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The effect of cell growth phase on the regulatory cross-talk between flagellar and Spi1 virulence gene expression

PLoS pathogens, 2014-03, Vol.10 (3), p.e1003987-e1003987

2014

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FliT Selectively Enhances Proteolysis of FlhC Subunit in FlhD4C2 Complex by an ATP-dependent Protease, ClpXP

The Journal of biological chemistry, 2014-11, Vol.289 (47), p.33001-33011

2014

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A role for the calmodulin kinase II-related anchoring protein (αkap) in maintaining the stability of nicotinic acetylcholine receptors

The Journal of neuroscience, 2012-04, Vol.32 (15), p.5177-5185

2012

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Signal-dependent Requirement for the Co-activator Protein RcsA in Transcription of the RcsB-regulated ugd Gene

The Journal of biological chemistry, 2003-12, Vol.278 (50), p.50588-50595

2003

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The Effect of Cell Growth Phase on the Regulatory Cross-Talk between Flagellar and Spi1 Virulence Gene Expression: e1003987

PLoS pathogens, 2014-03, Vol.10 (3)

2014

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Sensing by bacterial regulatory systems in host and non-host environments

Nature reviews. Microbiology, 2006-09, Vol.4 (9), p.705-709

2006

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Calcium/calmodulin kinase II-dependent acetylcholine receptor cycling at the mammalian neuromuscular junction in vivo

The Journal of neuroscience, 2010-09, Vol.30 (37), p.12455-12465

2010

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The PmrA/PmrB and RcsC/YojN/RcsB systems control expression of the Salmonella O‐antigen chain length determinant

Molecular microbiology, 2006-04, Vol.60 (1), p.39-50

2006

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Activation of the RcsC/YojN/RcsB phosphorelay system attenuates Salmonella virulence

Molecular microbiology, 2004-10, Vol.54 (2), p.386-395

2004

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Control of the Salmonella ugd gene by three two‐component regulatory systems

Molecular microbiology, 2003-01, Vol.47 (2), p.335-344

2003

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Nicotinic acetylcholine receptor stability at the NMJ deficient in α-syntrophin in vivo

The Journal of neuroscience, 2011-10, Vol.31 (43), p.15586-15596

2011

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Conflicting needs for a Salmonella hypervirulence gene in host and non‐host environments

Molecular microbiology, 2002-08, Vol.45 (4), p.1019-1027

2002

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Molecular mechanisms of Salmonella pathogenesis

Current opinion in infectious diseases, 2000-10, Vol.13 (5), p.519-522

2000

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Cell Division Inhibition in Salmonella typhimuriumHistidine-Constitutive Strains: an ftsI-Like Defect in the Presence of Wild-Type Penicillin-Binding Protein 3 Levels

Journal of Bacteriology, 1998-10, Vol.180 (19), p.5231

1998

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Cell division inhibition in Salmonella typhimurium histidine-constitutive strains: an ftsI-like defect in the presence of wild-type penicillin-binding protein 3 levels

Journal of bacteriology, 1998-10, Vol.180 (19), p.5231-5234

1998

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New methods in Salmonella genetics

Microbiologia (Madrid, Spain), 1994-12, Vol.10 (4), p.357

1994

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The effect of cell growth phase on the regulatory cross-talk between flagellar and Spi1 virulence gene expression

FliT Selectively Enhances Proteolysis of FlhC Subunit in FlhD4C2 Complex by an ATP-dependent Protease, ClpXP

A role for the calmodulin kinase II-related anchoring protein (αkap) in maintaining the stability of nicotinic acetylcholine receptors

Signal-dependent Requirement for the Co-activator Protein RcsA in Transcription of the RcsB-regulated ugd Gene

The Effect of Cell Growth Phase on the Regulatory Cross-Talk between Flagellar and Spi1 Virulence Gene Expression: e1003987

Sensing by bacterial regulatory systems in host and non-host environments

Calcium/calmodulin kinase II-dependent acetylcholine receptor cycling at the mammalian neuromuscular junction in vivo

The PmrA/PmrB and RcsC/YojN/RcsB systems control expression of the Salmonella O‐antigen chain length determinant

Activation of the RcsC/YojN/RcsB phosphorelay system attenuates Salmonella virulence

Control of the Salmonella ugd gene by three two‐component regulatory systems

Nicotinic acetylcholine receptor stability at the NMJ deficient in α-syntrophin in vivo

Conflicting needs for a Salmonella hypervirulence gene in host and non‐host environments

Molecular mechanisms of Salmonella pathogenesis

Cell Division Inhibition in Salmonella typhimuriumHistidine-Constitutive Strains: an ftsI-Like Defect in the Presence of Wild-Type Penicillin-Binding Protein 3 Levels

Cell division inhibition in Salmonella typhimurium histidine-constitutive strains: an ftsI-like defect in the presence of wild-type penicillin-binding protein 3 levels

New methods in Salmonella genetics

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