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K+ Efflux Is the Common Trigger of NLRP3 Inflammasome Activation by Bacterial Toxins and Particulate Matter

Immunity (Cambridge, Mass.), 2013-06, Vol.38 (6), p.1142-1153

2013

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A Critical Role for Hemolysins and Bacterial Lipoproteins in Staphylococcus aureus-Induced Activation of the Nlrp3 Inflammasome

The Journal of immunology (1950), 2009-09, Vol.183 (6), p.3942-3948

2009

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A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases

Nature medicine, 2015-03, Vol.21 (3), p.248-255

2015

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Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux

The Journal of immunology (1950), 2014-10, Vol.193 (8), p.4214-4222

2014

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Interleukin-22 Regulates the Complement System to Promote Resistance against Pathobionts after Pathogen-Induced Intestinal Damage

Immunity (Cambridge, Mass.), 2014-10, Vol.41 (4), p.620-632

2014

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Activation of the Nlrp3 inflammasome by Streptococcus pyogenes requires streptolysin O and NF-kappa B activation but proceeds independently of TLR signaling and P2X7 receptor

The Journal of immunology (1950), 2009-11, Vol.183 (9), p.5823-5829

2009

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The Nod-Like Receptor Family Member Naip5/Birc1e Restricts Legionella pneumophila Growth Independently of Caspase-1 Activation

Journal of Immunology, 2007-06, Vol.178 (12), p.8022-8027

2007

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Distinct Commensals Induce Interleukin-1β via NLRP3 Inflammasome in Inflammatory Monocytes to Promote Intestinal Inflammation in Response to Injury

Immunity (Cambridge, Mass.), 2015-04, Vol.42 (4), p.744-755

2015

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A bioluminescent caspase-1 activity assay rapidly monitors inflammasome activation in cells

Journal of immunological methods, 2017-08, Vol.447, p.1-13

2017

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Inflammasomes as microbial sensors

European journal of immunology, 2010-03, Vol.40 (3), p.611-615

2010

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Sensing and reacting to microbes through the inflammasomes

Nature immunology, 2012-04, Vol.13 (4), p.325

2012

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Caspase-11 Requires the Pannexin-1 Channel and the Purinergic P2X7 Pore to Mediate Pyroptosis and Endotoxic Shock

Immunity (Cambridge, Mass.), 2015-11, Vol.43 (5), p.923-932

2015

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Shigella type III secretion protein MxiI is recognized by Naip2 to induce Nlrc4 inflammasome activation independently of Pkcδ

PLoS pathogens, 2014-02, Vol.10 (2), p.e1003926-e1003926

2014

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The inflammasome: a caspase-1-activation platform that regulates immune responses and disease pathogenesis

Nature immunology, 2009-03, Vol.10 (3), p.241-247

2009

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3,4-Methylenedioxy-β-nitrostyrene Inhibits NLRP3 Inflammasome Activation by Blocking Assembly of the Inflammasome

The Journal of biological chemistry, 2014-01, Vol.289 (2), p.1142-1150

2014

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Staphylococcus δ-toxin induces allergic skin disease by activating mast cells

Nature (London), 2013-11, Vol.503 (7476), p.397-401

2013

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Bacterial RNA mediates activation of caspase-1 and IL-1β release independently of TLRs 3, 7, 9 and TRIF but is dependent on UNC93B

The Journal of immunology (1950), 2012-07, Vol.189 (1), p.328-336

2012

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Abstract 1319: Monitoring inflammasome activation with a bioluminescent caspase-1 assay

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.1319-1319

2015

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In vivo mapping of a protective linear neutralizing epitope at the N-terminus of alpha hemolysin from Staphylococcus aureus

Molecular immunology, 2014-07, Vol.60 (1), p.62-71

2014

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Cytosolic Double-Stranded RNA Activates the NLRP3 inflammasomevia MAVS-Induced Membrane Permeabilization and K+ Efflux

The Journal of immunology (1950), 2014-09, Vol.193 (8), p.4214-4222

2014

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Distinct Commensals Induce Interleukin-1[beta] via NLRP3 Inflammasome in Inflammatory Monocytes to Promote Intestinal Inflammation in Response to Injury

Immunity (Cambridge, Mass.), 2015-04, Vol.42 (4), p.744

2015

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A Critical Role for Hemolysins and Bacterial Lipoproteins in Staphylococcus aureus-Induced Activation of the Nlrp3 Inflammasome1

The Journal of immunology (1950), 2009-08, Vol.183 (6), p.3942-3948

2009

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A small molecule inhibitior of the NLRP3 inflammasome is a potential therapeutic for inflammatory diseases

Nature medicine, 2015-02, Vol.21 (3), p.248-255

2015

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The Inflammasome: A Caspase-1 Activation Platform Regulating Immune Responses and Disease Pathogenesis

Nature immunology, 2009-03, Vol.10 (3), p.241

2009

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Activation of the Nlrp3 Inflammasome by Streptococcus pyogenes Requires Streptolysin O and NF-κB Activation but Proceeds Independently of TLR Signaling and P2X7 Receptor1

The Journal of immunology (1950), 2009-10, Vol.183 (9), p.5823-5829

2009

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K+ Efflux Is the Common Trigger of NLRP3 Inflammasome Activation by Bacterial Toxins and Particulate Matter

A Critical Role for Hemolysins and Bacterial Lipoproteins in Staphylococcus aureus-Induced Activation of the Nlrp3 Inflammasome

A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases

Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux

Interleukin-22 Regulates the Complement System to Promote Resistance against Pathobionts after Pathogen-Induced Intestinal Damage

Activation of the Nlrp3 inflammasome by Streptococcus pyogenes requires streptolysin O and NF-kappa B activation but proceeds independently of TLR signaling and P2X7 receptor

The Nod-Like Receptor Family Member Naip5/Birc1e Restricts Legionella pneumophila Growth Independently of Caspase-1 Activation

Distinct Commensals Induce Interleukin-1β via NLRP3 Inflammasome in Inflammatory Monocytes to Promote Intestinal Inflammation in Response to Injury

A bioluminescent caspase-1 activity assay rapidly monitors inflammasome activation in cells

Inflammasomes as microbial sensors

Sensing and reacting to microbes through the inflammasomes

Caspase-11 Requires the Pannexin-1 Channel and the Purinergic P2X7 Pore to Mediate Pyroptosis and Endotoxic Shock

Shigella type III secretion protein MxiI is recognized by Naip2 to induce Nlrc4 inflammasome activation independently of Pkcδ

The inflammasome: a caspase-1-activation platform that regulates immune responses and disease pathogenesis

3,4-Methylenedioxy-β-nitrostyrene Inhibits NLRP3 Inflammasome Activation by Blocking Assembly of the Inflammasome

Staphylococcus δ-toxin induces allergic skin disease by activating mast cells

Bacterial RNA mediates activation of caspase-1 and IL-1β release independently of TLRs 3, 7, 9 and TRIF but is dependent on UNC93B

Abstract 1319: Monitoring inflammasome activation with a bioluminescent caspase-1 assay

In vivo mapping of a protective linear neutralizing epitope at the N-terminus of alpha hemolysin from Staphylococcus aureus

Cytosolic Double-Stranded RNA Activates the NLRP3 inflammasomevia MAVS-Induced Membrane Permeabilization and K+ Efflux

Distinct Commensals Induce Interleukin-1[beta] via NLRP3 Inflammasome in Inflammatory Monocytes to Promote Intestinal Inflammation in Response to Injury

A Critical Role for Hemolysins and Bacterial Lipoproteins in Staphylococcus aureus-Induced Activation of the Nlrp3 Inflammasome1

A small molecule inhibitior of the NLRP3 inflammasome is a potential therapeutic for inflammatory diseases

The Inflammasome: A Caspase-1 Activation Platform Regulating Immune Responses and Disease Pathogenesis

Activation of the Nlrp3 Inflammasome by Streptococcus pyogenes Requires Streptolysin O and NF-κB Activation but Proceeds Independently of TLR Signaling and P2X7 Receptor1

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