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Molecular pathways that drive diabetic kidney disease

The Journal of clinical investigation, 2023-02, Vol.133 (4), p.1-12

2023

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The transcriptomic signature of the aging podocyte

Kidney international, 2020-11, Vol.98 (5), p.1079-1081

2020

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Single-cell analysis highlights differences in druggable pathways underlying adaptive or fibrotic kidney regeneration

Nature communications, 2022-07, Vol.13 (1), p.4018-18, Article 4018

2022

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Growth hormone induces mitotic catastrophe of glomerular podocytes and contributes to proteinuria

Cell death & disease, 2021-04, Vol.12 (4), p.342-342, Article 342

2021

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Hypoxia induces ZEB2 in podocytes: Implications in the pathogenesis of proteinuria

Journal of cellular physiology, 2019-05, Vol.234 (5), p.6503-6518

2019

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NAD+ precursor supplementation prevents mtRNA/RIG-I-dependent inflammation during kidney injury

Nature metabolism, 2023-03

2023

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Acyl-CoA Synthetase Short-Chain Family 2 Is a Renal Disease Risk Gene: Controlling De Novo Lipogenesis in Kidney Tubules: FR-PO619

Journal of the American Society of Nephrology, 2023-11, Vol.34 (11S), p.573-573

2023

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Novel Actions of Growth Hormone in Podocytes: Implications for Diabetic Nephropathy

Frontiers in medicine, 2017-07, Vol.4, p.102-102

2017

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Growth Hormone and Metabolic Homeostasis

European medical journal. Diabetes, 2018-11, p.78-87

2018

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Lysine-Specific Demethylase 1A Is a Targetable Kidney Disease Causal Gene: FR-PO1196

Journal of the American Society of Nephrology, 2024-10, Vol.35 (10S)

2024

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The key role of altered tubule cell lipid metabolism in kidney disease development

Kidney international, 2024-07, Vol.106 (1), p.24-34

2024

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The Actin and Microtubule Network Regulator WHAMM Is Identified as a Key Kidney Disease Risk Gene: TH-PO1131

Journal of the American Society of Nephrology, 2024-10, Vol.35 (10S)

2024

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Deciphering Endoplasmic Reticulum Stress Activation and Its Role in Kidney Disease Progression via the STING Pathway: SA-PO1180

Journal of the American Society of Nephrology, 2024-10, Vol.35 (10S)

2024

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Establishing a Mouse Model of Heart Failure with Preserved Ejection Fraction Secondary to CKD: TH-PO203

Journal of the American Society of Nephrology, 2024-10, Vol.35 (10S)

2024

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Wasp Homologue Associated with Membranes and Microtubules Is a Kidney Disease Risk Gene: PO2502

Journal of the American Society of Nephrology, 2021-10, Vol.32 (10S), p.761-761

2021

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Changes in Gene Expression in Fatty Acid Metabolism and De Novo Lipogenesis Are Conserved in Human CKD and Animal Models: SA-OR96

Journal of the American Society of Nephrology, 2023-11, Vol.34 (11S), p.88-88

2023

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Growth hormone induces Notch1 signaling in podocytes and contributes to proteinuria in diabetic nephropathy

The Journal of biological chemistry, 2019-11, Vol.294 (44), p.16109-16122

2019

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An indispensable role for dynamin-related protein 1 in beige and brown adipogenesis

Journal of cell science, 2020-09, Vol.133 (18)

2020

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Growth hormone induces transforming growth factor-β1 in podocytes: Implications in podocytopathy and proteinuria

Biochimica et biophysica acta. Molecular cell research, 2023-02, Vol.1870 (2), p.119391-119391, Article 119391

2023

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Stabilization of hypoxia‐inducible factor 1α by cobalt chloride impairs podocyte morphology and slit‐diaphragm function

Journal of cellular biochemistry, 2019-05, Vol.120 (5), p.7667-7678

2019

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APOL1 risk variants in individuals of African genetic ancestry drive endothelial cell defects that exacerbate sepsis

Immunity (Cambridge, Mass.), 2021-11, Vol.54 (11), p.2632-2649.e6

2021

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Podocyte derived TNF‐α mediates monocyte differentiation and contributes to glomerular injury

The FASEB journal, 2022-12, Vol.36 (12), p.e22622-n/a

2022

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ACSS2 gene variants determine kidney disease risk by controlling de novo lipogenesis in kidney tubules

The Journal of clinical investigation, 2024-02, Vol.134 (4), p.1-16

2024

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Sustained mitochondrial biogenesis is essential to maintain caloric restriction-induced beige adipocytes

Metabolism, clinical and experimental, 2020-06, Vol.107, p.154225-154225, Article 154225

2020

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NAD + precursor supplementation prevents mtRNA/RIG-I-dependent inflammation during kidney injury

Nature metabolism, 2023-03, Vol.5 (3), p.414

2023

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Molecular pathways that drive diabetic kidney disease

The transcriptomic signature of the aging podocyte

Single-cell analysis highlights differences in druggable pathways underlying adaptive or fibrotic kidney regeneration

Growth hormone induces mitotic catastrophe of glomerular podocytes and contributes to proteinuria

Hypoxia induces ZEB2 in podocytes: Implications in the pathogenesis of proteinuria

NAD+ precursor supplementation prevents mtRNA/RIG-I-dependent inflammation during kidney injury

Acyl-CoA Synthetase Short-Chain Family 2 Is a Renal Disease Risk Gene: Controlling De Novo Lipogenesis in Kidney Tubules: FR-PO619

Novel Actions of Growth Hormone in Podocytes: Implications for Diabetic Nephropathy

Growth Hormone and Metabolic Homeostasis

Lysine-Specific Demethylase 1A Is a Targetable Kidney Disease Causal Gene: FR-PO1196

The key role of altered tubule cell lipid metabolism in kidney disease development

The Actin and Microtubule Network Regulator WHAMM Is Identified as a Key Kidney Disease Risk Gene: TH-PO1131

Deciphering Endoplasmic Reticulum Stress Activation and Its Role in Kidney Disease Progression via the STING Pathway: SA-PO1180

Establishing a Mouse Model of Heart Failure with Preserved Ejection Fraction Secondary to CKD: TH-PO203

Wasp Homologue Associated with Membranes and Microtubules Is a Kidney Disease Risk Gene: PO2502

Changes in Gene Expression in Fatty Acid Metabolism and De Novo Lipogenesis Are Conserved in Human CKD and Animal Models: SA-OR96

Growth hormone induces Notch1 signaling in podocytes and contributes to proteinuria in diabetic nephropathy

An indispensable role for dynamin-related protein 1 in beige and brown adipogenesis

Growth hormone induces transforming growth factor-β1 in podocytes: Implications in podocytopathy and proteinuria

Stabilization of hypoxia‐inducible factor 1α by cobalt chloride impairs podocyte morphology and slit‐diaphragm function

APOL1 risk variants in individuals of African genetic ancestry drive endothelial cell defects that exacerbate sepsis

Podocyte derived TNF‐α mediates monocyte differentiation and contributes to glomerular injury

ACSS2 gene variants determine kidney disease risk by controlling de novo lipogenesis in kidney tubules

Sustained mitochondrial biogenesis is essential to maintain caloric restriction-induced beige adipocytes

NAD + precursor supplementation prevents mtRNA/RIG-I-dependent inflammation during kidney injury

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