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Targeting amyloid clearance in Alzheimer's disease as a therapeutic strategy

British journal of pharmacology, 2019-09, Vol.176 (18), p.3447-3463

2019

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The amyloid precursor protein: A biochemical enigma in brain development, function and disease

FEBS letters, 2013-06, Vol.587 (13), p.2046-2054

2013

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Role of Prenatal Hypoxia in Brain Development, Cognitive Functions, and Neurodegeneration

Frontiers in neuroscience, 2018-11, Vol.12, p.825

2018

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Glucocorticoid-Dependent Mechanisms of Brain Tolerance to Hypoxia

International journal of molecular sciences, 2021-08, Vol.22 (15), p.7982

2021

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Angiotensin-converting enzyme 2 (ACE2): Two decades of revelations and re-evaluation

Peptides (New York, N.Y. : 1980), 2022-05, Vol.151, p.170766-170766, Article 170766

2022

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Lipid rafts and Alzheimer's disease: protein-lipid interactions and perturbation of signaling

Frontiers in physiology, 2012-01, Vol.3, p.189-189

2012

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Are amyloid-degrading enzymes viable therapeutic targets in Alzheimer's disease?

Journal of neurochemistry, 2012-01, Vol.120 (s1), p.167-185

2012

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Neuroadaptive Biochemical Mechanisms of Remote Ischemic Conditioning

International journal of molecular sciences, 2023-12, Vol.24 (23), p.17032

2023

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Editorial: Brain hypoxia and ischemia: New insights into neurodegeneration and neuroprotection, volume II

Frontiers in neuroscience, 2023-01, Vol.16, p.1125883-1125883

2023

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AChE and the amyloid precursor protein (APP) – Cross-talk in Alzheimer's disease

Chemico-biological interactions, 2016-11, Vol.259 (Pt B), p.301-306

2016

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Intermittent Hypoxic Training as an Effective Tool for Increasing the Adaptive Potential, Endurance and Working Capacity of the Brain

Frontiers in neuroscience, 2022-06, Vol.16, p.941740-941740

2022

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Caspase Inhibition Restores NEP Expression and Rescues Olfactory Deficit in Rats Caused by Prenatal Hypoxia

Journal of molecular neuroscience, 2022-07, Vol.72 (7), p.1516-1526

2022

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Special Issue in Honour of Anthony J (Tony) Turner

Neurochemical research, 2019-06, Vol.44 (6), p.1269-1270

2019

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The Aβ‐clearance protein transthyretin, like neprilysin, is epigenetically regulated by the amyloid precursor protein intracellular domain

Journal of neurochemistry, 2014-08, Vol.130 (3), p.419-431

2014

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Editorial: Brain Hypoxia and Ischemia: New Insights Into Neurodegeneration and Neuroprotection

Frontiers in neuroscience, 2019-07, Vol.13, p.770-770

2019

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Amyloid-degrading enzymes as therapeutic targets in Alzheimer's disease

Current Alzheimer research, 2008-04, Vol.5 (2), p.212

2008

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Neprilysin gene expression requires binding of the amyloid precursor protein intracellular domain to its promoter: implications for Alzheimer disease

EMBO reports, 2009-01, Vol.10 (1), p.94-100

2009

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Hypoxia Affects Neprilysin Expression Through Caspase Activation and an APP Intracellular Domain-dependent Mechanism

Frontiers in neuroscience, 2015-11, Vol.9, p.426-426

2015

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Effect of Global Brain Ischemia on Amyloid Precursor Protein Metabolism and Expression of Amyloid-Degrading Enzymes in Rat Cortex: Role in Pathogenesis of Alzheimer’s Disease

Biochemistry (Moscow), 2021-06, Vol.86 (6), p.680-692

2021

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The Transcriptionally Active Amyloid Precursor Protein (APP) Intracellular Domain Is Preferentially Produced from the 695 Isoform of APP in a β-Secretase-dependent Pathway

The Journal of biological chemistry, 2010-12, Vol.285 (53), p.41443-41454

2010

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Developmental Profile of Brain Neprilysin Expression Correlates with Olfactory Behaviour of Rats

Journal of molecular neuroscience, 2021-09, Vol.71 (9), p.1772-1785

2021

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Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease

Frontiers in aging neuroscience, 2014, Vol.6, p.235-235

2014

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Nuclear signalling by membrane protein intracellular domains: The AICD enigma

Cellular signalling, 2012-02, Vol.24 (2), p.402-409

2012

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Age-Dependent Electrocorticogram Dynamics and Epileptogenic Responsiveness in Rats Subjected to Prenatal Hypoxia

Developmental neuroscience, 2019, Vol.41 (1-2), p.56-66

2019

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Sodium valproate: an old drug with new roles

Trends in pharmacological sciences (Regular ed.), 2009-10, Vol.30 (10), p.509-514

2009

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Life Sciences & Biomedicine
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Animals
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Humans
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Neurosciences & Neurology
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Biochemistry & Molecular Biology
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Neprilysin
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Hypoxia
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Amyloid Precursor Protein
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Cell Biology
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Enzymes
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Alzheimer Disease - Metabolism
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Amyloid Beta-Protein Precursor - Metabolism
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Neurology
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Targeting amyloid clearance in Alzheimer's disease as a therapeutic strategy

The amyloid precursor protein: A biochemical enigma in brain development, function and disease

Role of Prenatal Hypoxia in Brain Development, Cognitive Functions, and Neurodegeneration

Glucocorticoid-Dependent Mechanisms of Brain Tolerance to Hypoxia

Angiotensin-converting enzyme 2 (ACE2): Two decades of revelations and re-evaluation

Lipid rafts and Alzheimer's disease: protein-lipid interactions and perturbation of signaling

Are amyloid-degrading enzymes viable therapeutic targets in Alzheimer's disease?

Neuroadaptive Biochemical Mechanisms of Remote Ischemic Conditioning

Editorial: Brain hypoxia and ischemia: New insights into neurodegeneration and neuroprotection, volume II

AChE and the amyloid precursor protein (APP) – Cross-talk in Alzheimer's disease

Intermittent Hypoxic Training as an Effective Tool for Increasing the Adaptive Potential, Endurance and Working Capacity of the Brain

Caspase Inhibition Restores NEP Expression and Rescues Olfactory Deficit in Rats Caused by Prenatal Hypoxia

Special Issue in Honour of Anthony J (Tony) Turner

The Aβ‐clearance protein transthyretin, like neprilysin, is epigenetically regulated by the amyloid precursor protein intracellular domain

Editorial: Brain Hypoxia and Ischemia: New Insights Into Neurodegeneration and Neuroprotection

Amyloid-degrading enzymes as therapeutic targets in Alzheimer's disease

Neprilysin gene expression requires binding of the amyloid precursor protein intracellular domain to its promoter: implications for Alzheimer disease

Hypoxia Affects Neprilysin Expression Through Caspase Activation and an APP Intracellular Domain-dependent Mechanism

Effect of Global Brain Ischemia on Amyloid Precursor Protein Metabolism and Expression of Amyloid-Degrading Enzymes in Rat Cortex: Role in Pathogenesis of Alzheimer’s Disease

The Transcriptionally Active Amyloid Precursor Protein (APP) Intracellular Domain Is Preferentially Produced from the 695 Isoform of APP in a β-Secretase-dependent Pathway

Developmental Profile of Brain Neprilysin Expression Correlates with Olfactory Behaviour of Rats

Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease

Nuclear signalling by membrane protein intracellular domains: The AICD enigma

Age-Dependent Electrocorticogram Dynamics and Epileptogenic Responsiveness in Rats Subjected to Prenatal Hypoxia

Sodium valproate: an old drug with new roles

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