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Combinatorial effects of multiple enhancer variants in linkage disequilibrium dictate levels of gene expression to confer susceptibility to common traits

Genome research, 2014-01, Vol.24 (1), p.1

2014

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Epigenomic Enhancer Profiling Defines a Signature of Colon Cancer

Science (American Association for the Advancement of Science), 2012-05, Vol.336 (6082), p.736-739

2012

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BCL9 Promotes Tumor Progression by Conferring Enhanced Proliferative, Metastatic, and Angiogenic Properties to Cancer Cells

Cancer research (Chicago, Ill.), 2009-10, Vol.69 (19), p.7577-7586

2009

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Hotspots of aberrant enhancer activity punctuate the colorectal cancer epigenome

Nature communications, 2017-02, Vol.8 (1), p.14400-14400, Article 14400

2017

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Induction of KIAA1199/CEMIP is associated with colon cancer phenotype and poor patient survival

Oncotarget, 2015-10, Vol.6 (31), p.30500-30515

2015

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Detection in Fecal DNA of Colon Cancer–Specific Methylation of the Nonexpressed Vimentin Gene

JNCI : Journal of the National Cancer Institute, 2005-08, Vol.97 (15), p.1124-1132

2005

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Discovery and Scoring of Protein Interaction Subnetworks Discriminative of Late Stage Human Colon Cancer

Molecular & cellular proteomics, 2009-04, Vol.8 (4), p.827-845

2009

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Transposon mutagenesis identifies candidate genes that cooperate with loss of transforming growth factor‐beta signaling in mouse intestinal neoplasms

International journal of cancer, 2017-02, Vol.140 (4), p.853-863

2017

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SLC5A8, a Sodium Transporter, Is a Tumor Suppressor Gene Silenced by Methylation in Human Colon Aberrant Crypt Foci and Cancers

Proceedings of the National Academy of Sciences - PNAS, 2003-07, Vol.100 (14), p.8412-8417

2003

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Two Distinct Categories of Focal Deletions in Cancer Genomes

PloS one, 2013-06, Vol.8 (6), p.e66264

2013

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Mutational inactivation of TGFBR2 in microsatellite unstable colon cancer arises from the cooperation of genomic instability and the clonal outgrowth of transforming growth factor β resistant cells

Genes chromosomes & cancer, 2008-02, Vol.47 (2), p.95-106

2008

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HLTF Gene Silencing in Human Colon Cancer

Proceedings of the National Academy of Sciences - PNAS, 2002-04, Vol.99 (7), p.4562-4567

2002

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Increased nm23-H1 and nm23-H2 Messenger RNA Expression and Absence of Mutations in Colon Carcinomas of Low and High Metastatic Potential

JNCI : Journal of the National Cancer Institute, 1993-01, Vol.85 (2), p.147-152

1993

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Microsatellite instability and mutations of the transforming growth factor β type II receptor gene in colorectal cancer

Cancer research (Chicago, Ill.), 1995-12, Vol.55 (23), p.5548-5550

1995

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Abstract 3126: CEMIP, a secreted protein highly induced in colon cancer and associated with poor patient survival

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.3126-3126

2016

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Mutational inactivation of transforming growth factor β receptor type II in microsatellite stable colon cancers

Cancer research (Chicago, Ill.), 1999-01, Vol.59 (2), p.320-324

1999

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Chromosomal autonomy of hMLH1 methylation in colon cancer

Oncogene, 2002-02, Vol.21 (9), p.1443-1449

2002

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Inactivation of the Type II TGF-β Receptor in Colon Cancer Cells with Microsatellite Instability

Science (American Association for the Advancement of Science), 1995-06, Vol.268 (5215), p.1336-1338

1995

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H3K4me3 inversely correlates with DNA methylation at a large class of non-CpG-island-containing start sites

Genome medicine, 2012-05, Vol.4 (5), p.47-47

2012

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A transforming growth factor beta receptor type II gene mutation common in colon and gastric but rare in endometrial cancers with microsatellite instability

Cancer research (Chicago, Ill.), 1995-12, Vol.55 (23), p.5545-5547

1995

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Mutation of the type II transforming growth factor-β receptor is coincident with the transformation of human colon adenomas to malignant carcinomas

Cancer research (Chicago, Ill.), 1998-07, Vol.58 (14), p.3101-3104

1998

Volltextzugriff (PDF)

Discovery and Scoring of Protein Interaction Subnetworks Discriminative of Late Stage Human Colon CancerS

Molecular & cellular proteomics, 2009-04, Vol.8 (4), p.827-845

2009

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Demonstration That Mutation of the Type II Transforming Growth Factor β Receptor Inactivates Its Tumor Suppressor Activity in Replication Error-positive Colon Carcinoma Cells (∗)

The Journal of biological chemistry, 1995-09, Vol.270 (37), p.22044-22049

1995

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Nm23—Into the Basement (Membrane)

JNCI : Journal of the National Cancer Institute, 1994-12, Vol.86 (24), p.1815-1817

1994

Volltextzugriff (PDF)

Chromosomal autonomy of hMLH1 methylation in colon cancer

Oncogene, 2002-02, Vol.21 (9), p.1443-1449

2002

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Combinatorial effects of multiple enhancer variants in linkage disequilibrium dictate levels of gene expression to confer susceptibility to common traits

Epigenomic Enhancer Profiling Defines a Signature of Colon Cancer

BCL9 Promotes Tumor Progression by Conferring Enhanced Proliferative, Metastatic, and Angiogenic Properties to Cancer Cells

Hotspots of aberrant enhancer activity punctuate the colorectal cancer epigenome

Induction of KIAA1199/CEMIP is associated with colon cancer phenotype and poor patient survival

Detection in Fecal DNA of Colon Cancer–Specific Methylation of the Nonexpressed Vimentin Gene

Discovery and Scoring of Protein Interaction Subnetworks Discriminative of Late Stage Human Colon Cancer

Transposon mutagenesis identifies candidate genes that cooperate with loss of transforming growth factor‐beta signaling in mouse intestinal neoplasms

SLC5A8, a Sodium Transporter, Is a Tumor Suppressor Gene Silenced by Methylation in Human Colon Aberrant Crypt Foci and Cancers

Two Distinct Categories of Focal Deletions in Cancer Genomes

Mutational inactivation of TGFBR2 in microsatellite unstable colon cancer arises from the cooperation of genomic instability and the clonal outgrowth of transforming growth factor β resistant cells

HLTF Gene Silencing in Human Colon Cancer

Increased nm23-H1 and nm23-H2 Messenger RNA Expression and Absence of Mutations in Colon Carcinomas of Low and High Metastatic Potential

Microsatellite instability and mutations of the transforming growth factor β type II receptor gene in colorectal cancer

Abstract 3126: CEMIP, a secreted protein highly induced in colon cancer and associated with poor patient survival

Mutational inactivation of transforming growth factor β receptor type II in microsatellite stable colon cancers

Chromosomal autonomy of hMLH1 methylation in colon cancer

Inactivation of the Type II TGF-β Receptor in Colon Cancer Cells with Microsatellite Instability

H3K4me3 inversely correlates with DNA methylation at a large class of non-CpG-island-containing start sites

A transforming growth factor beta receptor type II gene mutation common in colon and gastric but rare in endometrial cancers with microsatellite instability

Mutation of the type II transforming growth factor-β receptor is coincident with the transformation of human colon adenomas to malignant carcinomas

Discovery and Scoring of Protein Interaction Subnetworks Discriminative of Late Stage Human Colon CancerS

Demonstration That Mutation of the Type II Transforming Growth Factor β Receptor Inactivates Its Tumor Suppressor Activity in Replication Error-positive Colon Carcinoma Cells (∗)

Nm23—Into the Basement (Membrane)

Chromosomal autonomy of hMLH1 methylation in colon cancer

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