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Motifs of VDAC2 required for mitochondrial Bak import and tBid-induced apoptosis

Proceedings of the National Academy of Sciences - PNAS, 2015-10, Vol.112 (41), p.E5590-E5599

2015

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Bid-induced mitochondrial membrane permeabilization waves propagated by local reactive oxygen species (ROS) signaling

Proceedings of the National Academy of Sciences - PNAS, 2012-03, Vol.109 (12), p.4497-4502

2012

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MSTO1 is a cytoplasmic pro‐mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans

EMBO molecular medicine, 2023-08, Vol.15 (8), p.e17911-n/a

2023

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Reliance of ER–mitochondrial calcium signaling on mitochondrial EF-hand Ca2+ binding proteins: Miros, MICUs, LETM1 and solute carriers

Current opinion in cell biology, 2014-08, Vol.29, p.133-141

2014

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Cytosolic Ca2+ prevents the subplasmalemmal clustering of STIM1: an intrinsic mechanism to avoid Ca2+ overload

Journal of cell science, 2008-10, Vol.121 (Pt 19), p.3133-3139

2008

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VDAC2-specific cellular functions and the underlying structure

Biochimica et biophysica acta, 2016-10, Vol.1863 (10), p.2503-2514

2016

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The contribution of UCP2 and UCP3 to mitochondrial Ca2+ uptake is differentially determined by the source of supplied Ca2

Cell calcium (Edinburgh), 2010-05, Vol.47 (5), p.433-440

2010

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MSTO1 is a cytoplasmic pro-mitochondrial fusion protein

EMBO molecular medicine, 2017-07, Vol.9 (7), p.967-984

2017

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Uncoupling protein 3 adjusts mitochondrial Ca2+ uptake to high and low Ca2+ signals

Cell calcium (Edinburgh), 2010-11, Vol.48 (5), p.288-301

2010

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Mitochondrial fusion and Bid-mediated mitochondrial apoptosis are perturbed by alcohol with distinct dependence on its metabolism

Cell death & disease, 2018-10, Vol.9 (10), p.1028-16, Article 1028

2018

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Abstract 3492: Differential sensitivity of normal liver and hepatocarcinoma tissues to tBid induced apoptosis is determined by the abundance of VDAC2

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.3492-3492

2016

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MSTO1 is a cytoplasmic pro‐mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans

EMBO molecular medicine, 2017-07, Vol.9 (7), p.967-984

2017

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Mitochondrial Ca²⁺ uptake and not mitochondrial motility is required for STIM1-Orai1-dependent store-operated Ca²⁺ entry

Journal of cell science, 2010-08, Vol.123 (15), p.2553-2564

2010

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GPR55‐dependent and ‐independent ion signalling in response to lysophosphatidylinositol in endothelial cells

British journal of pharmacology, 2010-09, Vol.161 (2), p.308-320

2010

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The contribution of UCP2 and UCP3 to mitochondrial Ca(2+) uptake is differentially determined by the source of supplied Ca(2+)

Cell calcium (Edinburgh), 2010-05, Vol.47 (5), p.433-440

2010

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The Isoform Specific N Terminus of VDAC2 is Dispensable for tBid Induced Cytochrome C Release

Biophysical journal, 2012-01, Vol.102 (3), p.437a-437a

2012

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MSTO 1 is a cytoplasmic pro‐mitochondrial fusion protein

EMBO molecular medicine, 2017-07, Vol.9 (7), p.967-984

2017

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Uncoupling protein 3 adjusts mitochondrial Ca(2+) uptake to high and low Ca(2+) signals

Cell calcium (Edinburgh), 2010-11, Vol.48 (5), p.288

2010

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Which Domain of VDAC2 is Necessary for Bak Insertion to the Outer Mitochondrial Membrane and tBid - Induced Cytochrome C Release?

Biophysical journal, 2013-01, Vol.104 (2), p.656a-656a

2013

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The contribution of UCP2 and UCP3 to mitochondrial Ca 2+ uptake is differentially determined by the source of supplied Ca 2

Cell calcium (Edinburgh), 2010, Vol.47 (5), p.433-440

2010

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Alkaline phosphatase activity of Flexibacter chinensis under starvation stress in water microcosms

Enzyme and microbial technology, 2006-12, Vol.40 (1), p.13-16

2006

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The effect of starvation stress on the protein profiles in Flexibacter chinensis

Iranian biomedical journal, 2008-04, Vol.12 (2), p.67-75

2008

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De Novo Protein Production under Long Term Starvation in Flexibacter chinensis

Modern Multidisciplinary Applied Microbiology, 2006, p.50-54

2006

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Motifs of VDAC2 required for mitochondrial Bak import and tBid-induced apoptosis

Bid-induced mitochondrial membrane permeabilization waves propagated by local reactive oxygen species (ROS) signaling

MSTO1 is a cytoplasmic pro‐mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans

Reliance of ER–mitochondrial calcium signaling on mitochondrial EF-hand Ca2+ binding proteins: Miros, MICUs, LETM1 and solute carriers

Cytosolic Ca2+ prevents the subplasmalemmal clustering of STIM1: an intrinsic mechanism to avoid Ca2+ overload

VDAC2-specific cellular functions and the underlying structure

The contribution of UCP2 and UCP3 to mitochondrial Ca2+ uptake is differentially determined by the source of supplied Ca2

MSTO1 is a cytoplasmic pro-mitochondrial fusion protein

Uncoupling protein 3 adjusts mitochondrial Ca2+ uptake to high and low Ca2+ signals

Mitochondrial fusion and Bid-mediated mitochondrial apoptosis are perturbed by alcohol with distinct dependence on its metabolism

Abstract 3492: Differential sensitivity of normal liver and hepatocarcinoma tissues to tBid induced apoptosis is determined by the abundance of VDAC2

MSTO1 is a cytoplasmic pro‐mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans

Mitochondrial Ca²⁺ uptake and not mitochondrial motility is required for STIM1-Orai1-dependent store-operated Ca²⁺ entry

GPR55‐dependent and ‐independent ion signalling in response to lysophosphatidylinositol in endothelial cells

Mitochondrial Ca(2+) uptake by the voltage-dependent anion channel 2 regulates cardiac rhythmicity

The contribution of UCP2 and UCP3 to mitochondrial Ca(2+) uptake is differentially determined by the source of supplied Ca(2+)

The Isoform Specific N Terminus of VDAC2 is Dispensable for tBid Induced Cytochrome C Release

MSTO 1 is a cytoplasmic pro‐mitochondrial fusion protein

Uncoupling protein 3 adjusts mitochondrial Ca(2+) uptake to high and low Ca(2+) signals

Which Domain of VDAC2 is Necessary for Bak Insertion to the Outer Mitochondrial Membrane and tBid - Induced Cytochrome C Release?

The contribution of UCP2 and UCP3 to mitochondrial Ca 2+ uptake is differentially determined by the source of supplied Ca 2

Alkaline phosphatase activity of Flexibacter chinensis under starvation stress in water microcosms

The effect of starvation stress on the protein profiles in Flexibacter chinensis

De Novo Protein Production under Long Term Starvation in Flexibacter chinensis

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