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Light affects mitochondria to cause apoptosis to cultured cells: possible relevance to ganglion cell death in certain optic neuropathies

Journal of neurochemistry, 2008-06, Vol.105 (5), p.2013-2028

2008

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Magnetic resonance spectroscopy reveals mitochondrial dysfunction in amyotrophic lateral sclerosis

Brain (London, England : 1878), 2020-12, Vol.143 (12), p.3603-3618

2020

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Modulating Mitophagy in Mitochondrial Disease

Current medicinal chemistry, 2018-01, Vol.25 (40), p.5597

2018

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Mitochondrial impairment in patients with Parkinson disease with the G2019S mutation in LRRK2

Neurology, 2010-11, Vol.75 (22), p.2017

2010

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Peripheral Glycolysis in Neurodegenerative Diseases

International journal of molecular sciences, 2020-11, Vol.21 (23), p.8924

2020

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Rapamycin activation of 4E-BP prevents parkinsonian dopaminergic neuron loss

Nature neuroscience, 2009-09, Vol.12 (9), p.1129-1135

2009

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C9orf72 expansion within astrocytes reduces metabolic flexibility in amyotrophic lateral sclerosis

Brain (London, England : 1878), 2019-12, Vol.142 (12), p.3771-3790

2019

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Mitochondrial Dysfunction in Alzheimer's Disease: A Biomarker of the Future?

Biomedicines, 2021-01, Vol.9 (1), p.63

2021

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The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function

Nature communications, 2023-09, Vol.14 (1), p.5496-5496, Article 5496

2023

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Mitochondrial function and morphology are impaired in parkin-mutant fibroblasts

Annals of neurology, 2008-11, Vol.64 (5), p.555-565

2008

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Superoxide dismutase 1 mutation in a cellular model of amyotrophic lateral sclerosis shifts energy generation from oxidative phosphorylation to glycolysis

Neurobiology of aging, 2014-06, Vol.35 (6), p.1499-1509

2014

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The Parkinson's disease related mutant VPS35 (D620N) amplifies the LRRK2 response to endolysosomal stress

Biochemical journal, 2024-02, Vol.481 (4), p.265-278

2024

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The complex I subunit NDUFA10 selectively rescues Drosophila pink1 mutants through a mechanism independent of mitophagy

PLoS genetics, 2014-11, Vol.10 (11), p.e1004815-e1004815

2014

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Oxidative switch drives mitophagy defects in dopaminergic parkin mutant patient neurons

Scientific reports, 2020-09, Vol.10 (1), p.15485-14, Article 15485

2020

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Loss of IGF1R in Human Astrocytes Alters Complex I Activity and Support for Neurons

Neuroscience, 2018-10, Vol.390, p.46-59

2018

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Mutations in CHMP2B in lower motor neuron predominant amyotrophic lateral sclerosis (ALS)

PloS one, 2010-03, Vol.5 (3), p.e9872-e9872

2010

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FBXL4 ubiquitin ligase deficiency promotes mitophagy by elevating NIX levels

The EMBO journal, 2023-07, Vol.42 (13), p.e112799-n/a

2023

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Unexpected phenotypic and molecular changes of combined glucocerebrosidase and acid sphingomyelinase deficiency

Disease models & mechanisms, 2023-06, Vol.16 (6)

2023

Volltextzugriff (PDF)

Multimodal assessment of mitochondrial function in Parkinson's disease

Brain (London, England : 1878), 2024-01, Vol.147 (1), p.267-280

2024

Volltextzugriff (PDF)

Ursodeoxycholic Acid Improves Mitochondrial Function and Redistributes Drp1 in Fibroblasts from Patients with Either Sporadic or Familial Alzheimer's Disease

Journal of molecular biology, 2018-10, Vol.430 (21), p.3942-3953

2018

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TigarB causes mitochondrial dysfunction and neuronal loss in PINK1 deficiency

Annals of neurology, 2013-12, Vol.74 (6), p.837-847

2013

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A novel quantitative assay of mitophagy: Combining high content fluorescence microscopy and mitochondrial DNA load to quantify mitophagy and identify novel pharmacological tools against pathogenic heteroplasmic mtDNA

Pharmacological research, 2015-10, Vol.100, p.24-35

2015

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Deficits in mitochondrial function and glucose metabolism seen in sporadic and familial Alzheimer’s disease derived Astrocytes are ameliorated by increasing hexokinase 1

Alzheimer's & dementia, 2023-12, Vol.19 (S13), p.n/a

2023

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Light affects mitochondria to cause apoptosis to cultured cells: possible relevance to ganglion cell death in certain optic neuropathies

Magnetic resonance spectroscopy reveals mitochondrial dysfunction in amyotrophic lateral sclerosis

Modulating Mitophagy in Mitochondrial Disease

Mitochondrial impairment in patients with Parkinson disease with the G2019S mutation in LRRK2

Peripheral Glycolysis in Neurodegenerative Diseases

Rapamycin activation of 4E-BP prevents parkinsonian dopaminergic neuron loss

C9orf72 expansion within astrocytes reduces metabolic flexibility in amyotrophic lateral sclerosis

Mitochondrial Dysfunction in Alzheimer's Disease: A Biomarker of the Future?

The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function

Mitochondrial function and morphology are impaired in parkin-mutant fibroblasts

Superoxide dismutase 1 mutation in a cellular model of amyotrophic lateral sclerosis shifts energy generation from oxidative phosphorylation to glycolysis

The Parkinson's disease related mutant VPS35 (D620N) amplifies the LRRK2 response to endolysosomal stress

The complex I subunit NDUFA10 selectively rescues Drosophila pink1 mutants through a mechanism independent of mitophagy

Dysregulated mitophagy and mitochondrial organization in optic atrophy due to OPA1 mutations

Oxidative switch drives mitophagy defects in dopaminergic parkin mutant patient neurons

Loss of IGF1R in Human Astrocytes Alters Complex I Activity and Support for Neurons

USP30 sets a trigger threshold for PINK1-PARKIN amplification of mitochondrial ubiquitylation

Mutations in CHMP2B in lower motor neuron predominant amyotrophic lateral sclerosis (ALS)

FBXL4 ubiquitin ligase deficiency promotes mitophagy by elevating NIX levels

Unexpected phenotypic and molecular changes of combined glucocerebrosidase and acid sphingomyelinase deficiency

Multimodal assessment of mitochondrial function in Parkinson's disease

Ursodeoxycholic Acid Improves Mitochondrial Function and Redistributes Drp1 in Fibroblasts from Patients with Either Sporadic or Familial Alzheimer's Disease

TigarB causes mitochondrial dysfunction and neuronal loss in PINK1 deficiency

A novel quantitative assay of mitophagy: Combining high content fluorescence microscopy and mitochondrial DNA load to quantify mitophagy and identify novel pharmacological tools against pathogenic heteroplasmic mtDNA

Deficits in mitochondrial function and glucose metabolism seen in sporadic and familial Alzheimer’s disease derived Astrocytes are ameliorated by increasing hexokinase 1

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