UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 86

Sortiert nach

Crystal nephropathies: mechanisms of crystal-induced kidney injury

Nature reviews. Nephrology, 2017-04, Vol.13 (4), p.226

2017

Volltextzugriff (PDF)

Neutrophils and Neutrophil Extracellular Traps Drive Necroinflammation in COVID-19

Cells (Basel, Switzerland), 2020-06, Vol.9 (6), p.1383

2020

Volltextzugriff (PDF)

Matters of life and death. How neutrophils die or survive along NET release and is “NETosis” = necroptosis?

Cellular and molecular life sciences : CMLS, 2016-06, Vol.73 (11-12), p.2211-2219

2016

Volltextzugriff (PDF)

The macrophage phenotype and inflammasome component NLRP3 contributes to nephrocalcinosis-related chronic kidney disease independent from IL-1–mediated tissue injury

Kidney international, 2018-03, Vol.93 (3), p.656-669

2018

Volltextzugriff (PDF)

Synchronized renal tubular cell death involves ferroptosis

Proceedings of the National Academy of Sciences - PNAS, 2014-11, Vol.111 (47), p.16836-16841

2014

Volltextzugriff (PDF)

Multifactorial functions of the inflammasome component NLRP3 in pathogenesis of chronic kidney diseases

Kidney international, 2019-07, Vol.96 (1), p.58-66

Mulay, Shrikant R.

2019

Volltextzugriff (PDF)

Cytotoxicity of crystals involves RIPK3-MLKL-mediated necroptosis

Nature communications, 2016-01, Vol.7 (1), p.10274-10274, Article 10274

2016

Volltextzugriff (PDF)

Macrophage Phenotype Controls Long-Term AKI Outcomes—Kidney Regeneration versus Atrophy

Journal of the American Society of Nephrology, 2014-02, Vol.25 (2), p.292-304

2014

Volltextzugriff (PDF)

Intestinal Dysbiosis, Barrier Dysfunction, and Bacterial Translocation Account for CKD-Related Systemic Inflammation

Journal of the American Society of Nephrology, 2017-01, Vol.28 (1), p.76-83

2017

Volltextzugriff (PDF)

PMA and crystal‐induced neutrophil extracellular trap formation involves RIPK1‐RIPK3‐MLKL signaling

European journal of immunology, 2016-01, Vol.46 (1), p.223-229

2016

Volltextzugriff (PDF)

Editorial: Negative Regulators of Innate Immunity and Their Role in Host Responses to Injury and Infection

Frontiers in immunology, 2022-03, Vol.13, p.891919-891919

2022

Volltextzugriff (PDF)

Neutrophil Extracellular Trap-Related Extracellular Histones Cause Vascular Necrosis in Severe GN

Journal of the American Society of Nephrology, 2015-10, Vol.26 (10), p.2399-2413

2015

Volltextzugriff (PDF)

Cellular and Molecular Mechanisms of Kidney Injury in 2,8-Dihydroxyadenine Nephropathy

Journal of the American Society of Nephrology, 2020-04, Vol.31 (4), p.799-816

2020

Volltextzugriff (PDF)

RGMb protects against acute kidney injury by inhibiting tubular cell necroptosis via an MLKL-dependent mechanism

Proceedings of the National Academy of Sciences - PNAS, 2018-02, Vol.115 (7), p.E1475-E1484

2018

Volltextzugriff (PDF)

Toll-Like Receptor 4-Induced IL-22 Accelerates Kidney Regeneration

Journal of the American Society of Nephrology, 2014-05, Vol.25 (5), p.978-989

2014

Volltextzugriff (PDF)

p53-Independent Roles of MDM2 in NF-κB Signaling: Implications for Cancer Therapy, Wound Healing, and Autoimmune Diseases

Neoplasia (New York, N.Y.), 2012-12, Vol.14 (12), p.1097-1101

2012

Volltextzugriff (PDF)

Novel Insights into Crystal-Induced Kidney Injury

Kidney diseases, 2018-06, Vol.4 (2), p.49-57

2018

Volltextzugriff (PDF)

Crystal Clots as Therapeutic Target in Cholesterol Crystal Embolism

Circulation research, 2020-04, Vol.126 (8), p.e37-e52

2020

Volltextzugriff (PDF)

Hyperoxaluria Requires TNF Receptors to Initiate Crystal Adhesion and Kidney Stone Disease

Journal of the American Society of Nephrology, 2017-03, Vol.28 (3), p.761-768

2017

Volltextzugriff (PDF)

Regulated necrosis-related molecule mRNA expression in humans and mice and in murine acute tissue injury and systemic autoimmunity leading to progressive organ damage, and progressive fibrosis

Bioscience reports, 2016-12, Vol.36 (6)

2016

Volltextzugriff (PDF)

The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is “Inflammation” Always Inflammation?

Mediators of inflammation, 2016-01, Vol.2016, p.2856213-9

2016

Volltextzugriff (PDF)

Phagocytosis of environmental or metabolic crystalline particles induces cytotoxicity by triggering necroptosis across a broad range of particle size and shape

Scientific reports, 2017-11, Vol.7 (1), p.15523-11, Article 15523

2017

Volltextzugriff (PDF)

Targeting Inflammation in So-Called Acute Kidney Injury

Seminars in nephrology, 2016, Vol.36 (1), p.17-30

2016

Volltextzugriff (PDF)

Cathepsin S inhibition combines control of systemic and peripheral pathomechanisms of autoimmune tissue injury

Scientific reports, 2017-06, Vol.7 (1), p.2775-15, Article 2775

2017

Volltextzugriff (PDF)

Necroinflammation in Kidney Disease

Journal of the American Society of Nephrology, 2016-01, Vol.27 (1), p.27-39

2016

Volltextzugriff (PDF)

1 – 25 von 86

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(83)
Mit Volltextzugriff
(82)

Publikationsform

Artikel
(84)
Buchkapitel
(2)

Sprache

Englisch
(86)
Japanisch
(23)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Crystal nephropathies: mechanisms of crystal-induced kidney injury

Neutrophils and Neutrophil Extracellular Traps Drive Necroinflammation in COVID-19

Matters of life and death. How neutrophils die or survive along NET release and is “NETosis” = necroptosis?

The macrophage phenotype and inflammasome component NLRP3 contributes to nephrocalcinosis-related chronic kidney disease independent from IL-1–mediated tissue injury

Synchronized renal tubular cell death involves ferroptosis

Multifactorial functions of the inflammasome component NLRP3 in pathogenesis of chronic kidney diseases

Cytotoxicity of crystals involves RIPK3-MLKL-mediated necroptosis

Macrophage Phenotype Controls Long-Term AKI Outcomes—Kidney Regeneration versus Atrophy

Intestinal Dysbiosis, Barrier Dysfunction, and Bacterial Translocation Account for CKD-Related Systemic Inflammation

PMA and crystal‐induced neutrophil extracellular trap formation involves RIPK1‐RIPK3‐MLKL signaling

Editorial: Negative Regulators of Innate Immunity and Their Role in Host Responses to Injury and Infection

Neutrophil Extracellular Trap-Related Extracellular Histones Cause Vascular Necrosis in Severe GN

Cellular and Molecular Mechanisms of Kidney Injury in 2,8-Dihydroxyadenine Nephropathy

RGMb protects against acute kidney injury by inhibiting tubular cell necroptosis via an MLKL-dependent mechanism

Toll-Like Receptor 4-Induced IL-22 Accelerates Kidney Regeneration

p53-Independent Roles of MDM2 in NF-κB Signaling: Implications for Cancer Therapy, Wound Healing, and Autoimmune Diseases

Novel Insights into Crystal-Induced Kidney Injury

Crystal Clots as Therapeutic Target in Cholesterol Crystal Embolism

Hyperoxaluria Requires TNF Receptors to Initiate Crystal Adhesion and Kidney Stone Disease

Regulated necrosis-related molecule mRNA expression in humans and mice and in murine acute tissue injury and systemic autoimmunity leading to progressive organ damage, and progressive fibrosis

The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is “Inflammation” Always Inflammation?

Phagocytosis of environmental or metabolic crystalline particles induces cytotoxicity by triggering necroptosis across a broad range of particle size and shape

Targeting Inflammation in So-Called Acute Kidney Injury

Cathepsin S inhibition combines control of systemic and peripheral pathomechanisms of autoimmune tissue injury

Necroinflammation in Kidney Disease

Suchergebnisse filtern