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The Role of Ageing and Parenchymal Senescence on Macrophage Function and Fibrosis

Frontiers in immunology, 2021-06, Vol.12, p.700790-700790

2021

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Macrophages and Renal Fibrosis

Seminars in nephrology, 2010-05, Vol.30 (3), p.302-317

2010

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Conditional ablation of macrophages disrupts ovarian vasculature

Reproduction (Cambridge, England), 2011-06, Vol.141 (6), p.821-831

2011

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Depletion of cells of monocyte lineage prevents loss of renal microvasculature in murine kidney transplantation

Transplantation, 2008-11, Vol.86 (9), p.1267

2008

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Cellular senescence inhibits renal regeneration after injury in mice, with senolytic treatment promoting repair

Science translational medicine, 2021-05, Vol.13 (594)

2021

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Bone marrow transplantation modulates tissue macrophage phenotype and enhances cardiac recovery after subsequent acute myocardial infarction

Journal of molecular and cellular cardiology, 2016-01, Vol.90, p.120-128

2016

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Single-cell analysis of senescent epithelia reveals targetable mechanisms promoting fibrosis

JCI insight, 2022-11, Vol.7 (22)

2022

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Complementary Roles for Single-Nucleus and Single-Cell RNA Sequencing in Kidney Disease Research

Journal of the American Society of Nephrology, 2019-04, Vol.30 (4), p.712-713

2019

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Targeting Senescent Cells as Therapy for CKD

Kidney360, 2024-01, Vol.5 (1), p.142

2024

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Senolytic treatment preserves biliary regenerative capacity lost through cellular senescence during cold storage

Science translational medicine, 2022-12, Vol.14 (674), p.eabj4375

2022

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Alternatively Activated Macrophages Elicited by Helminth Infection Can Be Reprogrammed to Enable Microbial Killing

The Journal of immunology (1950), 2009-03, Vol.182 (5), p.3084-3094

2009

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Indian Hedgehog release from TNF-activated renal epithelia drives local and remote organ fibrosis

Science translational medicine, 2023-05, Vol.15 (698), p.eabn0736

2023

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Plasmodium chabaudi limits early Nippostrongylus brasiliensis-induced pulmonary immune activation and Th2 polarization in co-infected mice

BMC immunology, 2009-12, Vol.10 (1), p.60-60, Article 60

2009

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11β-HSD1 suppresses cardiac fibroblast CXCL2, CXCL5 and neutrophil recruitment to the heart post MI

Journal of endocrinology, 2017-06, Vol.233 (3), p.315-327

2017

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11 beta -HSD1 suppresses cardiac fibroblast CXCL2, CXCL5 and neutrophil recruitment to the heart post MI

Journal of endocrinology, 2017-06, Vol.233 (3), p.315-327

2017

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Cardiomyocyte and Vascular Smooth Muscle-Independent 11β-Hydroxysteroid Dehydrogenase 1 Amplifies Infarct Expansion, Hypertrophy, and the Development of Heart Failure After Myocardial Infarction in Male Mice

Endocrinology (Philadelphia), 2016-01, Vol.157 (1), p.346-357

2016

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The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an ‘M2’ phenotype in response to Th2 immunologic challenge

Immunobiology (1979), 2015-07, Vol.220 (7), p.924-933

2015

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Granulocyte macrophage-colony stimulating factor: A key modulator of renal mononuclear phagocyte plasticity

Immunobiology (1979), 2019-01, Vol.224 (1), p.60-74

2019

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Contrasting effects of Elg1-RFC and Ctf18-RFC inactivation in the absence of fully functional RFC in fission yeast

Nucleic acids research, 2005-01, Vol.33 (13), p.4078

2005

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Single-Cell Analysis of Senescent Epithelia Reveals Targetable Mechanisms Promoting Fibrosis: PO0643

Journal of the American Society of Nephrology, 2021-10, Vol.32 (10S), p.236-236

2021

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216 Immediate Pharmacological Inhibition of Local Glucocorticoid Generation increases Angiogenesis and Improves Cardiac Funcion after Myocardial Infarction

Heart (British Cardiac Society), 2014-06, Vol.100 (Suppl 3), p.A118-A118

2014

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Cardiomyocyte and Vascular Smooth Muscle-Independent 11[beta]-Hydroxysteroid Dehydrogenase 1 Amplifies Infarct Expansion, Hypertrophy, and the Development of Heart Failure After Myocardial Infarction in Male Mice

Endocrinology (Philadelphia), 2016-01, Vol.157 (1), p.346-357

2016

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P4 Myocardial ischaemic injury is unexpectedly increased in mice lacking the H2S metabolising enzyme thiosulfate sulfurtransferase

Nitric oxide, 2014-05, Vol.39, p.S17-S17

2014

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Survival analysis bewteen patients with invasive ductal and invasive lobular breast cancer

Geburtshilfe und Frauenheilkunde, 2006-09, Vol.66 (S 01)

2006

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MYELOID CELL 11β-HSD1 REGULATES THE INFLAMMATORY RESPONSE DURING MYOCARDIAL INFARCT HEALING AND PROTECTS THE HEART FROM DETRIMENTAL REMODELLING AFTER IRRADIATION AND BONE MARROW TRANSFER

Heart (British Cardiac Society), 2012-11, Vol.98 (Suppl 5), p.A2-A2

2012

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The Role of Ageing and Parenchymal Senescence on Macrophage Function and Fibrosis

Macrophages and Renal Fibrosis

Conditional ablation of macrophages disrupts ovarian vasculature

Depletion of cells of monocyte lineage prevents loss of renal microvasculature in murine kidney transplantation

Cellular senescence inhibits renal regeneration after injury in mice, with senolytic treatment promoting repair

Bone marrow transplantation modulates tissue macrophage phenotype and enhances cardiac recovery after subsequent acute myocardial infarction

Single-cell analysis of senescent epithelia reveals targetable mechanisms promoting fibrosis

Complementary Roles for Single-Nucleus and Single-Cell RNA Sequencing in Kidney Disease Research

Targeting Senescent Cells as Therapy for CKD

Senolytic treatment preserves biliary regenerative capacity lost through cellular senescence during cold storage

Alternatively Activated Macrophages Elicited by Helminth Infection Can Be Reprogrammed to Enable Microbial Killing

Indian Hedgehog release from TNF-activated renal epithelia drives local and remote organ fibrosis

Plasmodium chabaudi limits early Nippostrongylus brasiliensis-induced pulmonary immune activation and Th2 polarization in co-infected mice

11β-HSD1 suppresses cardiac fibroblast CXCL2, CXCL5 and neutrophil recruitment to the heart post MI

11 beta -HSD1 suppresses cardiac fibroblast CXCL2, CXCL5 and neutrophil recruitment to the heart post MI

Cardiomyocyte and Vascular Smooth Muscle-Independent 11β-Hydroxysteroid Dehydrogenase 1 Amplifies Infarct Expansion, Hypertrophy, and the Development of Heart Failure After Myocardial Infarction in Male Mice

The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an ‘M2’ phenotype in response to Th2 immunologic challenge

Granulocyte macrophage-colony stimulating factor: A key modulator of renal mononuclear phagocyte plasticity

Contrasting effects of Elg1-RFC and Ctf18-RFC inactivation in the absence of fully functional RFC in fission yeast

Single-Cell Analysis of Senescent Epithelia Reveals Targetable Mechanisms Promoting Fibrosis: PO0643

216 Immediate Pharmacological Inhibition of Local Glucocorticoid Generation increases Angiogenesis and Improves Cardiac Funcion after Myocardial Infarction

Cardiomyocyte and Vascular Smooth Muscle-Independent 11[beta]-Hydroxysteroid Dehydrogenase 1 Amplifies Infarct Expansion, Hypertrophy, and the Development of Heart Failure After Myocardial Infarction in Male Mice

P4 Myocardial ischaemic injury is unexpectedly increased in mice lacking the H2S metabolising enzyme thiosulfate sulfurtransferase

Survival analysis bewteen patients with invasive ductal and invasive lobular breast cancer

MYELOID CELL 11β-HSD1 REGULATES THE INFLAMMATORY RESPONSE DURING MYOCARDIAL INFARCT HEALING AND PROTECTS THE HEART FROM DETRIMENTAL REMODELLING AFTER IRRADIATION AND BONE MARROW TRANSFER

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