for Hemophilia B

Targeted approaches to induce immune tolerance for Pompe disease therapy

Immune responses and hypercoagulation in ERT for Pompe disease are mutation and rhGAA dose dependent

Tolerance induction to cytoplasmic beta-galactosidase by hepatic AAV gene transfer: implications for antigen presentation and immunotoxicity

Progress and prospects: immune responses to viral vectors

Induction and role of regulatory CD4+CD25+ T cells in tolerance to the transgene product following hepatic in vivo gene transfer

Effective gene therapy for haemophilic mice with pathogenic factor IX antibodies

Copackaged AAV9 Vectors Promote Simultaneous Immune Tolerance and Phenotypic Correction of Pompe Disease

Strategies to Prevent or Reverse Immune Responses Against Factor In Gene Replacement for Hemophilia B

Humoral and Anaphylactic Responses to Factor IX In Murine Hemophilia B Are Genotype Dependent and Can Be Reversed by Hepatic Gene Transfer

Improved induction of immune tolerance to factor IX by hepatic AAV-8 gene transfer

BAFF blockade prevents anti-drug antibody formation in a mouse model of Pompe disease

MAIT cells in the human oral mucosa are activated and perforinlow

Pompe disease gene therapy: Gene Therapy

Mapping the T helper cell response to acid α-glucosidase in Pompe mice

293. Optimal In Vivo Treg Induction and Suppression of Immune Responses By Synergistic Use of Rapamycin and FLT3 Ligand

Pharmacological chaperones prevent the precipitation of rhGAA by anti-GAA antibodies during enzyme replacement therapy

Prevention and Reversal of Antibody Responses Against Factor IX in Gene Therapy for Hemophilia B

Tolerance Induction to Factor VIII by Transient Co-administration with Rapamycin

B-cell Depletion and Immunomodulation Prior to Initiation of Enzyme Replacement Therapy Blocks the Immune Response to Acid Alpha Glucosidase in Infantile Onset Pompe Disease

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