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Mechanical Load Increases in Bone Formation via a Sclerostin‐Independent Pathway

Journal of bone and mineral research, 2014-11, Vol.29 (11), p.2456-2467

2014

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Sclerostin antibody enhances bone formation in a rat model of distraction osteogenesis

Journal of orthopaedic research, 2018-04, Vol.36 (4), p.1106-1113

2018

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Myogenic progenitors contribute to open but not closed fracture repair

BMC musculoskeletal disorders, 2011-12, Vol.12 (1), p.288-288, Article 288

2011

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α-Actinin-3 deficiency is associated with reduced bone mass in human and mouse

Bone (New York, N.Y.), 2011-10, Vol.49 (4), p.790-798

2011

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Rapid cell culture and pre-clinical screening of a transforming growth factor-beta (TGF-beta) inhibitor for orthopaedics

BMC musculoskeletal disorders, 2010-05, Vol.11 (1), p.105-105, Article 105

2010

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Sclerostin Antibody Augments the Anabolic Bone Formation Response in a Mouse Model of Mechanical Tibial Loading

Journal of bone and mineral research, 2018-03, Vol.33 (3), p.486-498

2018

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Pretreatment with Pamidronate Decreases Bone Formation but Increases Callus Bone Volume in a Rat Closed Fracture Model

Calcified tissue international, 2020-02, Vol.106 (2), p.172-179

2020

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Increased anabolic bone response in Dkk1 KO mice following tibial compressive loading

Bone (New York, N.Y.), 2020-02, Vol.131, p.115054-115054, Article 115054

2020

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Inhibition of sclerostin by systemic treatment with sclerostin antibody enhances healing of proximal tibial defects in ovariectomized rats

Journal of orthopaedic research, 2012-10, Vol.30 (10), p.1541-1548

2012

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Dkk1 KO Mice Treated with Sclerostin Antibody Have Additional Increases in Bone Volume

Calcified tissue international, 2018-09, Vol.103 (3), p.298-310

2018

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Sclerostin Antibody Increases Callus Size and Strength but does not Improve Fracture Union in a Challenged Open Rat Fracture Model

Calcified tissue international, 2017-08, Vol.101 (2), p.217-228

2017

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Homozygous Dkk1 Knockout Mice Exhibit High Bone Mass Phenotype Due to Increased Bone Formation

Calcified tissue international, 2018, Vol.102 (1), p.105-116

2018

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Intermittent PTH(1–34) does not increase union rates in open rat femoral fractures and exhibits attenuated anabolic effects compared to closed fractures

Bone (New York, N.Y.), 2010-03, Vol.46 (3), p.852-859

2010

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Modeling bone morphogenetic protein and bisphosphonate combination therapy in wild-type and Nf1 haploinsufficient mice

Journal of orthopaedic research, 2008-01, Vol.26 (1), p.65-74

2008

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Matrix Metalloproteinase–Driven Endochondral Fracture Union Proceeds Independently of Osteoclast Activity

Journal of bone and mineral research, 2013-07, Vol.28 (7), p.1550-1560

2013

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Models of tibial fracture healing in normal and Nf1-deficient mice

Journal of orthopaedic research, 2008-08, Vol.26 (8), p.1053-1060

2008

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Characterization of the bone phenotype and fracture repair in osteopetrotic Incisors absent rats

Journal of orthopaedic research, 2011-05, Vol.29 (5), p.726-733

2011

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MUSCLE CELL CONTRIBUTION TO INTRAMEMBRANOUS BONE HEALING IN A METAPHYSEAL DEFECT MODEL IN MICE

Bone (New York, N.Y.), 2010, Vol.46, p.S45-S45

2010

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HOMOZYGOUS DELETION OF DICKKOPF-1 RESULTS IN A HIGH BONE MASS PHENOTYPE

Bone (New York, N.Y.), 2010, Vol.46, p.S76-S77

2010

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MUSCLE PROGENITORS MAKE A MAJOR CONTRIBUTION TO OPEN FRACTURE REPAIR

Bone (New York, N.Y.), 2010, Vol.46, p.S52-S53

2010

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SCLEROSTIN NEUTRALISING ANTIBODY ENHANCES HEALING OF TIBIAL METAPHYSEAL DEFECT IN AN OVX RAT MODEL

Bone (New York, N.Y.), 2010, Vol.46, p.S25-S25

2010

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TREATMENT OF CONGENITAL PSEUDARTHROSIS OF THE TIBIA USING RECOMBINANT BONE MORPHOGENETIC PROTEIN AND BISPHOSPHONATES

Bone (New York, N.Y.), 2010, Vol.46, p.S20-S21

2010

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Bone healing and the involvement of muscle stem cells

Bone (New York, N.Y.), 2008-10, Vol.43, p.S36-S36

2008

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Normal endochondral fracture union follows continuous bisphosphonate pre-treatment in a rat closed fracture model

Bone (New York, N.Y.), 2008-03, Vol.42, p.S79-S80

2008

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Inhibiting the osteocyte-specific protein sclerostin increases bone mass and fracture resistance in multiple myeloma

Mechanical Load Increases in Bone Formation via a Sclerostin‐Independent Pathway

Sclerostin antibody enhances bone formation in a rat model of distraction osteogenesis

Myogenic progenitors contribute to open but not closed fracture repair

α-Actinin-3 deficiency is associated with reduced bone mass in human and mouse

Rapid cell culture and pre-clinical screening of a transforming growth factor-beta (TGF-beta) inhibitor for orthopaedics

Sclerostin Antibody Augments the Anabolic Bone Formation Response in a Mouse Model of Mechanical Tibial Loading

Pretreatment with Pamidronate Decreases Bone Formation but Increases Callus Bone Volume in a Rat Closed Fracture Model

Increased anabolic bone response in Dkk1 KO mice following tibial compressive loading

Inhibition of sclerostin by systemic treatment with sclerostin antibody enhances healing of proximal tibial defects in ovariectomized rats

Dkk1 KO Mice Treated with Sclerostin Antibody Have Additional Increases in Bone Volume

Sclerostin Antibody Increases Callus Size and Strength but does not Improve Fracture Union in a Challenged Open Rat Fracture Model

Homozygous Dkk1 Knockout Mice Exhibit High Bone Mass Phenotype Due to Increased Bone Formation

Intermittent PTH(1–34) does not increase union rates in open rat femoral fractures and exhibits attenuated anabolic effects compared to closed fractures

Modeling bone morphogenetic protein and bisphosphonate combination therapy in wild-type and Nf1 haploinsufficient mice

Matrix Metalloproteinase–Driven Endochondral Fracture Union Proceeds Independently of Osteoclast Activity

Models of tibial fracture healing in normal and Nf1-deficient mice

Characterization of the bone phenotype and fracture repair in osteopetrotic Incisors absent rats

MUSCLE CELL CONTRIBUTION TO INTRAMEMBRANOUS BONE HEALING IN A METAPHYSEAL DEFECT MODEL IN MICE

HOMOZYGOUS DELETION OF DICKKOPF-1 RESULTS IN A HIGH BONE MASS PHENOTYPE

MUSCLE PROGENITORS MAKE A MAJOR CONTRIBUTION TO OPEN FRACTURE REPAIR

SCLEROSTIN NEUTRALISING ANTIBODY ENHANCES HEALING OF TIBIAL METAPHYSEAL DEFECT IN AN OVX RAT MODEL

TREATMENT OF CONGENITAL PSEUDARTHROSIS OF THE TIBIA USING RECOMBINANT BONE MORPHOGENETIC PROTEIN AND BISPHOSPHONATES

Bone healing and the involvement of muscle stem cells

Normal endochondral fracture union follows continuous bisphosphonate pre-treatment in a rat closed fracture model

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