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Repeated phencyclidine in monkeys results in loss of parvalbumin-containing axo-axonic projections in the prefrontal cortex

Psychopharmacologia, 2007-06, Vol.192 (2), p.283-290

2007

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An Antisense Oligonucleotide Reverses the Footshock-Induced Expression of Fos in the Rat Medial Prefrontal Cortex and the Subsequent Expression of Conditioned Fear-Induced Immobility

The Journal of neuroscience, 1999-07, Vol.19 (13), p.5666-5673

1999

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Phencyclidine-induced Loss of Asymmetric Spine Synapses in Rodent Prefrontal Cortex is Reversed by Acute and Chronic Treatment with Olanzapine

Neuropsychopharmacology (New York, N.Y.), 2011-09, Vol.36 (10), p.2054-2061

2011

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Prenatal exposure to cocaine is associated with increased number of spine synapses in rat prelimbic cortex

Synapse (New York, N.Y.), 2007-10, Vol.61 (10), p.862-865

2007

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Clozapine Normalizes Prefrontal Cortex Dopamine Transmission in Monkeys Subchronically Exposed to Phencyclidine

Neuropsychopharmacology (New York, N.Y.), 2008-02, Vol.33 (3), p.491-496

2008

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TMT, a predator odor, elevates mesoprefrontal dopamine metabolic activity and disrupts short-term working memory in the rat

Brain research bulletin, 2000-08, Vol.52 (6), p.519-523

2000

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Apoptotic natural cell death in developing primate dopamine midbrain neurons occurs during a restricted period in the second trimester of gestation

Experimental neurology, 2007-04, Vol.204 (2), p.802-807

2007

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Fear-like biochemical and behavioral responses in rats to the predator odor, TMT, are dependent on the exposure environment

Synapse (New York, N.Y.), 2002-10, Vol.46 (1), p.11-18

2002

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Prenatal cocaine exposure disrupts non-spatial, short-term memory in adolescent and adult male rats

Behavioural brain research, 2002-02, Vol.129 (1), p.217-223

2002

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Noradrenergic α-2 agonists have anxiolytic-like actions on stress-related behavior and mesoprefrontal dopamine biochemistry

Brain research, 2004-11, Vol.1027 (1), p.173-178

2004

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Axo-Axonic Structures in the Medial Prefrontal Cortex of the Rat: Reduction by Prenatal Exposure to Cocaine

The Journal of neuroscience, 2003-06, Vol.23 (12), p.5227-5234

2003

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Prenatal exposure to cocaine selectively disrupts the development of parvalbumin containing local circuit neurons in the medial prefrontal cortex of the rat

Synapse (New York, N.Y.), 2005-04, Vol.56 (1), p.1-11

2005

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Male Rats Exposed to Cocaine in utero Demonstrate Elevated Expression of Fos in the Prefrontal Cortex in Response to Environment

Neuropsychopharmacology (New York, N.Y.), 2002-03, Vol.26 (3), p.275-285

2002

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Development of A9/A10 dopamine neurons during the second and third trimesters in the African green monkey

Journal of comparative neurology (1911), 2005-07, Vol.488 (2), p.215-223

2005

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Selective activation of the A10, but not A9, dopamine neurons in the rat by the predator odor, 2,5-dihydro-2,4,5-trimethylthiazoline

Neuroscience letters, 2002-08, Vol.328 (3), p.209-212

2002

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Impact of methamphetamine on dopamine neurons in primates is dependent on age: implications for development of Parkinson's disease

Neuroscience, 2011-08, Vol.189, p.277-285

2011

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The predator odor, TMT, displays a unique, stress-like pattern of dopaminergic and endocrinological activation in the rat

Brain research, 2000-05, Vol.864 (1), p.146-151

2000

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Divergent effects of putative anxiolytics on stress-induced Fos expression in the mesoprefrontal system of the rat

Synapse (New York, N.Y.), 2000-05, Vol.36 (2), p.143-154

2000

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Serotonergic lesions alter cocaine-induced locomotor behavior and stress-activation of the mesocorticolimbic dopamine system

Synapse (New York, N.Y.), 1996-07, Vol.23 (3), p.174-181

1996

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Prenatal cocaine exposure increases mesoprefrontal dopamine neuron responsivity to mild stress

Synapse (New York, N.Y.), 2001-11, Vol.42 (2), p.80-83

2001

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Prenatal exposure to cocaine reduces the number and enhances reactivity of A10 dopaminergic neurons to environmental stress

Synapse (New York, N.Y.), 2001-09, Vol.41 (4), p.337-344

2001

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Morphine acutely and persistently attenuates nonvesicular GABA release in rat nucleus accumbens

Synapse (New York, N.Y.), 2001-11, Vol.42 (2), p.87-94

2001

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Biochemical and behavioral anxiolytic-like effects of R(+)HA-966 at the level of the ventral tegmental area in rats

Psychopharmacologia, 1999-04, Vol.143 (3), p.227-234

1999

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Stress activation of mesocorticolimbic dopamine neurons: Effects of a glycine/NMDA receptor antagonist

European journal of pharmacology, 1993-07, Vol.238 (2), p.255-262

1993

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(S)-(-)-HA-966, a gamma-hydroxybutyrate-like agent, prevents enhanced mesocorticolimbic dopamine metabolism and behavioral correlates of restraint stress, conditioned fear and cocaine sensitization

The Journal of pharmacology and experimental therapeutics, 1997-11, Vol.283 (2), p.712

1997

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Repeated phencyclidine in monkeys results in loss of parvalbumin-containing axo-axonic projections in the prefrontal cortex

An Antisense Oligonucleotide Reverses the Footshock-Induced Expression of Fos in the Rat Medial Prefrontal Cortex and the Subsequent Expression of Conditioned Fear-Induced Immobility

Phencyclidine-induced Loss of Asymmetric Spine Synapses in Rodent Prefrontal Cortex is Reversed by Acute and Chronic Treatment with Olanzapine

Prenatal exposure to cocaine is associated with increased number of spine synapses in rat prelimbic cortex

Clozapine Normalizes Prefrontal Cortex Dopamine Transmission in Monkeys Subchronically Exposed to Phencyclidine

TMT, a predator odor, elevates mesoprefrontal dopamine metabolic activity and disrupts short-term working memory in the rat

Apoptotic natural cell death in developing primate dopamine midbrain neurons occurs during a restricted period in the second trimester of gestation

Fear-like biochemical and behavioral responses in rats to the predator odor, TMT, are dependent on the exposure environment

Prenatal cocaine exposure disrupts non-spatial, short-term memory in adolescent and adult male rats

Noradrenergic α-2 agonists have anxiolytic-like actions on stress-related behavior and mesoprefrontal dopamine biochemistry

Axo-Axonic Structures in the Medial Prefrontal Cortex of the Rat: Reduction by Prenatal Exposure to Cocaine

Prenatal exposure to cocaine selectively disrupts the development of parvalbumin containing local circuit neurons in the medial prefrontal cortex of the rat

Male Rats Exposed to Cocaine in utero Demonstrate Elevated Expression of Fos in the Prefrontal Cortex in Response to Environment

Development of A9/A10 dopamine neurons during the second and third trimesters in the African green monkey

Selective activation of the A10, but not A9, dopamine neurons in the rat by the predator odor, 2,5-dihydro-2,4,5-trimethylthiazoline

Impact of methamphetamine on dopamine neurons in primates is dependent on age: implications for development of Parkinson's disease

The predator odor, TMT, displays a unique, stress-like pattern of dopaminergic and endocrinological activation in the rat

Divergent effects of putative anxiolytics on stress-induced Fos expression in the mesoprefrontal system of the rat

Serotonergic lesions alter cocaine-induced locomotor behavior and stress-activation of the mesocorticolimbic dopamine system

Prenatal cocaine exposure increases mesoprefrontal dopamine neuron responsivity to mild stress

Prenatal exposure to cocaine reduces the number and enhances reactivity of A10 dopaminergic neurons to environmental stress

Morphine acutely and persistently attenuates nonvesicular GABA release in rat nucleus accumbens

Biochemical and behavioral anxiolytic-like effects of R(+)HA-966 at the level of the ventral tegmental area in rats

Stress activation of mesocorticolimbic dopamine neurons: Effects of a glycine/NMDA receptor antagonist

(S)-(-)-HA-966, a gamma-hydroxybutyrate-like agent, prevents enhanced mesocorticolimbic dopamine metabolism and behavioral correlates of restraint stress, conditioned fear and cocaine sensitization

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