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Bilirubin-Induced Oxidative Stress Leads to DNA Damage in the Cerebellum of Hyperbilirubinemic Neonatal Mice and Activates DNA Double-Strand Break Repair Pathways in Human Cells

Oxidative medicine and cellular longevity, 2018-01, Vol.2018 (2018), p.1-11

2018

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Gene Therapy in Patients with the Crigler–Najjar Syndrome

The New England journal of medicine, 2023-08, Vol.389 (7), p.620-631

2023

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Experimental models assessing bilirubin neurotoxicity

Pediatric research, 2020-01, Vol.87 (1), p.17-25

2020

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Attenuation of neuro-inflammation improves survival and neurodegeneration in a mouse model of severe neonatal hyperbilirubinemia

Brain, behavior, and immunity, 2018-05, Vol.70, p.166-178

2018

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Fibronectin splice variants: Understanding their multiple roles in health and disease using engineered mouse models

IUBMB life, 2011-07, Vol.63 (7), p.538-546

2011

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An Essential Role for Fibronectin Extra Type III Domain A in Pulmonary Fibrosis

American journal of respiratory and critical care medicine, 2008-03, Vol.177 (6), p.638-645

2008

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Inflammatory signature of cerebellar neurodegeneration during neonatal hyperbilirubinemia in Ugt1 -/- mouse model

Journal of neuroinflammation, 2017-03, Vol.14 (1), p.64-64

2017

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Fludarabine increases nuclease-free AAV- and CRISPR/Cas9-mediated homologous recombination in mice

Nature biotechnology, 2022-08, Vol.40 (8), p.1285-1294

2022

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Promoterless gene targeting without nucleases rescues lethality of a Crigler‐Najjar syndrome mouse model

EMBO molecular medicine, 2017-10, Vol.9 (10), p.1346-1355

2017

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Rescue of bilirubin‐induced neonatal lethality in a mouse model of Crigler‐Najjar syndrome type I by AAV9‐mediated gene transfer

The FASEB journal, 2012-03, Vol.26 (3), p.1052-1063

2012

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Integrin-α9 Is Required for Fibronectin Matrix Assembly during Lymphatic Valve Morphogenesis

Developmental cell, 2009-08, Vol.17 (2), p.175-186

2009

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Low efficacy of recombinant SV40 in Ugt1a1-/- mice with severe inherited hyperbilirubinemia

PloS one, 2021-01, Vol.16 (4), p.e0250605-e0250605

2021

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Formation of an invasion-permissive matrix requires TGFβ/SNAIL1-regulated alternative splicing of fibronectin

Breast cancer research : BCR, 2023-11, Vol.25 (1), p.143-143, Article 143

2023

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Life-long correction of hyperbilirubinemia with a neonatal liver-specific AAV-mediated gene transfer in a lethal mouse model of Crigler-Najjar Syndrome

Human gene therapy, 2014-09, Vol.25 (9), p.844-855

2014

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TDP-43 regulates β-adducin (Add2) transcript stability

RNA biology, 2014-10, Vol.11 (10), p.1280-1290

2014

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Lethality rescue and long-term amelioration of a citrullinemia type I mouse model by neonatal gene-targeting combined to SaCRISPR-Cas9

Molecular therapy. Methods & clinical development, 2023-12, Vol.31, p.101103-101103, Article 101103

2023

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Recipient-derived EDA fibronectin promotes cardiac allograft fibrosis

The Journal of pathology, 2012-03, Vol.226 (4), p.609-618

2012

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Age-dependent pattern of cerebellar susceptibility to bilirubin neurotoxicity in vivo in mice

Disease models & mechanisms, 2014-09, Vol.7 (9), p.1057-1068

2014

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Lack of Fibronectin Extra Domain A Alternative Splicing Exacerbates Endothelial Dysfunction in Diabetes

Scientific reports, 2016-11, Vol.6 (1), p.37965-37965, Article 37965

2016

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Promoterless Gene Targeting Approach Combined to CRISPR/Cas9 Efficiently Corrects Hemophilia B Phenotype in Neonatal Mice

Frontiers in genome editing, 2022-03, Vol.4, p.785698

2022

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Brief Report: Alternative Splicing of Extra Domain A (EIIIA) of Fibronectin Plays a Tissue‐Specific Role in Hematopoietic Homeostasis

Stem cells (Dayton, Ohio), 2016-08, Vol.34 (8), p.2263-2268

2016

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Regulated Splicing of the Fibronectin EDA Exon Is Essential for Proper Skin Wound Healing and Normal Lifespan

The Journal of cell biology, 2003-07, Vol.162 (1), p.149-160

2003

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Control of fibroblast fibronectin expression and alternative splicing via the PI3K/Akt/mTOR pathway

Experimental cell research, 2010-10, Vol.316 (16), p.2644-2653

2010

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Generation of Ugt1-deficient murine liver cell lines using TALEN technology

PloS one, 2014-08, Vol.9 (8), p.e104816

2014

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A Major Fraction of Fibronectin Present in the Extracellular Matrix of Tissues Is Plasma-derived

The Journal of biological chemistry, 2007-09, Vol.282 (38), p.28057-28062

2007

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Bilirubin-Induced Oxidative Stress Leads to DNA Damage in the Cerebellum of Hyperbilirubinemic Neonatal Mice and Activates DNA Double-Strand Break Repair Pathways in Human Cells

Gene Therapy in Patients with the Crigler–Najjar Syndrome

Experimental models assessing bilirubin neurotoxicity

Attenuation of neuro-inflammation improves survival and neurodegeneration in a mouse model of severe neonatal hyperbilirubinemia

Fibronectin splice variants: Understanding their multiple roles in health and disease using engineered mouse models

An Essential Role for Fibronectin Extra Type III Domain A in Pulmonary Fibrosis

Inflammatory signature of cerebellar neurodegeneration during neonatal hyperbilirubinemia in Ugt1 -/- mouse model

Fludarabine increases nuclease-free AAV- and CRISPR/Cas9-mediated homologous recombination in mice

Promoterless gene targeting without nucleases rescues lethality of a Crigler‐Najjar syndrome mouse model

Rescue of bilirubin‐induced neonatal lethality in a mouse model of Crigler‐Najjar syndrome type I by AAV9‐mediated gene transfer

Integrin-α9 Is Required for Fibronectin Matrix Assembly during Lymphatic Valve Morphogenesis

Low efficacy of recombinant SV40 in Ugt1a1-/- mice with severe inherited hyperbilirubinemia

Formation of an invasion-permissive matrix requires TGFβ/SNAIL1-regulated alternative splicing of fibronectin

Life-long correction of hyperbilirubinemia with a neonatal liver-specific AAV-mediated gene transfer in a lethal mouse model of Crigler-Najjar Syndrome

TDP-43 regulates β-adducin (Add2) transcript stability

Lethality rescue and long-term amelioration of a citrullinemia type I mouse model by neonatal gene-targeting combined to SaCRISPR-Cas9

Recipient-derived EDA fibronectin promotes cardiac allograft fibrosis

Age-dependent pattern of cerebellar susceptibility to bilirubin neurotoxicity in vivo in mice

Lack of Fibronectin Extra Domain A Alternative Splicing Exacerbates Endothelial Dysfunction in Diabetes

Promoterless Gene Targeting Approach Combined to CRISPR/Cas9 Efficiently Corrects Hemophilia B Phenotype in Neonatal Mice

Brief Report: Alternative Splicing of Extra Domain A (EIIIA) of Fibronectin Plays a Tissue‐Specific Role in Hematopoietic Homeostasis

Regulated Splicing of the Fibronectin EDA Exon Is Essential for Proper Skin Wound Healing and Normal Lifespan

Control of fibroblast fibronectin expression and alternative splicing via the PI3K/Akt/mTOR pathway

Generation of Ugt1-deficient murine liver cell lines using TALEN technology

A Major Fraction of Fibronectin Present in the Extracellular Matrix of Tissues Is Plasma-derived

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