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CC chemokine receptor 2 deficiency aggravates cognitive impairments and amyloid pathology in a transgenic mouse model of Alzheimer's disease

The Journal of neuroscience, 2011-04, Vol.31 (16), p.6208-6220

2011

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A deficiency in CCR2+ monocytes: the hidden side of Alzheimer's disease

Journal of molecular cell biology, 2013-10, Vol.5 (5), p.284

2013

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Hematopoietic CC-chemokine receptor 2 (CCR2) competent cells are protective for the cognitive impairments and amyloid pathology in a transgenic mouse model of Alzheimer's disease

Molecular medicine (Cambridge, Mass.), 2012-02, Vol.18 (2), p.297-313

2012

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Brain-derived neurotrophic factor and hypothalamic-pituitary-adrenal axis adaptation processes in a depressive-like state induced by chronic restraint stress

Molecular and cellular neuroscience, 2011-01, Vol.46 (1), p.55-66

2011

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Use of the guinea pig in studies on the development and prevention of acquired sensorineural hearing loss, with an emphasis on noise

The Journal of the Acoustical Society of America, 2019-11, Vol.146 (5), p.3743-3769

2019

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Ototoxicity: a high risk to auditory function that needs to be monitored in drug development

Frontiers in molecular neuroscience, 2024-05, Vol.17, p.1379743-1379743

2024

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Knockdown of the CXCL12/CXCR7 chemokine pathway results in learning deficits and neural progenitor maturation impairment in mice

Brain, behavior, and immunity, 2019-08, Vol.80, p.697-710

2019

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N-acetylcysteine Treatment Reduces Age-related Hearing Loss and Memory Impairment in the Senescence-Accelerated Prone 8 (SAMP8) Mouse Model

Aging and disease, 2018-08, Vol.9 (4), p.664-673

2018

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Neuroactive steroids modulate HPA axis activity and cerebral brain-derived neurotrophic factor (BDNF) protein levels in adult male rats

Psychoneuroendocrinology, 2007-09, Vol.32 (8), p.1062-1078

2007

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Age-related changes in synaptic markers and monocyte subsets link the cognitive decline of APP(Swe)/PS1 mice

Frontiers in cellular neuroscience, 2012, Vol.6, p.51-51

2012

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Age-related changes in synaptic markers and monocyte subsets link the cognitive decline of APPSwe/PS1 mice

Frontiers in cellular neuroscience, 2012-11, Vol.6

2012

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Blockade of Tau Hyperphosphorylation and Aβ1―42 Generation by the Aminotetrahydrofuran Derivative ANAVEX2-73, a Mixed Muscarinic and σ1 Receptor Agonist, in a Nontransgenic Mouse Model of Alzheimer's Disease

Neuropsychopharmacology, 2013-08, Vol.38 (9), p.1706-1723

2013

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Time-Course and Regional Analyses of the Physiopathological Changes Induced after Cerebral Injection of an Amyloid β Fragment in Rats

The American journal of pathology, 2011-07, Vol.179 (1), p.315-334

2011

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Altered Memory Capacities and Response to Stress in p300 CBP-Associated Factor (PCAF) Histone Acetylase Knockout Mice

Neuropsychopharmacology, 2008-06, Vol.33 (7), p.1584-1602

2008

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In vivo and ex vivo analyses of amyloid toxicity in the Tc1 mouse model of Down syndrome

Journal of psychopharmacology (Oxford), 2018-02, Vol.32 (2), p.174-190

2018

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A single brain-derived neurotrophic factor injection modifies hypothalamo–pituitary–adrenocortical axis activity in adult male rats

Molecular and cellular neuroscience, 2004-11, Vol.27 (3), p.280-295

2004

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Involvement of brain-derived neurotrophic factor in the regulation of hypothalamic somatostatin in vivo

Journal of endocrinology, 2006-03, Vol.188 (3), p.425-433

2006

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Blockade of Tau Hyperphosphorylation and A beta sub(1-42) Generation by the Aminotetrahydrofuran Derivative ANAVEX2-73, a Mixed Muscarinic and sigma sub(1) Receptor Agonist, in a Nontransgenic Mouse Model of Alzheimer's Disease

Neuropsychopharmacology (New York, N.Y.), 2013-08, Vol.38 (9), p.1706-1723

2013

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Blockade of Tau Hyperphosphorylation and A[beta]1-42 Generation by the Aminotetrahydrofuran Derivative ANAVEX2-73, a Mixed Muscarinic and [sigma]1 Receptor Agonist, in a Nontransgenic Mouse Model of Alzheimer's Disease

Neuropsychopharmacology (New York, N.Y.), 2013-08, Vol.38 (9), p.1706

2013

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N-acetylcysteine Treatment Reduces Age-related Hearing Loss and Memory Impairment in the Senescence-Accelerated Prone 8

Aging and disease, 2018-08, Vol.9 (4), p.664

2018

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P4‐200: INTRANASAL FORMULATION OF ERYTHROPOIETIN (EPO) SHOWED POTENT PROTECTIVE ACTIVITY AGAINST AMYLOID TOXICITY IN NON‐TRANSGENIC AND TRANSGENIC MOUSE MODELS OF ALZHEIMER'S DISEASE

Alzheimer's & dementia, 2014-07, Vol.10 (4S_Part_23), p.P861-P861

2014

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INTRANASAL FORMULATION OF ERYTHROPOIETIN (EPO) SHOWED POTENT PROTECTIVE ACTIVITY AGAINST AMYLOID TOXICITY IN NON-TRANSGENIC AND TRANSGENIC MOUSE MODELS OF ALZHEIMER'S DISEASE

Alzheimer's & dementia, 2014-07, Vol.10 (4), p.P861-P861

2014

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Leucettine L41, a DYRK1A-preferential DYRKs/CLKs inhibitor, prevents memory impairments and neurotoxicity induced by oligomeric Aβ25–35 peptide administration in mice

European neuropsychopharmacology, 2015-11, Vol.25 (11), p.2170-2182

2015

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Intranasal formulation of erythropoietin (EPO) showed potent protective activity against amyloid toxicity in the Aβ25-35 non-transgenic mouse model of Alzheimer’s disease

Journal of psychopharmacology (Oxford), 2013-11, Vol.27 (11), p.1044-1057

2013

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Toll-Like Receptor 2-Independent and MyD88-Dependent Gene Expression in the Mouse Brain

Journal of innate immunity, 2009, Vol.1 (5), p.480-493

2009

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CC chemokine receptor 2 deficiency aggravates cognitive impairments and amyloid pathology in a transgenic mouse model of Alzheimer's disease

A deficiency in CCR2+ monocytes: the hidden side of Alzheimer's disease

Hematopoietic CC-chemokine receptor 2 (CCR2) competent cells are protective for the cognitive impairments and amyloid pathology in a transgenic mouse model of Alzheimer's disease

Brain-derived neurotrophic factor and hypothalamic-pituitary-adrenal axis adaptation processes in a depressive-like state induced by chronic restraint stress

Use of the guinea pig in studies on the development and prevention of acquired sensorineural hearing loss, with an emphasis on noise

Ototoxicity: a high risk to auditory function that needs to be monitored in drug development

Knockdown of the CXCL12/CXCR7 chemokine pathway results in learning deficits and neural progenitor maturation impairment in mice

N-acetylcysteine Treatment Reduces Age-related Hearing Loss and Memory Impairment in the Senescence-Accelerated Prone 8 (SAMP8) Mouse Model

Neuroactive steroids modulate HPA axis activity and cerebral brain-derived neurotrophic factor (BDNF) protein levels in adult male rats

Age-related changes in synaptic markers and monocyte subsets link the cognitive decline of APP(Swe)/PS1 mice

Age-related changes in synaptic markers and monocyte subsets link the cognitive decline of APPSwe/PS1 mice

Blockade of Tau Hyperphosphorylation and Aβ1―42 Generation by the Aminotetrahydrofuran Derivative ANAVEX2-73, a Mixed Muscarinic and σ1 Receptor Agonist, in a Nontransgenic Mouse Model of Alzheimer's Disease

Time-Course and Regional Analyses of the Physiopathological Changes Induced after Cerebral Injection of an Amyloid β Fragment in Rats

Altered Memory Capacities and Response to Stress in p300 CBP-Associated Factor (PCAF) Histone Acetylase Knockout Mice

In vivo and ex vivo analyses of amyloid toxicity in the Tc1 mouse model of Down syndrome

A single brain-derived neurotrophic factor injection modifies hypothalamo–pituitary–adrenocortical axis activity in adult male rats

Involvement of brain-derived neurotrophic factor in the regulation of hypothalamic somatostatin in vivo

Blockade of Tau Hyperphosphorylation and A beta sub(1-42) Generation by the Aminotetrahydrofuran Derivative ANAVEX2-73, a Mixed Muscarinic and sigma sub(1) Receptor Agonist, in a Nontransgenic Mouse Model of Alzheimer's Disease

Blockade of Tau Hyperphosphorylation and A[beta]1-42 Generation by the Aminotetrahydrofuran Derivative ANAVEX2-73, a Mixed Muscarinic and [sigma]1 Receptor Agonist, in a Nontransgenic Mouse Model of Alzheimer's Disease

N-acetylcysteine Treatment Reduces Age-related Hearing Loss and Memory Impairment in the Senescence-Accelerated Prone 8

P4‐200: INTRANASAL FORMULATION OF ERYTHROPOIETIN (EPO) SHOWED POTENT PROTECTIVE ACTIVITY AGAINST AMYLOID TOXICITY IN NON‐TRANSGENIC AND TRANSGENIC MOUSE MODELS OF ALZHEIMER'S DISEASE

INTRANASAL FORMULATION OF ERYTHROPOIETIN (EPO) SHOWED POTENT PROTECTIVE ACTIVITY AGAINST AMYLOID TOXICITY IN NON-TRANSGENIC AND TRANSGENIC MOUSE MODELS OF ALZHEIMER'S DISEASE

Leucettine L41, a DYRK1A-preferential DYRKs/CLKs inhibitor, prevents memory impairments and neurotoxicity induced by oligomeric Aβ25–35 peptide administration in mice

Intranasal formulation of erythropoietin (EPO) showed potent protective activity against amyloid toxicity in the Aβ25-35 non-transgenic mouse model of Alzheimer’s disease

Toll-Like Receptor 2-Independent and MyD88-Dependent Gene Expression in the Mouse Brain

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