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MAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets

Clinical science (1979), 2008-10, Vol.115 (7), p.203-218

Muslin, Anthony J

2008

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Akt1 is required for physiological cardiac growth

Circulation (New York, N.Y.), 2006-05, Vol.113 (17), p.2097-2104

2006

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Reversion of cardiac dysfunction by a novel orally available calcium/calmodulin-dependent protein kinase II inhibitor, RA306, in a genetic model of dilated cardiomyopathy

Cardiovascular research, 2020-02, Vol.116 (2), p.329-338

2020

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14-3-3 proteins: regulation of subcellular localization by molecular interference

Cellular Signalling, 2000-12, Vol.12 (11), p.703-709

2000

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First‐in‐human study of the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple oral doses of SAR247799, a selective G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist for endothelial protection

British journal of clinical pharmacology, 2021-02, Vol.87 (2), p.598-611

2021

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Akt2 Regulates Cardiac Metabolism and Cardiomyocyte Survival

The Journal of biological chemistry, 2006-10, Vol.281 (43), p.32841-32851

2006

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Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload

Circulation (New York, N.Y.), 2004-08, Vol.110 (6), p.718-723

2004

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Multiple docking sites on substrate proteins form a modular system that mediates recognition by ERK MAP kinase

Genes & development, 1999-01, Vol.13 (2), p.163-175

1999

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Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

Science (American Association for the Advancement of Science), 1995-04, Vol.268 (5207), p.100-102

1995

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Cation Diffusion Facilitator Proteins Modulate Raf-1 Activity

The Journal of biological chemistry, 2004-06, Vol.279 (26), p.27807-27815

2004

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Molecular Basis for Zinc Transporter 1 Action as an Endogenous Inhibitor of L-type Calcium Channels

The Journal of biological chemistry, 2009-11, Vol.284 (47), p.32434-32443

2009

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Zinc Ions and Cation Diffusion Facilitator Proteins Regulate Ras-Mediated Signaling

Developmental cell, 2002-05, Vol.2 (5), p.567-578

2002

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Interaction of 14-3-3 with Signaling Proteins Is Mediated by the Recognition of Phosphoserine

Cell, 1996-03, Vol.84 (6), p.889-897

1996

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Role of Raf Proteins in Cardiac Hypertrophy and Cardiomyocyte Survival

Trends in cardiovascular medicine, 2005-08, Vol.15 (6), p.225-229

Muslin, Anthony J.

2005

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RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm

Kidney international, 2015-04, Vol.87 (4), p.771-783

2015

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The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis

The Journal of clinical investigation, 2003-03, Vol.111 (6), p.833-841

2003

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RGS4, a GTPase activator, improves renal function in ischemia–reperfusion injury

Kidney international, 2011-08, Vol.80 (3), p.263-271

2011

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ZnT-1 protects HL-1 cells from simulated ischemia–reperfusion through activation of Ras–ERK signaling

Journal of molecular medicine (Berlin, Germany), 2012-02, Vol.90 (2), p.127-138

2012

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14-3-3 proteins block apoptosis and differentially regulate MAPK cascades

The EMBO Journal, 2000-02, Vol.19 (3), p.349-358

2000

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14-3-3 Protein Protects Against Cardiac Endoplasmic Reticulum Stress (ERS) and ERS-Initiated Apoptosis in Experimental Diabetes

Journal of Pharmacological Sciences, 2010, Vol.113(4), pp.325-334

2010

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RGS4 Controls Renal Blood Flow and Inhibits Cyclosporine‐Mediated Nephrotoxicity

American journal of transplantation, 2010-02, Vol.10 (2), p.231-241

2010

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HOX11 interacts with protein phosphatases PP2A and PP1 and disrupts a G2/M cell-cycle checkpoint

Nature (London), 1997-01, Vol.385 (6615), p.454-458

1997

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The Application of Phenotypic High-Throughput Screening Techniques to Cardiovascular Research

Trends in cardiovascular medicine, 2009-08, Vol.19 (6), p.207-212

2009

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RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload

The Journal of clinical investigation, 1999-09, Vol.104 (5), p.567-576

1999

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Uremic cardiac hypertrophy is reversed by rapamycin but not by lowering of blood pressure

Kidney international, 2009-04, Vol.75 (8), p.800-808

2009

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MAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets

Akt1 is required for physiological cardiac growth

Reversion of cardiac dysfunction by a novel orally available calcium/calmodulin-dependent protein kinase II inhibitor, RA306, in a genetic model of dilated cardiomyopathy

14-3-3 proteins: regulation of subcellular localization by molecular interference

First‐in‐human study of the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple oral doses of SAR247799, a selective G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist for endothelial protection

Akt2 Regulates Cardiac Metabolism and Cardiomyocyte Survival

Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload

Multiple docking sites on substrate proteins form a modular system that mediates recognition by ERK MAP kinase

Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

Cation Diffusion Facilitator Proteins Modulate Raf-1 Activity

Molecular Basis for Zinc Transporter 1 Action as an Endogenous Inhibitor of L-type Calcium Channels

Zinc Ions and Cation Diffusion Facilitator Proteins Regulate Ras-Mediated Signaling

Interaction of 14-3-3 with Signaling Proteins Is Mediated by the Recognition of Phosphoserine

Role of Raf Proteins in Cardiac Hypertrophy and Cardiomyocyte Survival

RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm

The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis

RGS4, a GTPase activator, improves renal function in ischemia–reperfusion injury

ZnT-1 protects HL-1 cells from simulated ischemia–reperfusion through activation of Ras–ERK signaling

14-3-3 proteins block apoptosis and differentially regulate MAPK cascades

14-3-3 Protein Protects Against Cardiac Endoplasmic Reticulum Stress (ERS) and ERS-Initiated Apoptosis in Experimental Diabetes

RGS4 Controls Renal Blood Flow and Inhibits Cyclosporine‐Mediated Nephrotoxicity

HOX11 interacts with protein phosphatases PP2A and PP1 and disrupts a G2/M cell-cycle checkpoint

The Application of Phenotypic High-Throughput Screening Techniques to Cardiovascular Research

RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload

Uremic cardiac hypertrophy is reversed by rapamycin but not by lowering of blood pressure

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