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Neuroinflammation and fractalkine signaling in Alzheimer's disease

Journal of neuroinflammation, 2019-02, Vol.16 (1), p.30-30, Article 30

2019

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T cell infiltration and upregulation of MHCII in microglia leads to accelerated neuronal loss in an α-synuclein rat model of Parkinson's disease

Journal of neuroinflammation, 2020-08, Vol.17 (1), p.242-242, Article 242

2020

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Reelin Signaling in Neurodevelopmental Disorders and Neurodegenerative Diseases

Brain sciences, 2023-10, Vol.13 (10), p.1479

2023

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Lung infection by Pseudomonas aeruginosa induces neuroinflammation and blood-brain barrier dysfunction in mice

Journal of neuroinflammation, 2023-05, Vol.20 (1), p.127-127, Article 127

2023

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Therapeutic Delivery of Soluble Fractalkine Ameliorates Vascular Dysfunction in the Diabetic Retina

International journal of molecular sciences, 2024-02, Vol.25 (3), p.1727

2024

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Fractalkine isoforms differentially regulate microglia-mediated inflammation and enhance visual function in the diabetic retina

Journal of neuroinflammation, 2024-02, Vol.21 (1), p.42-18, Article 42

2024

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Aberrant AZIN2 and polyamine metabolism precipitates tau neuropathology

The Journal of clinical investigation, 2021-02, Vol.131 (4), p.1-22

2021

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CNS-Wide over Expression of Fractalkine Improves Cognitive Functioning in a Tauopathy Model

Journal of neuroimmune pharmacology, 2019-06, Vol.14 (2), p.312-325

2019

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Toward Development of Neuron Specific Transduction After Systemic Delivery of Viral Vectors

Frontiers in neurology, 2021-08, Vol.12, p.685802-685802

2021

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Diverse activation of microglia by chemokine (C-C motif) ligand 2 overexpression in brain

Journal of neuroinflammation, 2013-07, Vol.10 (1), p.86-86, Article 856

2013

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Sustained Arginase 1 Expression Modulates Pathological Tau Deposits in a Mouse Model of Tauopathy

The Journal of neuroscience, 2015-11, Vol.35 (44), p.14842-14860

2015

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Intracranial injection of AAV expressing NEP but not IDE reduces amyloid pathology in APP+PS1 transgenic mice

PloS one, 2013-03, Vol.8 (3), p.e59626-e59626

2013

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CX3CL1/CX3CR1 signaling targets for the treatment of neurodegenerative diseases

Pharmacology & therapeutics (Oxford), 2022-03, Vol.231, p.107989-107989, Article 107989

2022

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Adenylyl cyclase 2 expression and function in neurological diseases

CNS neuroscience & therapeutics, 2024-07, Vol.30 (7), p.e14880-n/a

2024

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Spermidine/spermine-N 1 -acetyltransferase ablation impacts tauopathy-induced polyamine stress response

Alzheimer's research & therapy, 2019-06, Vol.11 (1), p.58-24

2019

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371 Fractalkine isoforms using gene therapy differentially regulate microglia activation and vascular damage in the diabetic retina

Journal of clinical and translational science, 2023-04, Vol.7 (s1), p.110-110

2023

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Recovery of Angelman syndrome rat deficits with UBE3A protein supplementation

Molecular and cellular neuroscience, 2022-05, Vol.120, p.103724-103724, Article 103724

2022

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Reelin central fragment supplementation improves cognitive deficits in a mouse model of Fragile X Syndrome

Experimental neurology, 2022-11, Vol.357, p.114170-114170, Article 114170

2022

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Small-Scale Recombinant Adeno-Associated Virus Purification

Methods in molecular biology (Clifton, N.J.), 2016, Vol.1382, p.95

2016

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Fractalkine Over Expression Suppresses α-Synuclein-mediated Neurodegeneration

Molecular therapy, 2015-01, Vol.23 (1), p.17-23

2015

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A novel Reelin construct, R36, recovered behavioural deficits in the heterozygous reeler mouse

The European journal of neuroscience, 2023-05, Vol.57 (10), p.1657-1670

2023

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The soluble isoform of CX3CL1 is necessary for neuroprotection in a mouse model of Parkinson's disease

The Journal of neuroscience, 2012-10, Vol.32 (42), p.14592-14601

2012

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Concentration and proteolysis of CX3CL1 may regulate the microglial response to CX3CL1

Glia, 2023-02, Vol.71 (2), p.245-258

2023

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Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency

Cellular signalling, 2023-09, Vol.109, p.110763-110763, Article 110763

2023

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Fractalkine overexpression suppresses tau pathology in a mouse model of tauopathy

Neurobiology of aging, 2013-06, Vol.34 (6), p.1540-1548

2013

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Neuroinflammation and fractalkine signaling in Alzheimer's disease

T cell infiltration and upregulation of MHCII in microglia leads to accelerated neuronal loss in an α-synuclein rat model of Parkinson's disease

Reelin Signaling in Neurodevelopmental Disorders and Neurodegenerative Diseases

Lung infection by Pseudomonas aeruginosa induces neuroinflammation and blood-brain barrier dysfunction in mice

Therapeutic Delivery of Soluble Fractalkine Ameliorates Vascular Dysfunction in the Diabetic Retina

Fractalkine isoforms differentially regulate microglia-mediated inflammation and enhance visual function in the diabetic retina

Aberrant AZIN2 and polyamine metabolism precipitates tau neuropathology

CNS-Wide over Expression of Fractalkine Improves Cognitive Functioning in a Tauopathy Model

Toward Development of Neuron Specific Transduction After Systemic Delivery of Viral Vectors

Diverse activation of microglia by chemokine (C-C motif) ligand 2 overexpression in brain

Sustained Arginase 1 Expression Modulates Pathological Tau Deposits in a Mouse Model of Tauopathy

Intracranial injection of AAV expressing NEP but not IDE reduces amyloid pathology in APP+PS1 transgenic mice

CX3CL1/CX3CR1 signaling targets for the treatment of neurodegenerative diseases

Adenylyl cyclase 2 expression and function in neurological diseases

Spermidine/spermine-N 1 -acetyltransferase ablation impacts tauopathy-induced polyamine stress response

371 Fractalkine isoforms using gene therapy differentially regulate microglia activation and vascular damage in the diabetic retina

Recovery of Angelman syndrome rat deficits with UBE3A protein supplementation

Reelin central fragment supplementation improves cognitive deficits in a mouse model of Fragile X Syndrome

Small-Scale Recombinant Adeno-Associated Virus Purification

Fractalkine Over Expression Suppresses α-Synuclein-mediated Neurodegeneration

A novel Reelin construct, R36, recovered behavioural deficits in the heterozygous reeler mouse

The soluble isoform of CX3CL1 is necessary for neuroprotection in a mouse model of Parkinson's disease

Concentration and proteolysis of CX3CL1 may regulate the microglial response to CX3CL1

Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency

Fractalkine overexpression suppresses tau pathology in a mouse model of tauopathy

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