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Phase diagram of strongly-coupled Rashba systems

New journal of physics, 2024-09, Vol.26 (9), p.93015

2024

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Disturbance of the gut microbiota in early-life selectively affects visceral pain in adulthood without impacting cognitive or anxiety-related behaviors in male rats

Neuroscience, 2014-09, Vol.277, p.885-901

2014

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Nanomodulation of Macrophages in Multiple Sclerosis

Cells (Basel, Switzerland), 2019-06, Vol.8 (6), p.543

2019

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Enhancing arginase 2 expression using target site blockers as a strategy to modulate macrophage phenotype

Molecular therapy. Nucleic acids, 2022-09, Vol.29, p.643-655

2022

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Natural killer cells protect mice from DSS-induced colitis by regulating neutrophil function via the NKG2A receptor

Mucosal immunology, 2013-09, Vol.6 (5), p.1016-1026

2013

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Mitochondrial arginase-2 is essential for IL-10 metabolic reprogramming of inflammatory macrophages

Nature communications, 2021-03, Vol.12 (1), p.1460-1460, Article 1460

2021

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Bcl‐3 deficiency protects against dextran‐sodium sulphate‐induced colitis in the mouse

Clinical and experimental immunology, 2013-08, Vol.173 (2), p.332-342

2013

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TL1A/TNFSF15 directly induces proinflammatory cytokines, including TNFα, from CD3+CD161+ T cells to exacerbate gut inflammation

Mucosal immunology, 2013-09, Vol.6 (5), p.886-899

2013

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Use of bioluminescence imaging to track neutrophil migration and its inhibition in experimental colitis

Clinical and experimental immunology, 2010-10, Vol.162 (1), p.188-196

2010

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Down-regulation of p38 mitogen-activated protein kinase activation and proinflammatory cytokine production by mitogen-activated protein kinase inhibitors in inflammatory bowel disease

Clinical and experimental immunology, 2010-10, Vol.162 (1), p.108-115

2010

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A theoretical investigation of 38-atom CuPd clusters: the effect of potential parameterisation on structure and segregation

Physical chemistry chemical physics : PCCP, 2021-08, Vol.23 (3), p.1595-15964

2021

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P040 Caspase-8 blockade attenuates NSAID-induced colitis and cell death via NLRP3 inflammasome and NF-κB activation

Journal of Crohn's and colitis, 2023-01, Vol.17 (Supplement_1), p.i207-i207

2023

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Attenuation of Oxytocin and Serotonin 2A Receptor Signaling through Novel Heteroreceptor Formation

ACS chemical neuroscience, 2019-07, Vol.10 (7), p.3225-3240

2019

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OC1.04.3 ACTIVATION AND MODULATION OF THE MITOGEN-ACTIVATED PROTEIN KINASE PATHWAY IN INFLAMMATORY BOWEL DISEASE

Digestive and liver disease, 2008, Vol.40, p.S16-S16

2008

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Turning on T-cell death and turning off Crohn's disease

Gastroenterology (New York, N.Y. 1943), 2000-10, Vol.119 (4), p.1166-1168

2000

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Altered Mechanisms of Apoptosis in Colon Cancer: Fas Resistance and Counterattack in the Tumor-Immune Conflict

Annals of the New York Academy of Sciences, 2000-06, Vol.910 (1), p.178-195

2000

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Targeting the Fas/Fas ligand pathway in cancer

Expert opinion on therapeutic targets, 2005-10, Vol.9 (5), p.1031-1044

2005

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TL1A/TNFSF15 directly induces proinflammatory cytokines, including TNF[alpha], from CD3+CD161+ T cells to exacerbate gut inflammation

Mucosal immunology, 2013-09, Vol.6 (5), p.886

2013

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Interferon-γ sensitizes colonic epithelial cell lines to physiological and therapeutic inducers of colonocyte apoptosis

Journal of cellular physiology, 2000-12, Vol.185 (3), p.331-338

2000

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N-acetylcysteine (NAC) downregulates type I insulin-like growth factor receptor (IGF-IR) and epidermal growth factor receptor (EGFR) on colorectal carcinoma cell lines – possible mechanism underlying NAC-mediated chemoprevention and chemotherapy

Endoscopy, 2006, Vol.37 (12)

2006

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Type I Insulin-like Growth Factor Receptor Expression on Colorectal Adenocarcinoma Cell Lines Is Decreased in Response to the Chemopreventive Agent N-Acetyl-l-Cysteine

Annals of the New York Academy of Sciences, 2002-11, Vol.973 (1), p.555-558

2002

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Molecular mechanisms of cytokine induced apoptosis in colorectal cancer cells

Nally, K

2002

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IGF-1 protects colonic epithelial cells from cytokine induced apoptosis by maintaining cellular phospho-tyrosine content and preventing cytochrome C release

Gastroenterology (New York, N.Y. 1943), 2001-04, Vol.120 (5), p.A316-A316

2001

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The chemopreventative agent N-acetyl-L-cysteine (NAC) decreases epidermal growth factor receptor (EGFR) levels on colonic carcinoma cell lines

Gastroenterology (New York, N.Y. 1943), 2001-04, Vol.120 (5), p.A615-A615

2001

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Pro-inflammatory cytokines suppress p53 function in colonic epithelial cells — A link between inflammation and tumorigenesis

Gastroenterology (New York, N.Y. 1943), 2001-04, Vol.120 (5), p.A190-A190

2001

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Phase diagram of strongly-coupled Rashba systems

Disturbance of the gut microbiota in early-life selectively affects visceral pain in adulthood without impacting cognitive or anxiety-related behaviors in male rats

Nanomodulation of Macrophages in Multiple Sclerosis

Enhancing arginase 2 expression using target site blockers as a strategy to modulate macrophage phenotype

Natural killer cells protect mice from DSS-induced colitis by regulating neutrophil function via the NKG2A receptor

Mitochondrial arginase-2 is essential for IL-10 metabolic reprogramming of inflammatory macrophages

Bcl‐3 deficiency protects against dextran‐sodium sulphate‐induced colitis in the mouse

TL1A/TNFSF15 directly induces proinflammatory cytokines, including TNFα, from CD3+CD161+ T cells to exacerbate gut inflammation

Use of bioluminescence imaging to track neutrophil migration and its inhibition in experimental colitis

Down-regulation of p38 mitogen-activated protein kinase activation and proinflammatory cytokine production by mitogen-activated protein kinase inhibitors in inflammatory bowel disease

A theoretical investigation of 38-atom CuPd clusters: the effect of potential parameterisation on structure and segregation

P040 Caspase-8 blockade attenuates NSAID-induced colitis and cell death via NLRP3 inflammasome and NF-κB activation

Attenuation of Oxytocin and Serotonin 2A Receptor Signaling through Novel Heteroreceptor Formation

OC1.04.3 ACTIVATION AND MODULATION OF THE MITOGEN-ACTIVATED PROTEIN KINASE PATHWAY IN INFLAMMATORY BOWEL DISEASE

Turning on T-cell death and turning off Crohn's disease

Altered Mechanisms of Apoptosis in Colon Cancer: Fas Resistance and Counterattack in the Tumor-Immune Conflict

Targeting the Fas/Fas ligand pathway in cancer

TL1A/TNFSF15 directly induces proinflammatory cytokines, including TNF[alpha], from CD3+CD161+ T cells to exacerbate gut inflammation

Interferon-γ sensitizes colonic epithelial cell lines to physiological and therapeutic inducers of colonocyte apoptosis

N-acetylcysteine (NAC) downregulates type I insulin-like growth factor receptor (IGF-IR) and epidermal growth factor receptor (EGFR) on colorectal carcinoma cell lines – possible mechanism underlying NAC-mediated chemoprevention and chemotherapy

Type I Insulin-like Growth Factor Receptor Expression on Colorectal Adenocarcinoma Cell Lines Is Decreased in Response to the Chemopreventive Agent N-Acetyl-l-Cysteine

Molecular mechanisms of cytokine induced apoptosis in colorectal cancer cells

IGF-1 protects colonic epithelial cells from cytokine induced apoptosis by maintaining cellular phospho-tyrosine content and preventing cytochrome C release

The chemopreventative agent N-acetyl-L-cysteine (NAC) decreases epidermal growth factor receptor (EGFR) levels on colonic carcinoma cell lines

Pro-inflammatory cytokines suppress p53 function in colonic epithelial cells — A link between inflammation and tumorigenesis

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