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RAX, the PKR activator, sensitizes cells to inflammatory cytokines, serum withdrawal, chemotherapy, and viral infection

Blood, 2006-08, Vol.108 (3), p.821-829

2006

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A Single Amino Acid Change in the L-Polymerase Protein of Vesicular Stomatitis Virus Completely Abolishes Viral mRNA Cap Methylation

Journal of Virology, 2005-06, Vol.79 (12), p.7327-7337

2005

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Two N-terminal regions of the Sendai virus L RNA polymerase protein participate in oligomerization

Virology (New York, N.Y.), 2007-06, Vol.363 (1), p.189-197

2007

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Characterization of the Amino Acid Residues of Sendai Virus C Protein That Are Critically Involved in Its Interferon Antagonism and RNA Synthesis Down-Regulation

Journal of Virology, 2004-07, Vol.78 (14), p.7443-7454

2004

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The phosphoprotein (P) and L binding sites reside in the N-terminus of the L subunit of the measles virus RNA polymerase

Virology (New York, N.Y.), 2004-10, Vol.327 (2), p.297-306

2004

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The L–L oligomerization domain resides at the very N-terminus of the sendai virus L RNA polymerase protein

Virology (New York, N.Y.), 2003-09, Vol.313 (2), p.525-536

2003

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Identification of a new region in the vesicular stomatitis virus L polymerase protein which is essential for mRNA cap methylation

Virology (New York, N.Y.), 2006-07, Vol.350 (2), p.394-405

2006

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The Phosphoprotein (P) Binding Site Resides in the N Terminus of the L Polymerase Subunit of Sendai Virus

Journal of Virology, 2002-03, Vol.76 (6), p.3078-3083

2002

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The C-Terminal 88 Amino Acids of the Sendai Virus P Protein Have Multiple Functions Separable by Mutation

Journal of Virology, 2002-01, Vol.76 (1), p.68-77

2002

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The L polymerase protein of parainfluenza virus 3 forms an oligomer and can interact with the heterologous Sendai virus L, P and C proteins

Virology (New York, N.Y.), 2004-01, Vol.318 (1), p.439-450

2004

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Intragenic Complementation and Oligomerization of the L Subunit of the Sendai Virus RNA Polymerase

Virology (New York, N.Y.), 2002-12, Vol.304 (2), p.235-245

2002

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Sendai Virus Wild-Type and Mutant C Proteins Show a Direct Correlation between L Polymerase Binding and Inhibition of Viral RNA Synthesis

Virology (New York, N.Y.), 2001-09, Vol.288 (1), p.96-108

2001

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Mapping the phosphoprotein binding site on Sendai virus NP protein assembled into nucleocapsids

Virology (New York, N.Y.), 2004-08, Vol.325 (2), p.216-224

2004

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The Sendai Virus C Protein Binds the L Polymerase Protein to Inhibit Viral RNA Synthesis

Virology (New York, N.Y.), 1997-09, Vol.235 (2), p.261-270

1997

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Dissection of Individual Functions of the Sendai Virus Phosphoprotein in Transcription

Journal of Virology, 1999-08, Vol.73 (8), p.6474-6483

1999

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Different Substitutions at Conserved Amino Acids in Domains II and III in the Sendai L RNA Polymerase Protein Inactivate Viral RNA Synthesis

Virology (New York, N.Y.), 2002-12, Vol.304 (1), p.135-145

2002

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A Highly Conserved Region of the Sendai Virus Nucleocapsid Protein Contributes to the NP–NP Binding Domain

Virology (New York, N.Y.), 1997-03, Vol.229 (2), p.322-335

1997

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Domains of the Measles Virus N Protein Required for Binding to P Protein and Self-Assembly

Virology (New York, N.Y.), 1996-02, Vol.216 (1), p.272-277

1996

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Mutations in Conserved Domain II of the Large (L) Subunit of the Sendai Virus RNA Polymerase Abolish RNA Synthesis

Virology (New York, N.Y.), 1999-09, Vol.262 (2), p.375-383

1999

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Requirements for Measles Virus Induction of RANTES Chemokine in Human Astrocytoma-Derived U373 Cells

Journal of Virology, 1999-04, Vol.73 (4), p.3117-3124

1999

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Comparison of Identical Temperature-Sensitive Mutations in the L Polymerase Proteins of Sendai and Parainfluenza3 Viruses

Virology (New York, N.Y.), 2000-10, Vol.276 (1), p.190-201

2000

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An Amino-Proximal Domain of the L Protein Binds to the P Protein in the Measles Virus RNA Polymerase Complex

Virology (New York, N.Y.), 1994-12, Vol.205 (2), p.540-545

1994

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Role of Primary Constitutive Phosphorylation of Sendai Virus P and V Proteins in Viral Replication and Pathogenesis

Virology (New York, N.Y.), 1999-10, Vol.263 (1), p.195-208

1999

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Viral proteins required for the in Vitro replication of vesicular stomatitis virus defective interfering particle genome RNA

Virology (New York, N.Y.), 1988-02, Vol.162 (2), p.369-376

1988

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The vesicular stomatitis virus L protein possesses the mRNA methyltransferase activities

Virology (New York, N.Y.), 1988-03, Vol.163 (1), p.222-225

1988

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Rna, Viral - Biosynthesis
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Cell Line
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Transcription, Genetic
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Sendai Virus
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RAX, the PKR activator, sensitizes cells to inflammatory cytokines, serum withdrawal, chemotherapy, and viral infection

A Single Amino Acid Change in the L-Polymerase Protein of Vesicular Stomatitis Virus Completely Abolishes Viral mRNA Cap Methylation

Two N-terminal regions of the Sendai virus L RNA polymerase protein participate in oligomerization

Characterization of the Amino Acid Residues of Sendai Virus C Protein That Are Critically Involved in Its Interferon Antagonism and RNA Synthesis Down-Regulation

The phosphoprotein (P) and L binding sites reside in the N-terminus of the L subunit of the measles virus RNA polymerase

The L–L oligomerization domain resides at the very N-terminus of the sendai virus L RNA polymerase protein

Identification of a new region in the vesicular stomatitis virus L polymerase protein which is essential for mRNA cap methylation

The Phosphoprotein (P) Binding Site Resides in the N Terminus of the L Polymerase Subunit of Sendai Virus

The C-Terminal 88 Amino Acids of the Sendai Virus P Protein Have Multiple Functions Separable by Mutation

The L polymerase protein of parainfluenza virus 3 forms an oligomer and can interact with the heterologous Sendai virus L, P and C proteins

Intragenic Complementation and Oligomerization of the L Subunit of the Sendai Virus RNA Polymerase

Sendai Virus Wild-Type and Mutant C Proteins Show a Direct Correlation between L Polymerase Binding and Inhibition of Viral RNA Synthesis

Mapping the phosphoprotein binding site on Sendai virus NP protein assembled into nucleocapsids

The Sendai Virus C Protein Binds the L Polymerase Protein to Inhibit Viral RNA Synthesis

Dissection of Individual Functions of the Sendai Virus Phosphoprotein in Transcription

Different Substitutions at Conserved Amino Acids in Domains II and III in the Sendai L RNA Polymerase Protein Inactivate Viral RNA Synthesis

A Highly Conserved Region of the Sendai Virus Nucleocapsid Protein Contributes to the NP–NP Binding Domain

Domains of the Measles Virus N Protein Required for Binding to P Protein and Self-Assembly

Mutations in Conserved Domain II of the Large (L) Subunit of the Sendai Virus RNA Polymerase Abolish RNA Synthesis

Requirements for Measles Virus Induction of RANTES Chemokine in Human Astrocytoma-Derived U373 Cells

Comparison of Identical Temperature-Sensitive Mutations in the L Polymerase Proteins of Sendai and Parainfluenza3 Viruses

An Amino-Proximal Domain of the L Protein Binds to the P Protein in the Measles Virus RNA Polymerase Complex

Role of Primary Constitutive Phosphorylation of Sendai Virus P and V Proteins in Viral Replication and Pathogenesis

Viral proteins required for the in Vitro replication of vesicular stomatitis virus defective interfering particle genome RNA

The vesicular stomatitis virus L protein possesses the mRNA methyltransferase activities

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