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GRIM-19: A master regulator of cytokine induced tumor suppression, metastasis and energy metabolism

Cytokine & growth factor reviews, 2017-02, Vol.33, p.1-18

2017

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Regulation of the Death-Associated Protein Kinase 1 Expression and Autophagy via ATF6 Requires Apoptosis Signal-Regulating Kinase 1

Molecular and cellular biology, 2014-11, Vol.34 (21), p.4033-4048

2014

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice

Proceedings of the National Academy of Sciences - PNAS, 2013-11, Vol.110 (45), p.18035-18035

2013

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Inhibition of Mcl-1 Promotes Senescence in Cancer Cells: Implications for Preventing Tumor Growth and Chemotherapy Resistance

Molecular and Cellular Biology, 2012-05, Vol.32 (10), p.1879-1892

2012

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Critical Role for Transcription Factor C/EBP-β in Regulating the Expression of Death-Associated Protein Kinase 1

Molecular and Cellular Biology, 2008-04, Vol.28 (8), p.2528-2548

2008

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p16INK4A enhances the transcriptional and the apoptotic functions of p53 through DNA‐dependent interaction

Molecular carcinogenesis, 2017-07, Vol.56 (7), p.1687-1702

2017

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Publisher's Note: Identification and characterization of GRIM-1, a cell-death-associated gene product

Journal of cell science, 2024-05, Vol.137 (10)

2024

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Bacteria and genetically modified bacteria as cancer therapeutics: Current advances and challenges

Cytokine (Philadelphia, Pa.), 2017-01, Vol.89, p.160-172

2017

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GRIM-19 disrupts E6/E6AP complex to rescue p53 and induce apoptosis in cervical cancers

PloS one, 2011-07, Vol.6 (7), p.e22065-e22065

2011

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GRIM-1, a novel growth suppressor, inhibits rRNA maturation by suppressing small nucleolar RNAs

PloS one, 2011-09, Vol.6 (9), p.e24082-e24082

2011

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TLR5-Derived, TIR-Interacting Decoy Peptides to Inhibit TLR Signaling

The Journal of immunology (1950), 2023-05, Vol.210 (9), p.1419-1427

2023

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Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product

Journal of cell science, 2021-11, Vol.134 (22)

2021

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Interferons, signal transduction pathways, and the central nervous system

Journal of interferon & cytokine research, 2014-08, Vol.34 (8), p.559-576

2014

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The Cyclin-dependent Kinase Inhibitor p16INK4a Physically Interacts with Transcription Factor Sp1 and Cyclin-dependent Kinase 4 to Transactivate MicroRNA-141 and MicroRNA-146b-5p Spontaneously and in Response to Ultraviolet Light-induced DNA Damage

The Journal of biological chemistry, 2013-12, Vol.288 (49), p.35511-35525

2013

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Oncosuppressor protein p53 and cyclin-dependent kinase inhibitor p21 regulate interstitial cystitis associated gene expression

Cytokine (Philadelphia, Pa.), 2018-10, Vol.110, p.110-115

2018

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GRIM-19 and p16INK4a Synergistically Regulate Cell Cycle Progression and E2F1-responsive Gene Expression

The Journal of biological chemistry, 2010-09, Vol.285 (36), p.27545-27552

2010

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Regulation of snoRNAs in cancer: close encounters with interferon

Journal of interferon & cytokine research, 2013-04, Vol.33 (4), p.189-198

2013

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Tumor-derived Mutations in the Gene Associated with Retinoid Interferon-induced Mortality (GRIM-19) Disrupt Its Anti-signal Transducer and Activator of Transcription 3 (STAT3) Activity and Promote Oncogenesis

The Journal of biological chemistry, 2013-03, Vol.288 (11), p.7930-7941

2013

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p16 INK4A enhances the transcriptional and the apoptotic functions of p53 through DNA-dependent interaction

Molecular carcinogenesis, 2017-07, Vol.56 (7), p.1687-1702

2017

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Cytokine-induced tumor suppressors: A GRIM story

Cytokine (Philadelphia, Pa.), 2010-10, Vol.52 (1-2), p.128-142

2010

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GRIM-19 and p16(INK4a) synergistically regulate cell cycle progression and E2F1-responsive gene expression

The Journal of biological chemistry, 2010-09, Vol.285 (36), p.27545-27552

2010

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The Med1 Subunit of Transcriptional Mediator Plays a Central Role in Regulating CCAAT/Enhancer-binding Protein-β-driven Transcription in Response to Interferon-γ

The Journal of biological chemistry, 2008-05, Vol.283 (19), p.13077-13086

2008

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Identification of a Structural Motif in the Tumor-Suppressive Protein GRIM-19 Required for Its Antitumor Activity

The American journal of pathology, 2010-08, Vol.177 (2), p.896-907

2010

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Tumor-suppressive activity of the cell death activator GRIM-19 on a constitutively active signal transducer and activator of transcription 3

Cancer research (Chicago, Ill.), 2007-07, Vol.67 (13), p.6212-6220

2007

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Identification and characterization of GRIM-1, a cell-death-associated gene product

Journal of cell science, 2010-08, Vol.123 (16), p.2781-2791

2010

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GRIM-19: A master regulator of cytokine induced tumor suppression, metastasis and energy metabolism

Regulation of the Death-Associated Protein Kinase 1 Expression and Autophagy via ATF6 Requires Apoptosis Signal-Regulating Kinase 1

Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice

Inhibition of Mcl-1 Promotes Senescence in Cancer Cells: Implications for Preventing Tumor Growth and Chemotherapy Resistance

Critical Role for Transcription Factor C/EBP-β in Regulating the Expression of Death-Associated Protein Kinase 1

p16INK4A enhances the transcriptional and the apoptotic functions of p53 through DNA‐dependent interaction

Publisher's Note: Identification and characterization of GRIM-1, a cell-death-associated gene product

Bacteria and genetically modified bacteria as cancer therapeutics: Current advances and challenges

GRIM-19 disrupts E6/E6AP complex to rescue p53 and induce apoptosis in cervical cancers

GRIM-1, a novel growth suppressor, inhibits rRNA maturation by suppressing small nucleolar RNAs

TLR5-Derived, TIR-Interacting Decoy Peptides to Inhibit TLR Signaling

Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product

Interferons, signal transduction pathways, and the central nervous system

The Cyclin-dependent Kinase Inhibitor p16INK4a Physically Interacts with Transcription Factor Sp1 and Cyclin-dependent Kinase 4 to Transactivate MicroRNA-141 and MicroRNA-146b-5p Spontaneously and in Response to Ultraviolet Light-induced DNA Damage

Oncosuppressor protein p53 and cyclin-dependent kinase inhibitor p21 regulate interstitial cystitis associated gene expression

GRIM-19 and p16INK4a Synergistically Regulate Cell Cycle Progression and E2F1-responsive Gene Expression

Regulation of snoRNAs in cancer: close encounters with interferon

Tumor-derived Mutations in the Gene Associated with Retinoid Interferon-induced Mortality (GRIM-19) Disrupt Its Anti-signal Transducer and Activator of Transcription 3 (STAT3) Activity and Promote Oncogenesis

p16 INK4A enhances the transcriptional and the apoptotic functions of p53 through DNA-dependent interaction

Cytokine-induced tumor suppressors: A GRIM story

GRIM-19 and p16(INK4a) synergistically regulate cell cycle progression and E2F1-responsive gene expression

The Med1 Subunit of Transcriptional Mediator Plays a Central Role in Regulating CCAAT/Enhancer-binding Protein-β-driven Transcription in Response to Interferon-γ

Identification of a Structural Motif in the Tumor-Suppressive Protein GRIM-19 Required for Its Antitumor Activity

Tumor-suppressive activity of the cell death activator GRIM-19 on a constitutively active signal transducer and activator of transcription 3

Identification and characterization of GRIM-1, a cell-death-associated gene product

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