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MAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets

Clinical science (1979), 2008-10, Vol.115 (7), p.203

Muslin, Anthony J

2008

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Akt1 is required for physiological cardiac growth

Circulation (New York, N.Y.), 2006-05, Vol.113 (17), p.2097-2104

2006

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Reversion of cardiac dysfunction by a novel orally available calcium/calmodulin-dependent protein kinase II inhibitor, RA306, in a genetic model of dilated cardiomyopathy

Cardiovascular research, 2020-02, Vol.116 (2), p.329-338

2020

Volltextzugriff (PDF)

First‐in‐human study of the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple oral doses of SAR247799, a selective G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist for endothelial protection

British journal of clinical pharmacology, 2021-02, Vol.87 (2), p.598-611

2021

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Akt2 Regulates Cardiac Metabolism and Cardiomyocyte Survival

The Journal of biological chemistry, 2006-10, Vol.281 (43), p.32841-32851

2006

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Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload

Circulation (New York, N.Y.), 2004-08, Vol.110 (6), p.718-723

2004

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Molecular Basis for Zinc Transporter 1 Action as an Endogenous Inhibitor of L-type Calcium Channels

The Journal of biological chemistry, 2009-11, Vol.284 (47), p.32434-32443

2009

Volltextzugriff (PDF)

RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm

Kidney international, 2015-04, Vol.87 (4), p.771-783

2015

Volltextzugriff (PDF)

The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis

The Journal of clinical investigation, 2003-03, Vol.111 (6), p.833-841

2003

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RGS4, a GTPase activator, improves renal function in ischemia–reperfusion injury

Kidney international, 2011-08, Vol.80 (3), p.263-271

2011

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ZnT-1 protects HL-1 cells from simulated ischemia–reperfusion through activation of Ras–ERK signaling

Journal of molecular medicine (Berlin, Germany), 2012-02, Vol.90 (2), p.127-138

2012

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Cation Diffusion Facilitator Proteins Modulate Raf-1 Activity

The Journal of biological chemistry, 2004-06, Vol.279 (26), p.27807-27815

2004

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14-3-3 Protein Protects Against Cardiac Endoplasmic Reticulum Stress (ERS) and ERS-Initiated Apoptosis in Experimental Diabetes

Journal of Pharmacological Sciences, 2010, Vol.113(4), pp.325-334

2010

Volltextzugriff (PDF)

Role of Raf Proteins in Cardiac Hypertrophy and Cardiomyocyte Survival

Trends in cardiovascular medicine, 2005-08, Vol.15 (6), p.225-229

Muslin, Anthony J.

2005

Volltextzugriff (PDF)

The Application of Phenotypic High-Throughput Screening Techniques to Cardiovascular Research

Trends in cardiovascular medicine, 2009-08, Vol.19 (6), p.207-212

2009

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Zinc Ions and Cation Diffusion Facilitator Proteins Regulate Ras-Mediated Signaling

Developmental cell, 2002-05, Vol.2 (5), p.567-578

2002

Volltextzugriff (PDF)

Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

Science (American Association for the Advancement of Science), 1995-04, Vol.268 (5207), p.100-102

1995

Volltextzugriff (PDF)

Uremic cardiac hypertrophy is reversed by rapamycin but not by lowering of blood pressure

Kidney international, 2009-04, Vol.75 (8), p.800-808

2009

Volltextzugriff (PDF)

Akt2: A Critical Regulator of Cardiomyocyte Survival and Metabolism

Pediatric cardiology, 2011-03, Vol.32 (3), p.317-322

Muslin, Anthony J.

2011

Volltextzugriff (PDF)

The 14-3-3τ Phosphoserine-Binding Protein Is Required for Cardiomyocyte Survival

Molecular and Cellular Biology, 2007-02, Vol.27 (4), p.1455-1466

2007

Volltextzugriff (PDF)

14-3-3 proteins: regulation of subcellular localization by molecular interference

Cellular Signalling, 2000-12, Vol.12 (11), p.703-709

2000

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Endothelial‐protective effects of a G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist, SAR247799, in type‐2 diabetes rats and a randomized placebo‐controlled patient trial

British journal of clinical pharmacology, 2021-05, Vol.87 (5), p.2303-2320

2021

Volltextzugriff (PDF)

Role of 14-3-3-Mediated p38 Mitogen-Activated Protein Kinase Inhibition in Cardiac Myocyte Survival

Circulation research, 2003-11, Vol.93 (11), p.1026-1028

2003

Volltextzugriff (PDF)

Dominant negative 14-3-3 promotes cardiomyocyte apoptosis in early stage of type I diabetes mellitus through activation of JNK

Biochemical and biophysical research communications, 2004-07, Vol.320 (3), p.773-780

2004

Volltextzugriff (PDF)

ATM-dependent suppression of stress signaling reduces vascular disease in metabolic syndrome

Cell metabolism, 2006-11, Vol.4 (5), p.377-389

2006

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MAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets

Akt1 is required for physiological cardiac growth

Reversion of cardiac dysfunction by a novel orally available calcium/calmodulin-dependent protein kinase II inhibitor, RA306, in a genetic model of dilated cardiomyopathy

First‐in‐human study of the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple oral doses of SAR247799, a selective G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist for endothelial protection

Akt2 Regulates Cardiac Metabolism and Cardiomyocyte Survival

Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload

Molecular Basis for Zinc Transporter 1 Action as an Endogenous Inhibitor of L-type Calcium Channels

RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm

The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis

RGS4, a GTPase activator, improves renal function in ischemia–reperfusion injury

ZnT-1 protects HL-1 cells from simulated ischemia–reperfusion through activation of Ras–ERK signaling

Cation Diffusion Facilitator Proteins Modulate Raf-1 Activity

14-3-3 Protein Protects Against Cardiac Endoplasmic Reticulum Stress (ERS) and ERS-Initiated Apoptosis in Experimental Diabetes

Role of Raf Proteins in Cardiac Hypertrophy and Cardiomyocyte Survival

The Application of Phenotypic High-Throughput Screening Techniques to Cardiovascular Research

Zinc Ions and Cation Diffusion Facilitator Proteins Regulate Ras-Mediated Signaling

Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

Uremic cardiac hypertrophy is reversed by rapamycin but not by lowering of blood pressure

Akt2: A Critical Regulator of Cardiomyocyte Survival and Metabolism

The 14-3-3τ Phosphoserine-Binding Protein Is Required for Cardiomyocyte Survival

14-3-3 proteins: regulation of subcellular localization by molecular interference

Endothelial‐protective effects of a G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist, SAR247799, in type‐2 diabetes rats and a randomized placebo‐controlled patient trial

Role of 14-3-3-Mediated p38 Mitogen-Activated Protein Kinase Inhibition in Cardiac Myocyte Survival

Dominant negative 14-3-3 promotes cardiomyocyte apoptosis in early stage of type I diabetes mellitus through activation of JNK

ATM-dependent suppression of stress signaling reduces vascular disease in metabolic syndrome

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