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Neuroinflammation and Central Sensitization in Chronic and Widespread Pain

Anesthesiology (Philadelphia), 2018-08, Vol.129 (2), p.343-366

2018

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Acute inflammatory response via neutrophil activation protects against the development of chronic pain

Science translational medicine, 2022-05, Vol.14 (644), p.eabj9954

2022

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β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines

Pain (Amsterdam), 2014-07, Vol.155 (7), p.1346-1355

2014

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Vagus nerve stimulation rescues persistent pain following orthopedic surgery in adult mice

Pain (Amsterdam), 2024-08, Vol.165 (8), p.e80-e92

2024

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miRNA-711 Binds and Activates TRPA1 Extracellularly to Evoke Acute and Chronic Pruritus

Neuron (Cambridge, Mass.), 2018-08, Vol.99 (3), p.449-463.e6

2018

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Genetic basis for individual variations in pain perception and the development of a chronic pain condition

Human molecular genetics, 2005-01, Vol.14 (1), p.135-143

2005

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Genetic architecture of human pain perception

Trends in genetics, 2007-12, Vol.23 (12), p.605-613

2007

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Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation

Brain, behavior, and immunity, 2015-11, Vol.50, p.196-202

2015

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Alternative Splicing of G Protein–Coupled Receptors: Relevance to Pain Management

Mayo Clinic proceedings, 2015-08, Vol.90 (8), p.1135-1151

2015

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Transforming Growth Factor-β-Activated Kinase 1 (TAK1) Mediates Chronic Pain and Cytokine Production in Mouse Models of Inflammatory, Neuropathic, and Primary Pain

The journal of pain, 2023-09, Vol.24 (9), p.1633-1644

2023

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Incidence and Risk Factors for Pelvic Pain After Mesh Implant Surgery for the Treatment of Pelvic Floor Disorders

Journal of minimally invasive gynecology, 2017-01, Vol.24 (1), p.67-73

2017

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Epistasis between polymorphisms in COMT, ESR1, and GCH1 influences COMT enzyme activity and pain

Pain (Amsterdam), 2014-11, Vol.155 (11), p.2390-2399

2014

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Sensory Neuron-TRPV4 Modulates Temporomandibular Disorder Pain Via CGRP in Mice

The journal of pain, 2023-05, Vol.24 (5), p.782-795

2023

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Catechol- O-methyltransferase gene polymorphisms are associated with multiple pain-evoking stimuli

Pain (Amsterdam), 2006-12, Vol.125 (3), p.216-224

2006

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Mu Opioid Splice Variant MOR-1K Contributes to the Development of Opioid-Induced Hyperalgesia

PloS one, 2015-08, Vol.10 (8), p.e0135711

2015

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Rationale and design of a multicenter randomized clinical trial of vestibulodynia: understanding pathophysiology and determining appropriate treatments (vestibulodynia: UPDATe)

Annals of medicine (Helsinki), 2022-12, Vol.54 (1), p.2884-2896

2022

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A mouse model of chronic primary pain that integrates clinically relevant genetic vulnerability, stress, and minor injury

Science translational medicine, 2024-04, Vol.16 (742), p.eadj0395

2024

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Sustained stimulation of β 2 - and β 3 -adrenergic receptors leads to persistent functional pain and neuroinflammation

Brain, behavior, and immunity, 2018-10, Vol.73, p.520

2018

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Idiopathic pain disorders – Pathways of vulnerability

Pain (Amsterdam), 2006-08, Vol.123 (3), p.226-230

2006

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Expression of ectopic heat shock protein 90 in male and female primary afferent nociceptors regulates inflammatory pain

Pain (Amsterdam), 2022-06, Vol.163 (6), p.1091-1101

2022

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IL-23 Enhances C-Fiber-Mediated and Blue Light-Induced Spontaneous Pain in Female Mice

Frontiers in immunology, 2021-12, Vol.12, p.787565

2021

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Role of Blood-Brain Barrier Dysfunction in Delirium following Non-cardiac Surgery in Older Adults

Annals of neurology, 2023-12, Vol.94 (6), p.1024-1035

2023

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Epiregulin and EGFR interactions are involved in pain processing

The Journal of clinical investigation, 2017-09, Vol.127 (9), p.3353-3366

2017

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Low catechol-O-methyltransferase and stress potentiate functional pain and depressive behavior, especially in female mice

Pain (Amsterdam), 2020-02, Vol.161 (2), p.446-458

2020

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Persistent Catechol-O-methyltransferase–dependent Pain Is Initiated by Peripheral β-Adrenergic Receptors

Anesthesiology (Philadelphia), 2016-05, Vol.124 (5), p.1122-1135

2016

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Neuroinflammation and Central Sensitization in Chronic and Widespread Pain

Acute inflammatory response via neutrophil activation protects against the development of chronic pain

β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines

Vagus nerve stimulation rescues persistent pain following orthopedic surgery in adult mice

miRNA-711 Binds and Activates TRPA1 Extracellularly to Evoke Acute and Chronic Pruritus

Genetic basis for individual variations in pain perception and the development of a chronic pain condition

Genetic architecture of human pain perception

Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation

Alternative Splicing of G Protein–Coupled Receptors: Relevance to Pain Management

Transforming Growth Factor-β-Activated Kinase 1 (TAK1) Mediates Chronic Pain and Cytokine Production in Mouse Models of Inflammatory, Neuropathic, and Primary Pain

Incidence and Risk Factors for Pelvic Pain After Mesh Implant Surgery for the Treatment of Pelvic Floor Disorders

Epistasis between polymorphisms in COMT, ESR1, and GCH1 influences COMT enzyme activity and pain

Sensory Neuron-TRPV4 Modulates Temporomandibular Disorder Pain Via CGRP in Mice

Catechol- O-methyltransferase gene polymorphisms are associated with multiple pain-evoking stimuli

Mu Opioid Splice Variant MOR-1K Contributes to the Development of Opioid-Induced Hyperalgesia

Rationale and design of a multicenter randomized clinical trial of vestibulodynia: understanding pathophysiology and determining appropriate treatments (vestibulodynia: UPDATe)

A mouse model of chronic primary pain that integrates clinically relevant genetic vulnerability, stress, and minor injury

Sustained stimulation of β 2 - and β 3 -adrenergic receptors leads to persistent functional pain and neuroinflammation

Idiopathic pain disorders – Pathways of vulnerability

Expression of ectopic heat shock protein 90 in male and female primary afferent nociceptors regulates inflammatory pain

IL-23 Enhances C-Fiber-Mediated and Blue Light-Induced Spontaneous Pain in Female Mice

Role of Blood-Brain Barrier Dysfunction in Delirium following Non-cardiac Surgery in Older Adults

Epiregulin and EGFR interactions are involved in pain processing

Low catechol-O-methyltransferase and stress potentiate functional pain and depressive behavior, especially in female mice

Persistent Catechol-O-methyltransferase–dependent Pain Is Initiated by Peripheral β-Adrenergic Receptors

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