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Prolactin synergizes with canonical Wnt signals to drive development of ER+ mammary tumors via activation of the Notch pathway

Cancer letters, 2021-04, Vol.503, p.231-239

2021

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High rates of chromosome missegregation suppress tumor progression but do not inhibit tumor initiation

Molecular biology of the cell, 2016-07, Vol.27 (13), p.1981-1989

2016

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Quantitative Trait Loci Associated with Susceptibility to Bladder and Kidney Infections Induced by Escherichia coli in Female C3H/HeJ Mice

The Journal of infectious diseases, 2009-02, Vol.199 (3), p.355-361

2009

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Tumorigenesis in the Multiple Intestinal Neoplasia Mouse: Redundancy of Negative Regulators and Specificity of Modifiers

Proceedings of the National Academy of Sciences - PNAS, 2000-03, Vol.97 (7), p.3461-3466

2000

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Pathway Pathology : Histological Differences Between ErbB/Ras and Wnt Pathway Transgenic Mammary Tumors

The American journal of pathology, 2002-09, Vol.161 (3), p.1087-1097

2002

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Loss of Apc+ in intestinal adenomas from Min mice

Cancer research (Chicago, Ill.), 1994-11, Vol.54 (22), p.5947-5952

1994

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Genetic Evaluation of Candidate Genes for the Mom1 Modifier of Intestinal Neoplasia in Mice

Genetics (Austin), 1996-12, Vol.144 (4), p.1777-1785

1996

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Prevalence of ATM Sequence Variants in Northern Plains American Indian Cancer Patients

Frontiers in oncology, 2013-01, Vol.3, p.318-318

2013

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Tumorigenesis in the multiple intestinal neoplasia mouse: Redundancy of negative regulators and specificity of modifiers

Proceedings of the National Academy of Sciences - PNAS, 2000-03, Vol.97 (7), p.3461-3466

2000

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Studies of neoplasia in the Min mouse

Biochimica et biophysica acta, 1997-04, Vol.1332 (2), p.F25-F48

1997

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Up-regulation of fibroblast growth factor-binding protein, by β-catenin during colon carcinogenesis

Cancer research (Chicago, Ill.), 2003-12, Vol.63 (23), p.8085-8089

2003

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N-ethyl-N-nitrosourea treatment of multiple intestinal neoplasia (Min) mice : age-related effects on the formation of intestinal adenomas : cystic crypts, and epidermoid cysts

Cancer research (Chicago, Ill.), 1995-10, Vol.55 (19), p.4479-4485

1995

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A Resistant Genetic Background Leading to Incomplete Penetrance of Intestinal Neoplasia and Reduced Loss of Heterozygosity in ApcMin/+ Mice

Proceedings of the National Academy of Sciences - PNAS, 1998-09, Vol.95 (18), p.10826-10831

1998

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Somatic mutational mechanisms involved in intestinal tumor formation in Min mice

Cancer research (Chicago, Ill.), 1997-05, Vol.57 (10), p.1999-2006

1997

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Transgenic Mouse Models That Explore the Multistep Hypothesis of Intestinal Neoplasia

The Journal of cell biology, 1993-11, Vol.123 (4), p.877-893

1993

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Abstract 3273: Prolactin promotes breast lesion progression and alters tumor histotype

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.3273-3273

2010

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ROSA26 LacZ-neo ᴿ insertion confers resistance to mammary tumors in Apc ᴹⁱⁿ/⁺ mice

Mammalian genome, 2001-08, Vol.12 (8), p.606-611

2001

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ROSA26 mice carry a modifier of Min-induced mammary and intestinal tumor development

Mammalian genome, 2000-12, Vol.11 (12), p.1058-1062

2000

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Genetic identification of Mom-1, a major modifier locus affecting Min-induced intestinal neoplasia in the mouse

Cell, 1993-11, Vol.75 (4), p.631-639

1993

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Quantitative Trait Loci Associated with Susceptibility to Bladder and Kidney Infections Induced by Escherichia coli in Female C3H/HeJMice

The Journal of infectious diseases, 2009-02, Vol.199 (3), p.355-361

2009

Volltextzugriff (PDF)

Quantitative Trait Loci Associated with Susceptibility to Escherichia coli Bladder and Kidney Infections in C3H/HeJ Female Mice

The Journal of infectious diseases, 2009-02, Vol.199 (3), p.355-361

2009

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Sulindac suppresses tumorigenesis in the Min mouse

Carcinogenesis (New York), 1996-08, Vol.17 (8), p.1757-1760

1996

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The Min (Multiple Intestinal Neoplasia) Mutation: Its Effect on Gut Epithelial Cell Differentiation and Interaction with a Modifier System

The Journal of cell biology, 1992-03, Vol.116 (6), p.1517-1526

1992

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The ROSA26 LacZ-neoR insertion confers resistance to mammary tumors in ApcMin/+ mice

Mammalian genome, 2001-08, Vol.12 (8), p.606

2001

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Pathway Pathology

The American journal of pathology, 2002-09, Vol.161 (3), p.1087-1097

2002

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Prolactin synergizes with canonical Wnt signals to drive development of ER+ mammary tumors via activation of the Notch pathway

High rates of chromosome missegregation suppress tumor progression but do not inhibit tumor initiation

Quantitative Trait Loci Associated with Susceptibility to Bladder and Kidney Infections Induced by Escherichia coli in Female C3H/HeJ Mice

Tumorigenesis in the Multiple Intestinal Neoplasia Mouse: Redundancy of Negative Regulators and Specificity of Modifiers

Pathway Pathology : Histological Differences Between ErbB/Ras and Wnt Pathway Transgenic Mammary Tumors

Loss of Apc+ in intestinal adenomas from Min mice

Genetic Evaluation of Candidate Genes for the Mom1 Modifier of Intestinal Neoplasia in Mice

Prevalence of ATM Sequence Variants in Northern Plains American Indian Cancer Patients

Tumorigenesis in the multiple intestinal neoplasia mouse: Redundancy of negative regulators and specificity of modifiers

Studies of neoplasia in the Min mouse

Up-regulation of fibroblast growth factor-binding protein, by β-catenin during colon carcinogenesis

N-ethyl-N-nitrosourea treatment of multiple intestinal neoplasia (Min) mice : age-related effects on the formation of intestinal adenomas : cystic crypts, and epidermoid cysts

A Resistant Genetic Background Leading to Incomplete Penetrance of Intestinal Neoplasia and Reduced Loss of Heterozygosity in ApcMin/+ Mice

Somatic mutational mechanisms involved in intestinal tumor formation in Min mice

Transgenic Mouse Models That Explore the Multistep Hypothesis of Intestinal Neoplasia

Abstract 3273: Prolactin promotes breast lesion progression and alters tumor histotype

ROSA26 LacZ-neo ᴿ insertion confers resistance to mammary tumors in Apc ᴹⁱⁿ/⁺ mice

ROSA26 mice carry a modifier of Min-induced mammary and intestinal tumor development

Genetic identification of Mom-1, a major modifier locus affecting Min-induced intestinal neoplasia in the mouse

Quantitative Trait Loci Associated with Susceptibility to Bladder and Kidney Infections Induced by Escherichia coli in Female C3H/HeJMice

Quantitative Trait Loci Associated with Susceptibility to Escherichia coli Bladder and Kidney Infections in C3H/HeJ Female Mice

Sulindac suppresses tumorigenesis in the Min mouse

The Min (Multiple Intestinal Neoplasia) Mutation: Its Effect on Gut Epithelial Cell Differentiation and Interaction with a Modifier System

The ROSA26 LacZ-neoR insertion confers resistance to mammary tumors in ApcMin/+ mice

Pathway Pathology

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